Professor, Surgery, Brown University Program in Medicine
Family Medicine
Dr. Jorge E. Albina, MD
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Jorge Albina practices Family Medicine. Jorge E. Albina is also a published MD. He has 78 paper published. The most recent paper is: Regulation of arginase isoforms I and II by IL-4 in cultured murine peritoneal macrophages. Jorge Albina accepts Medicare payments and is listed with Medicare.gov.
Publications
- Toll-like receptor 4 signaling regulates the acute local inflammatory response to injury and the fibrosis/neovascularization of sterile wounds.
- Wound macrophages as key regulators of repair: origin, phenotype, and function.
- The phenotype of murine wound macrophages.
- The phenotype of murine wound macrophages.
- Disruption of interleukin-1 signaling improves the quality of wound healing.
- Prostaglandin E2 suppresses lipopolysaccharide-stimulated IFN-beta production.
- Use of Ly6G-specific monoclonal antibody to deplete neutrophils in mice.
- Polymicrobial sepsis enhances clearance of apoptotic immune cells by splenic macrophages.
- Integrin engagement mediates the human polymorphonuclear leukocyte response to a fungal pathogen-associated molecular pattern.
- Beta-glucan is a fungal determinant for adhesion-dependent human neutrophil functions.
- Biography: Henry T. Randall, MD.
- The effect of PGG-beta-glucan on neutrophil chemotaxis in vivo.
- Ankyrin repeat and SOCS box 3 (ASB3) mediates ubiquitination and degradation of tumor necrosis factor receptor II.
- Modulation of macrophage phenotype by soluble product(s) released from neutrophils.
- Transcriptional regulation of TNF-alpha production in neutropenia.
- Macrophage arginase regulation by CCAAT/enhancer-binding protein beta.
- Polymicrobial sepsis induces divergent effects on splenic and peritoneal dendritic cell function in mice.
- Beta-glucan affects leukocyte navigation in a complex chemotactic gradient.
- The lectin-like domain of complement receptor 3 protects endothelial barrier function from activated neutrophils.
- Determination of the role of hypoxia-inducible factor 1 in wound healing.
- Oxygen and the regulation of gene expression in wounds.
- Detection of reactive oxygen intermediate production by macrophages.
- Bacterial colonization and the expression of inducible nitric oxide synthase in murine wounds.
- HIF-1 expression in healing wounds: HIF-1alpha induction in primary inflammatory cells by TNF-alpha.
- Receptor-mediated phagocytosis of rat macrophages is regulated differentially for opsonized particles and non-opsonized particles containing beta-glucan.
- Macrophage-induced neutrophil apoptosis.
- TA1/LAT-1/CD98 light chain and system L activity, but not 4F2/CD98 heavy chain, respond to arginine availability in rat hepatic cells. Loss Of response in tumor cells.
- Trauma-hemorrhage delays wound healing potentially by increasing pro-inflammatory cytokines at the wound site.
- Acyl phosphatase activity of NO-inhibited glyceraldehyde-3-phosphate dehydrogenase (GAPDH): a potential mechanism for uncoupling glycolysis from ATP generation in NO-p...
- Vestigial respiratory burst activity in wound macrophages.
- Promotion of neutrophil chemotaxis through differential regulation of beta 1 and beta 2 integrins.
- Predictive versus measured energy expenditure using limits-of-agreement analysis in hospitalized, obese patients.
- Molecular and metabolic evidence for the restricted expression of inducible nitric oxide synthase in healing wounds.
- Regulation of arginase isoforms I and II by IL-4 in cultured murine peritoneal macrophages.
- Macrophage phagocytosis of wound neutrophils.
- Problems of total parenteral nutrition.
- Effect of IL-6 overexpression on the metastatic potential of rat hepatocellular carcinoma cells.
- Role of nitric oxide in mediation of macrophage cytotoxicity and apoptosis.
- Distinct arginase isoforms expressed in primary and transformed macrophages: regulation by oxygen tension.
- Effects of hepatocellular mitogens on cytokine-induced nitric oxide synthesis in human hepatocytes.
