Professor, Medicine, Case Western Reserve University School of Medicine
Nephrology
Dr. Jeffrey R. Schelling, MD
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Jeffrey R. Schelling practices Nephrology. He graduated from Case Western Reserve University School of Medicine. Jeffrey Schelling is a published physician. He published 68 publications, including: 'Management of tumor lysis syndrome with standard continuous arteriovenous hemodialysis: case report and a review of the literature.' Jeffrey Schelling accepts Medicare.
Publications
- Mesangial cell αvβ8-integrin regulates glomerular capillary integrity and repair.
- Renal Function and Proteinuria after Successful Immunosuppressive Therapies in Patients With FSGS.
- APOL1 Genetic Variants in Focal Segmental Glomerulosclerosis and HIV-Associated Nephropathy.
- Phosphoinositide Binding Differentially Regulates NHE1 Na+/H+ Exchanger-Dependent Proximal Tubule Cell Survival.
- Use of diffusion tensor MRI to identify early changes in Diabetic nephropathy.
- Mesangial cell integrin αvβ8 provides glomerular endothelial cell cytoprotection by sequestering TGF-β and regulating PECAM-1.
- Ouabain stimulates Na-K-ATPase through a sodium/hydrogen exchanger-1 (NHE-1)-dependent mechanism in human Kidney proximal tubule cells.
- Albuminuria risk in Hispanic populations: not so black and white.
- Podocyte injury induces nuclear translocation of WTIP via microtubule-dependent transport.
- Evidence-based approach for prevention of radiocontrast-induced Nephropathy.
- Tissue transglutaminase inhibition as treatment for Diabetic glomerular scarring: it's good to be glueless.
- MYH9 is a major-effect risk gene for focal segmental glomeruloSclerosis.
- Regulation of cell survival by Na+/H+ exchanger-1.
- Risk factors for development and progression of diabetic Kidney disease and treatment patterns among diabetic siblings of patients with diabetic Kidney disease.
- NPHS2 variation in sporadic focal segmental glomeruloSclerosis.
- Beta8 integrin binds Rho GDP dissociation inhibitor-1 and activates Rac1 to inhibit mesangial cell myofibroblast differentiation.
- Mechanical force-activated phospholipase D is mediated by Galpha12/13-Rho and calmodulin-dependent kinase in Renal epithelial cells.
- Diabetes and the Kidney.
- WT1-interacting protein and ZO-1 translocate into podocyte nuclei after puromycin aminonucleoside treatment.
- Association of metabolic syndrome in nondiabetic patients with increased risk for chronic Kidney disease: the fat lady sings.
- A dipstick protein and specific gravity algorithm accurately predicts pathological proteinuria.
- Variants in the Wilms' tumor gene are associated With focal segmental glomerulosclerosis in the African American population.
- The metabolic syndrome as a risk factor for chronic Kidney disease: more than a fat chance?
- Renal phenotype is exacerbated in Os and lpr double mutant mice.
- The NHE1 Na+/H+ exchanger recruits ezrin/radixin/moesin proteins to regulate Akt-dependent cell survival.
- A WT1 co-regulator controls podocyte phenotype by shuttling between adhesion structures and nucleus.
- Linkage analysis of candidate loci for end-stage Renal disease due to diabetic nephropathy.
- Renal tubular epithelial cell apoptosis is associated With caspase cleavage of the NHE1 Na+/H+ exchanger.
- Serial analysis of gene expression.
- Fas activation induces Renal tubular epithelial cell beta 8 integrin expression and function in the absence of apoptosis.
- Approaches to understanding susceptibility to Nephropathy: from genetics to genomics.
- A Genome-Wide Search for Linkage of Estimated Glomerular Filtration Rate (eGFR) in the Family Investigation of Nephropathy and Diabetes (FIND).
- Apoptosis and JNK activation are differentially regulated by Fas expression level in Renal tubular epithelial cells.
- Activation of EphA receptor tyrosine kinase inhibits the Ras/MAPK pathway.
- A family-based strategy to identify genes for Diabetic nephropathy.
- Inaccuracy of clinical phenotyping parameters for Hypertensive nephrosclerosis.
