Professor, Medicine, Case Western Reserve University School of Medicine
Pulmonology
Dr. Jonathan S. Stamler, MD
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Jonathan Stamler works in the field of Pulmonology. He researched medicine at Icahn School of Medicine at Mount Sinai. Icahn School of Medicine at Mount Sinai is ranked 20/62 in Research/PrimaryCare. He received 2 awards: "Investigator Alumnus" and "Elected Member". Dr. Jonathan S. Stamler is a published doctor as well. He has 101 publications published. The latest was: Hemoglobin βCys93 is essential for cardiovascular function and integrated response to hypoxia. He is registered with Medicare and accepts Medicare payments.
Publications
- Hemoglobin βCys93 is essential for cardiovascular function and integrated response to hypoxia.
- Identification of S-nitroso-CoA reductases that regulate protein S-nitrosylation.
- Redox pioneer: professor stuart a. Lipton.
- S-nitrosylation therapy to improve oxygen delivery of banked blood.
- Insights into BAY 60-2770 Activation and S-Nitrosylation-Dependent Desensitization of Soluble Guanylyl Cyclase via Crystal Structures of Homologous Nostoc H-NOX Domain...
- Repletion of S-Nitrosohemoglobin Improves Organ Function and Physiological Status in Swine After Brain Death.
- S-nitrosylation: integrator of cardiovascular performance and oxygen delivery.
- Is the flavohemoglobin a nitric oxide dioxygenase?
- Off-target thiol alkylation by the NADPH oxidase inhibitor 3-benzyl-7-(2-benzoxazolyl)thio-1,2,3-triazolo4,5-dpyrimidine (VAS2870).
- Endogenous protein S-Nitrosylation in E. coli: regulation by OxyR.
- Regulation by S-nitrosylation of Protein Posttranslational Modification.
- Oxygen-coupled redox regulation of the skeletal muscle ryanodine receptor-Ca2+ release channel by NADPH oxidase 4.
- Glioma stem cell proliferation and tumor growth are promoted by nitric oxide synthase-2.
- S-nitrosylation at the interface of autophagy and disease.
- The transfusion problem: role of aberrant S-nitrosylation.
- Pharmacologically augmented S-nitrosylated hemoglobin improves recovery from murine subarachnoid hemorrhage.
- Site-specific analysis of protein S-acylation by resin-assisted capture.
- Host S-nitrosylation inhibits clostridial small molecule-activated glucosylating toxins.
- Identification of S-nitrosylated targets of thioredoxin using a quantitative proteomic approach.
- EPR spectroscopy of nitrite complexes of methemoglobin.
- GSNO reductase and beta2-adrenergic receptor gene-gene interaction: bronchodilator responsiveness to albuterol.
- S-nitrosylation in cardiovascular signaling.
- Thioredoxin-interacting protein (Txnip) is a feedback regulator of S-nitrosylation.
- Inclusion of a nitric oxide congener in the insufflation gas repletes S-nitrosohemoglobin and stabilizes physiologic status during prolonged carbon dioxide pneumoperit...
- A protein microarray-based analysis of S-nitrosylation.
- S-Nitrosylation of cardiac ion channels.
- S-nitrosoglutathione reductase: an important regulator in human asthma.
- Assays for S-nitrosothiols and S-nitrosylated proteins and mechanistic insights into cardioprotection.
- Protection from lipopolysaccharide-induced lung injury by augmentation of airway S-nitrosothiols.
- Proteomic analysis of S-nitrosylation and denitrosylation by resin-assisted capture.
- Nitrite-methemoglobin inadequate for hypoxic vasodilation.
- Endogenous S-nitrosothiols protect against myocardial injury.
- A genetic analysis of nitrosative stress.
- Detection of protein S-nitrosylation with the biotin-switch technique.
- Oxygen-regulated beta(2)-adrenergic receptor hydroxylation by EGLN3 and ubiquitylation by pVHL.
- Regulation of the cardiac muscle ryanodine receptor by O(2) tension and S-nitrosoglutathione.
- Myocyte specific overexpression of myoglobin impairs angiogenesis after hind-limb ischemia.
- SNO-hemoglobin and hypoxic vasodilation.
- Nitroglycerin-mediated S-nitrosylation of proteins: a field comes full cycle.
- Hypoxic vasodilation by red blood cells: evidence for an s-nitrosothiol-based signal.
- S-nitrosylation of beta-arrestin regulates beta-adrenergic receptor trafficking.
- Redox-based regulation of signal transduction: principles, pitfalls, and promises.
- Regulated protein denitrosylation by cytosolic and mitochondrial thioredoxins.
- A SNO storm in skeletal muscle.
- Fiber type-specific nitric oxide protects oxidative myofibers against cachectic stimuli.
- Interactions of NO with hemoglobin: from microbes to man.
- S-nitrosohemoglobin deficiency: a mechanism for loss of physiological activity in banked blood.
- NOS2 regulation of NF-kappaB by S-nitrosylation of p65.
- Inhaled ethyl nitrite prevents hyperoxia-impaired postnatal alveolar development in newborn rats.
- Efficacy of ethyl nitrite in reversing surgical vasospasm.
- Assessment and application of the biotin switch technique for examining protein S-nitrosylation under conditions of pharmacologically induced oxidative stress.