- NO is not sufficient to explain maximal cytotoxicity of tumoricidal macrophages against an NO-sensitive cell line.
- B cell lymphoma-2 transfected P815 cells resist reactive nitrogen intermediate-mediated macrophage-dependent cytotoxicity.
- Interleukin-6 production by rat hepatocellular carcinoma cells is associated with metastatic potential but not with tumorigenicity.
- On the expression of nitric oxide synthase by human macrophages. Why no NO?
- Macrophage activation by culture in an anoxic environment.
- Impact of nitric oxide on macrophage glucose metabolism and glyceraldehyde-3-phosphate dehydrogenase activity.
- Nitric oxide in inflammation and immunity.
- Interleukin-6 activity in wounds.
- Activated murine macrophages induce apoptosis in tumor cells through nitric oxide-dependent or -independent mechanisms.
- Detrimental effects of an omega-3 fatty acid-enriched diet on wound healing.
- Suppression of arthritis by an inhibitor of nitric oxide synthase.
- Role of ornithine as a proline precursor in healing wounds.
- Modulation of glucose metabolism in macrophages by products of nitric oxide synthase.
- Nitric oxide-mediated apoptosis in murine peritoneal macrophages.
- Adequate energy intake and improved prealbumin concentration as indicators of the response to total parenteral nutrition.
- Nitric oxide production is required for murine resident peritoneal macrophages to suppress mitogen-stimulated T cell proliferation. Role of IFN-gamma in the induction ...
- Suppression of lymphocyte proliferation through the nitric oxide synthesizing pathway.
- Concomitant macrophage activation and fibroblast/lymphocyte inhibition by wound fluid: the "arginine-deficiency of inflammation" is a partial explanation.
- Temporal expression of different pathways of 1-arginine metabolism in healing wounds.
- Subclavian vein stenosis and thrombosis: a potential serious complication in chronic hemodialysis patients.
- Regulation of macrophage physiology by L-arginine: role of the oxidative L-arginine deiminase pathway.
- Subcutaneous infusion ports for administration of parenteral nutrition at home.
- Transitional feeding: the relationship between nutritional intake and plasma protein concentrations.
- Use of retinol-binding protein and prealbumin as indicators of the response to nutrition therapy.
- Regulation of macrophage functions by L-arginine.
- Catastrophic weight loss after vertical banded gastroplasty: malnutrition and neurologic alterations.
- Arginine metabolism in wounds.
- Glutamine metabolism in rat skeletal muscle wounded with lambda-carrageenan.
- Detection of total parenteral nutrition-induced fatty liver infiltration in the rat by in vitro proton nuclear magnetic resonance.
- Amino acid metabolism after lambda-carrageenan injury to rat skeletal muscle.
- Nitrogen utilization from elemental diets.
- Evidence for aerobic glycolysis in lambda-carrageenan-wounded skeletal muscle.
- Effect of starvation on the local and systemic metabolic effects of the lambda-carrageenan wound.
- Effects of hypocaloric dextrose and amino acids on body composition and nitrogen balance in rats.
- [Fat emulsions for parenteral use].
- [Hypertrophy of the ileocecal valve. Malabsorption syndrome].
- The monocyte to macrophage transition in the murine sterile wound.
Schools
Universidad Nacional De La Plata Facultad De Ciencias Medicas
Doctors Specialties
- Surgery
- Family Medicine
Accepted Insurances
Hospital
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Rhode Island Hospital
Drug Facts
| NPI NUMBER | 1558367987 | |
| NPPES Provider LastName | ALBINA | |
| NPPES Provider FirstName | JORGE | |
| NPPES Provider ZIPCode | 029034923 | |
| NPPES Provider State | RI | |
| Specialty Description | General Practice | |
| Total Claim Count | 40.0 | |
| Distinct Opioid Count | 1.0 | |
| Opioid Claim Count | 26.0 | |
| Percent Opioid Claims | 65.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1558367987 |
| Last Name Of The Provider | ALBINA |
| First Name Of The Provider | JORGE |
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