Associate Professor, Medicine, University of Colorado School of Medicine
Nephrology
Dr. Joshua M. Thurman, MD
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Joshua M. Thurman practices Nephrology. He researched medicine at University of Chicago Pritzker School of Medicine. University of Chicago Pritzker School of Medicine has a rank of 10 in research and a 19 in primary care He received awards:"CMS Meaningful Use Stage 1 Certification" and "Elected Member". Joshua Thurman is a published doctor. He has 68 publications published. The most current publication "A study of antibody and T cell recognition of rhoptry-associated protein-1 (RAP-1) and RAP-2 recombinant proteins and peptides of Plasmodium falciparum in migrants and..." He is registered with Medicare.
Publications
- γδ T cells protect against LPS-induced lung injury.
- Annexin A2 Enhances Complement Activation by Inhibiting Factor H.
- New anti-complement drugs: not so far away.
- Mycophenolate mofetil enhances the negative effects of sirolimus and tacrolimus on rat Kidney cell metabolism.
- The role of the complement system in Acute kidney injury.
- Nanosized contrast agents to noninvasively detect Kidney inflammation by magnetic resonance imaging.
- Renal ischemia-reperfusion injury amplifies the humoral immune response.
- Proteomics and metabolomics in Renal transplantation-quo vadis?
- Noninvasive detection of complement activation through radiologic imaging.
- IgM Contributes to Glomerular Injury in FSGS.
- Therapeutic inhibition of the alternative complement pathway attenuates Chronic EAE.
- Factor friction: protective and pathogenic roles for complement factors in immune complex Glomerulonephritis.
- Deletion of the Complement C5a Receptor Alleviates the Severity of Acute Pneumococcal Otitis Media following Influenza A Virus Infection in Mice.
- A study of antibody and T cell recognition of rhoptry-associated protein-1 (RAP-1) and RAP-2 recombinant proteins and peptides of Plasmodium falciparum in migrants and...
- Bacillus cereus induces permeability of an in vitro blood-retina barrier.
- Analysis of inhibitory epitopes in the Plasmodium falciparum rhoptry protein RAP-1 including identification of a second inhibitory epitope.
- Peeking into the black box: new biomarkers for Acute kidney injury.
- Type I interferon signaling is involved in the spontaneous development of lupus-like Disease in B6.Nba2 and (B6.Nba2 x NZW)F(1) mice.
- Genetic susceptibility to polyI:C-induced IFNalpha/beta-dependent accelerated Disease in lupus-prone mice.
- Binding of avian IgY to type VII collagen does not activate complement and leucocytes and fails to induce subepidermal blistering in mice.
- Microvascular destruction identifies murine allografts that cannot be rescued from airway fibrosis.
- New approaches to the treatment of dense deposit Disease.
- The alternative pathway of complement is activated in the glomeruli and tubulointerstitium of mice With adriamycin nephropathy.
- The alternative pathway of complement activation is critical for blister induction in experimental epidermolysis bullosa acquisita.
- Inhibition of the alternative complement activation pathway in traumatic brain injury by a monoclonal anti-factor B antibody: a randomized placebo-controlled study in ...
- C3a is required for the production of CXC chemokines by tubular epithelial cells after Renal ishemia/reperfusion.
- Acute renal failure after bilateral nephrectomy is associated with cytokine-mediated pulmonary injury.
- Candidate inhibitors of porcine complement.
- Triggers of inflammation after Renal ischemia/reperfusion.
- Complement activation induces dysregulation of angiogenic factors and causes fetal rejection and growth restriction.
- Sublytic membrane-attack-complex activation and VEGF secretion in retinal pigment epithelial cells.
- The Critical Role of Complement Alternative Pathway Regulator Factor H in Allergen-Induced Airway Hyperresponsiveness and Inflammation.
- Identification of a central role for complement in osteoarthritis.
- Detection of glomerular complement C3 fragments by magnetic resonance imaging in murine lupus Nephritis.
- IgG Autoantibodies against Deposited C3 Inhibit Macrophage-Mediated Apoptotic Cell Engulfment in Systemic Autoimmunity.
- Binding of factor H to tubular epithelial cells limits interstitial complement activation in ischemic injury.
- Essential role of factor B of the alternative complement pathway in complement activation and opsonophagocytosis during Acute pneumococcal otitis media in mice.