- Regulation of beta-adrenergic receptor signaling by S-nitrosylation of G-protein-coupled receptor kinase 2.
- Structural and functional consequences of coenzyme binding to the inactive asian variant of mitochondrial aldehyde dehydrogenase: roles of residues 475 and 487.
- Assessment of nitric oxide signals by triiodide chemiluminescence.
- A classification scheme for redox-based modifications of proteins.
- S-nitrosylation TRiPs a calcium switch.
- Bioactivation of nitroglycerin by the mitochondrial aldehyde dehydrogenase.
- S-nitrosylating agents: a novel class of compounds that increase cystic fibrosis transmembrane conductance regulator expression and maturation in epithelial cells.
- Maintenance of nitric oxide and redox homeostasis by the salmonella flavohemoglobin hmp.
- Nitrosative stress in the ER: a new role for S-nitrosylation in neurodegenerative diseases.
- S-nitrosothiol signaling in respiratory biology.
- An S-nitrosothiol (SNO) synthase function of hemoglobin that utilizes nitrite as a substrate.
- Nitric oxide regulates endocytosis by S-nitrosylation of dynamin.
- S-nitrosoglutathione inhibits alpha1-adrenergic receptor-mediated vasoconstriction and ligand binding in pulmonary artery.
- A nitric oxide processing defect of red blood cells created by hypoxia: deficiency of S-nitrosohemoglobin in pulmonary hypertension.
- A novel inhaled organic nitrate that affects pulmonary vascular tone in a piglet model of hypoxia-induced pulmonary hypertension.
- An essential role for mitochondrial aldehyde dehydrogenase in nitroglycerin bioactivation.
- Avicinylation (thioesterification): a protein modification that can regulate the response to oxidative and nitrosative stress.
- Protection from experimental asthma by an endogenous bronchodilator.
- Oxygen regulation of tumor perfusion by S-nitrosohemoglobin reveals a pressor activity of nitric oxide.
- Assessments of the chemistry and vasodilatory activity of nitrite with hemoglobin under physiologically relevant conditions.
- NO/redox disequilibrium in the failing heart and cardiovascular system.
- Impaired vasodilation by red blood cells in sickle cell disease.
- A method to attenuate pneumoperitoneum-induced reductions in splanchnic blood flow.
- Role of circulating S-nitrosothiols in control of blood pressure.
- Chemical physiology of blood flow regulation by red blood cells: the role of nitric oxide and S-nitrosohemoglobin.
- Maternal insufflation during the second trimester equivalent produces hypercapnia, acidosis, and prolonged hypoxia in fetal sheep.
- Relationship of blood transfusion and clinical outcomes in patients with acute coronary syndromes.
- Role of mitochondrial aldehyde dehydrogenase in nitroglycerin-induced vasodilation of coronary and systemic vessels: an intact canine model.
- S-nitrosylation: physiological regulation of NF-kappaB.
- New insights into protein S-nitrosylation. Mitochondria as a model system.
- Cardioprotective effects of erythropoietin in the reperfused ischemic heart: a potential role for cardiac fibroblasts.
- S-nitrosothiols in the blood: roles, amounts, and methods of analysis.
- Essential roles of S-nitrosothiols in vascular homeostasis and endotoxic shock.
- Central role of mitochondrial aldehyde dehydrogenase and reactive oxygen species in nitroglycerin tolerance and cross-tolerance.
- Inhibition of iNOS with 1400W improves contractile function and alters nos gene and protein expression in reperfused skeletal muscle.
- Concerted regulation of skeletal muscle contractility by oxygen tension and endogenous nitric oxide.
- Translation of academic discovery into societal benefit: proposal for a balanced approach--part 2.
- Regulation of ion channel structure and function by reactive oxygen-nitrogen species.
- Translation of academic discovery into societal benefit: proposal for a balanced approach--part 1.
- A novel protective effect of erythropoietin in the infarcted heart.
- Screening for nitric oxide-dependent protein-protein interactions.
- Hemoglobin and nitric oxide.
- S-nitrosohemoglobin is distinguished from other nitrosovasodilators by unique oxygen-dependent responses that support an allosteric mechanism of action.
- Nitroxyl disulfides, novel intermediates in transnitrosation reactions.
- Reperfusion injury is reduced in skeletal muscle by inhibition of inducible nitric oxide synthase.
- Nitric oxide, NOC-12, and S-nitrosoglutathione modulate the skeletal muscle calcium release channel/ryanodine receptor by different mechanisms. An allosteric function ...
- Routes to S-nitroso-hemoglobin formation with heme redox and preferential reactivity in the beta subunits.
- Nitric oxide in RBCs.
- Nitrosative stress-induced apoptosis through inhibition of NF-kappa B.
Schools
Mount Sinai School of Medicine
Brigham and Women39s Hospital
Doctors Specialties
- Pulmonology
Accepted Insurances
Awards
- Investigator Alumnus
- Elected Member
Education
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Brigham and Women's Hospital -
Icahn School of Medicine at Mount Sinai
Hospital
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University Hospitals Case Medical Center -
Duke University Hospital
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1255415170 |
| Last Name Of The Provider | STAMLER |
| First Name Of The Provider | JONATHAN |
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