- Serum C-peptide concentrations poorly phenotype type 2 diabetic end-stage Renal disease patients.
- Use of serial analysis of gene expression to generate Kidney expression libraries.
- Fatal hypermagnesemia.
- Involvement of Fas-dependent apoptosis in renal tubular epithelial cell deletion in chronic renal Failure.
- Hypoxia induces renal tubular epithelial cell apoptosis in Chronic renal disease.
- Genetic susceptibility to end-stage Renal disease.
- The IL-1 receptor and Rho directly associate to drive cell activation in inflammation.
- Interleukin-1 stimulates Jun N-terminal/stress-activated protein kinase by an arachidonate-dependent mechanism in mesangial cells.
- Management of tumor lysis syndrome With standard continuous arteriovenous hemodialysis: case report and a review of the literature.
- Fas-dependent fratricidal apoptosis is a mechanism of tubular epithelial cell deletion in chronic renal Failure.
- Angiotensin II activates the beta 1 isoform of phospholipase C in vascular smooth muscle cells.
- Annexin II inhibition of G protein-regulated inositol trisphosphate formation in rat aortic smooth muscle.
- Didanosine administration in a human immunodeficiency virus-positive Renal transplant patient.
- Angiotensin II-dependent proximal tubule sodium transport requires receptor-mediated endocytosis.
- Glucocorticoid uncoupling of antiogensin II-dependent phospholipase C activation in rat vascular smooth muscle cells.
- Angiotensin II-dependent proximal tubule sodium transport is mediated by cAMP modulation of phospholipase C.
- Increased osmolal gap in alcoholic ketoAcidosis and lactic Acidosis.
- Aldosterone enhances angiotensin II receptor binding and inositol phosphate Responses.
- Cytoskeleton-dependent endocytosis is required for apical type 1 angiotensin II receptor-mediated phospholipase C activation in cultured rat proximal tubule cells.
- HIV infection changes glomerular podocyte cytoskeletal composition and results in distinct cellular mechanical properties.
- The NHE1 Na+/H+ exchanger regulates cell survival by activating and targeting ezrin to specific plasma membrane domains.
- Nucleic acid-based techniques for post-transcriptional regulation of molecular targets.
- DNA expression analysis: serial analysis of gene expression, microarrays and Kidney disease.
- Kidney disease, genotype and the pathogenesis of vasculopathy.
- Genome-wide scans for diabetic nephropathy and albuminuria in multiethnic populations: the family investigation of nephropathy and Diabetes (FIND).
- Generation of Kidney transcriptomes using serial analysis of gene expression.
- MYH9 is associated With nondiabetic end-stage renal disease in African Americans.
- Heritability of the severity of Diabetic retinopathy: the FIND-Eye study.
- Genome-wide scan for estimated glomerular filtration rate in multi-ethnic diabetic populations: the Family Investigation of Nephropathy and Diabetes (FIND).
- Genome-wide linkage scans for type 2 Diabetes mellitus in four ethnically diverse populations-significant evidence for linkage on chromosome 4q in African Americans: t...
- Genomewide linkage scan for diabetic renal Failure and albuminuria: the FIND study.
Schools
Case Western Reserve University School of Medicine
Case West Res University Affil Hosps
University Colo School Med
Procedures Preformed
- Hemodialysis
- Peritoneal Dialysis
Conditions Treated
- Acidosis
- Acute Kidney Failure
- Calcium Metabolism Disorders
- Chronic Kidney Diseases
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Doctors Specialties
- Nephrology
Accepted Insurances
Education
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University of Colorado -
Case Western Reserve University - University Hospitals -
Case Western Reserve University School of Medicine
Hospital
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MetroHealth Medical Center
Drug Facts
| NPI NUMBER | 1154348613 | |
| NPPES Provider LastName | SCHELLING | |
| NPPES Provider FirstName | JEFFREY | |
| NPPES Provider ZIPCode | 441091900 | |
| NPPES Provider State | OH | |
| Specialty Description | Internal Medicine | |
| Total Claim Count | 137.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1154348613 |
| Last Name Of The Provider | SCHELLING |
| First Name Of The Provider | JEFFREY |
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