- Delineation of the complement receptor type 2-C3d complex by site-directed mutagenesis and molecular docking.
- Dynamic control of the complement system by modulated expression of regulatory proteins.
- The role of endothelial cell injury in thrombotic microangiopathy.
- B cell subsets contribute to Renal injury and Renal protection after ischemia/reperfusion.
- A targeted inhibitor of the complement alternative pathway reduces RPE injury and angiogenesis in models of age-related macular degeneration.
- The complement inhibitors Crry and factor H are critical for preventing autologous complement activation on Renal tubular epithelial cells.
- An allelic variant of Crry in the murine Sle1c lupus susceptibility interval is not impaired in its ability to regulate complement activation.
- Renal inflammation: targeted iron oxide nanoparticles for molecular MR imaging in mice.
- Aseptic injury to epithelial cells alters cell surface complement regulation in a tissue specific fashion.
- Enhanced susceptibility to Acute pneumococcal otitis media in mice deficient in complement C1qa, factor B, and factor B/C2.
- Targeted inhibition of the complement alternative pathway With complement receptor 2 and factor H attenuates collagen antibody-induced arthritis in mice.
- Alternative pathway of complement in children With diarrhea-associated hemolytic uremic syndrome.
- Oxidative stress renders retinal pigment epithelial cells susceptible to complement-mediated injury.
- Ca2+/calmodulin-dependent kinase II triggers cell membrane injury by inducing complement factor B gene expression in the mouse Heart.
- Evidence for non-traditional activation of complement factor C3 during murine liver regeneration.
- Alternative complement pathway activation is essential for inflammation and joint destruction in the passive transfer model of collagen-induced arthritis.
- Reduced neuronal cell death after experimental brain injury in mice lacking a functional alternative pathway of complement activation.
- Factor B of the alternative complement pathway regulates development of airway hyperresponsiveness and inflammation.
- Pharmacological complement inhibition at the C3 convertase level promotes neuronal survival, neuroprotective intracerebral gene expression, and neurological outcome af...
- Treatment With an inhibitory monoclonal antibody to mouse factor B protects mice from induction of apoptosis and renal ischemia/reperfusion injury.
- Altered Renal tubular expression of the complement inhibitor Crry permits complement activation after ischemia/reperfusion.
- The central role of the alternative complement pathway in human Disease.
- Acute tubular necrosis is characterized by activation of the alternative pathway of complement.
- A novel inhibitor of the alternative complement pathway prevents antiphospholipid antibody-induced pregnancy loss in mice.
- The alternative pathway of complement in Disease: opportunities for therapeutic targeting.
- Complement C5a receptors and neutrophils mediate fetal injury in the antiphospholipid Syndrome.
- Effect of primary polydipsia on aquaporin and sodium transporter abundance.
- Comparative effects of angiotensin-converting enzyme inhibitors and angiotensin receptor blockers on blood pressure and the Kidney.
- Urinary concentrating defect in hypothyroid rats: role of sodium, potassium, 2-chloride co-transporter, and aquaporins.
- Lack of a functional alternative complement pathway ameliorates ischemic Acute renal failure in mice.
Schools
University Of Chicago Division Of The Biological Sciences The Pritzker School Of Medicine
Beth Israel Deaconess Medical Center Program
University Of Colorado University Hospital Program
Procedures Preformed
- Hemodialysis
- Peritoneal Dialysis
Conditions Treated
- Acidosis
- Acute Kidney Failure
- Alkalosis
- Calcium Metabolism Disorders
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Doctors Specialties
- Nephrology
Accepted Insurances
Awards
- CMS Meaningful Use Stage 1 Certification
- Elected Member
Education
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University of Colorado -
Beth Israel Deaconess Medical Center -
University of Chicago Pritzker School of Medicine
Hospital
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University of Colorado Hospital
Drug Facts
| NPI NUMBER | 1346330248 | |
| NPPES Provider LastName | THURMAN | |
| NPPES Provider FirstName | JOSHUA | |
| NPPES Provider ZIPCode | 800452545 | |
| NPPES Provider State | CO | |
| Specialty Description | Nephrology | |
| Total Claim Count | 117.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1346330248 |
| Last Name Of The Provider | THURMAN |
| First Name Of The Provider | JOSHUA |
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