Assistant Professor, Pediatrics, UCSF School of Medicine

Pediatrics

Joseph T. Shieh is an expert in the field of Pediatrics. Joseph T. Shieh, MD is a published MD as well. He has 19 publications published. The latest was: Microglia express CCR5, CXCR4, and CCR3, but of these, CCR5 is the principal coreceptor for human immunodeficiency virus type 1 dementia isolates.

Publications

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• Clinical and ultrastructural spectrum of diffuse lung disease associated with surfactant protein C mutations.
• Consanguinity and the risk of congenital heart disease.
• Characterization of cultured microglia that can be infected by HIV-1.
• Chemokine receptor utilization by human immunodeficiency virus type 1 isolates that replicate in microglia.
• Determinants of syncytium formation in microglia by human immunodeficiency virus type 1: role of the V1/V2 domains.
• Microglia express CCR5, CXCR4, and CCR3, but of these, CCR5 is the principal coreceptor for human immunodeficiency virus type 1 dementia isolates.
• Neuroimaging findings in macrocephaly-capillary malformation: a longitudinal study of 17 patients.
• The coxsackievirus and adenovirus receptor is a transmembrane component of the tight junction.
• Elevated miR-499 levels blunt the cardiac stress response.
• A Genome-Wide Screen Reveals a Role for microRNA-1 in Modulating Cardiac Cell Polarity.
• Array comparative genomic hybridization analysis in patients with anophthalmia, microphthalmia, and coloboma.
• Heart malformation: what are the chances it could happen again?
• Novel deoxyguanosine kinase gene mutations and viral infection predispose apparently healthy children to fulminant liver failure.
• Clinical features and management issues in Mowat-Wilson syndrome.
• Systemic hyalinosis: a distinctive early childhood-onset disorder characterized by mutations in the anthrax toxin receptor 2 gene (ANTRX2).
• Nablus mask-like facial syndrome is caused by a microdeletion of 8q detected by array-based comparative genomic hybridization.
• Triplication of 8p22-8p23 in a patient with features similar to Kabuki syndrome.
• Interaction with decay-accelerating factor facilitates coxsackievirus B infection of polarized epithelial cells.

Schools

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• Perelman School of Medicine University of Pennsylvania

Doctors Specialties

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• Clinical Genetics
• Pediatrics

Accepted Insurances

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Education

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• University of Washington

Hospital

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• UCSF Medical Center

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