Physician
Emergency Medicine
Dr. James W. Beeson, MD
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James Beeson is an expert in the field of Emergency Medicine. He went to medical school at Indiana University School of Medicine. Indiana University School of Medicine is ranked 45 in research and 47 in primary care medicine James Beeson is a published physician. He has 82 publications published. The most current publication "2'-Fluoropyrimidine RNA-based aptamers to the 165-amino acid form of vascular endothelial growth factor (VEGF165). Inhibition of receptor binding and VEGF-induced vasc..."
Publications
- High Affinity Antibodies to Plasmodium falciparum Merozoite Antigens Are Associated with Protection from Malaria.
- Antibody to P. falciparum in Pregnancy Varies with Intermittent Preventive Treatment Regime and Bed Net Use.
- Discovery of GAMA, a Plasmodium falciparum merozoite micronemal protein, as a novel blood-stage vaccine candidate antigen.
- IgG against Plasmodium falciparum variant surface antigens and growth inhibitory antibodies in Mozambican children receiving intermittent preventive treatment with sul...
- Reticulocyte and erythrocyte binding-like proteins function cooperatively in invasion of human erythrocytes by malaria parasites.
- Placental malaria-associated inflammation disturbs the insulin-like growth factor axis of fetal growth regulation.
- Super-resolution dissection of coordinated events during malaria parasite invasion of the human erythrocyte.
- Quantifying the importance of MSP1-19 as a target of growth-inhibitory and protective antibodies against Plasmodium falciparum in humans.
- A phase 1 trial of MSP2-C1, a blood-stage malaria vaccine containing 2 isoforms of MSP2 formulated with Montanide® ISA 720.
- Antibodies to a full-length VAR2CSA immunogen are broadly strain-transcendent but do not cross-inhibit different placental-type parasite isolates.
- Intermittent preventive treatment to reduce the burden of malaria in children: new evidence on integration and delivery.
- Evidence that the erythrocyte invasion ligand PfRh2 is a target of protective immunity against Plasmodium falciparum malaria.
- Association between naturally acquired antibodies to erythrocyte-binding antigens of Plasmodium falciparum and protection from malaria and high-density parasitemia.
- Complement receptor 1 is the host erythrocyte receptor for Plasmodium falciparum PfRh4 invasion ligand.
- Minimal association of common red blood cell polymorphisms with Plasmodium falciparum infection and uncomplicated malaria in Papua New Guinean school children.
- Isolation of viable Plasmodium falciparum merozoites to define erythrocyte invasion events and advance vaccine and drug development.
- Development of fluorescent Plasmodium falciparum for in vitro growth inhibition assays.
- Interactions with heparin-like molecules during erythrocyte invasion by Plasmodium falciparum merozoites.
- Antibodies to chondroitin sulfate A-binding infected erythrocytes: dynamics and protection during pregnancy in women receiving intermittent preventive treatment.
- Immunization with VAR2CSA-DBL5 recombinant protein elicits broadly cross-reactive antibodies to placental Plasmodium falciparum-infected erythrocytes.
- Evaluation of the antigenic diversity of placenta-binding Plasmodium falciparum variants and the antibody repertoire among pregnant women.
- Using an improved phagocytosis assay to evaluate the effect of HIV on specific antibodies to pregnancy-associated malaria.
- The relationship between anti-merozoite antibodies and incidence of Plasmodium falciparum malaria: A systematic review and meta-analysis.
- Strain-specific duffy binding protein antibodies correlate with protection against infection with homologous compared to heterologous plasmodium vivax strains in Papua...
- The future for blood-stage vaccines against malaria.
- Cellular tumor necrosis factor, gamma interferon, and interleukin-6 responses as correlates of immunity and risk of clinical Plasmodium falciparum malaria in children ...
- Antibodies to reticulocyte binding protein-like homologue 4 inhibit invasion of Plasmodium falciparum into human erythrocytes.
- Sir2 paralogues cooperate to regulate virulence genes and antigenic variation in Plasmodium falciparum.
- Immunoglobulin G subclass-specific responses against Plasmodium falciparum merozoite antigens are associated with control of parasitemia and protection from symptomati...
- Reticulocyte-binding protein homologue 5 - an essential adhesin involved in invasion of human erythrocytes by Plasmodium falciparum.
- Analysis of structure and function of the giant protein Pf332 in Plasmodium falciparum.
- Recent insights into humoral and cellular immune responses against malaria.
- Exported proteins required for virulence and rigidity of Plasmodium falciparum-infected human erythrocytes.
- Characteristics of Plasmodium falciparum-infected-erythrocyte adhesion to chondroitin sulfate.
- Estimating the burden of global mortality in children aged <5 years by pathogen-specific causes.
- Polymorphic and conserved targets of antibodies against Plasmodium falciparum during pregnancy.
- Truncation of Plasmodium berghei merozoite surface protein 8 does not affect in vivo blood-stage development.
- Characterization of VAR2CSA-deficient Plasmodium falciparum-infected erythrocytes selected for adhesion to the BeWo placental cell line.
- Antibody-mediated growth inhibition of Plasmodium falciparum: relationship to age and protection from parasitemia in Kenyan children and adults.
- Acquisition of growth-inhibitory antibodies against blood-stage Plasmodium falciparum.
- Variation in use of erythrocyte invasion pathways by Plasmodium falciparum mediates evasion of human inhibitory antibodies.
- Towards a vaccine against Plasmodium vivax malaria.
- Serum lipoproteins promote efficient presentation of the malaria virulence protein PfEMP1 at the erythrocyte surface.
- Structural basis for binding of Plasmodium falciparum erythrocyte membrane protein 1 to chondroitin sulfate and placental tissue and the influence of protein polymorph...
- Antibodies among men and children to placental-binding Plasmodium falciparum-infected erythrocytes that express var2csa.
- The risk of malarial infections and disease in Papua New Guinean children.
- Skeleton-binding protein 1 functions at the parasitophorous vacuole membrane to traffic PfEMP1 to the Plasmodium falciparum-infected erythrocyte surface.
- Development and optimization of high-throughput methods to measure Plasmodium falciparum-specific growth inhibitory antibodies.
- Antigenic differences and conservation among placental Plasmodium falciparum-infected erythrocytes and acquisition of variant-specific and cross-reactive antibodies.
- A var gene promoter controls allelic exclusion of virulence genes in Plasmodium falciparum malaria.
- The immunology and pathogenesis of malaria during pregnancy.
- Targets of protective antibodies to malaria during pregnancy.
- Promising functional readouts of immunity in a blood-stage malaria vaccine trial.
- Placental malaria induces variant-specific antibodies of the cytophilic subtypes immunoglobulin G1 (IgG1) and IgG3 that correlate with adhesion inhibitory activity.
- An alternative agglutination assay to measure antibodies to variant surface antigens of Plasmodium falciparum-infected erythrocytes.
- Cross-reactive surface epitopes on chondroitin sulfate A-adherent Plasmodium falciparum-infected erythrocytes are associated with transcription of var2csa.
- Broad analysis reveals a consistent pattern of var gene transcription in Plasmodium falciparum repeatedly selected for a defined adhesion phenotype.
- Clinical features and pathogenesis of severe malaria.
- Malaria in pregnancy and the endemicity spectrum: what can we learn?
- Impairment of humoral immunity to Plasmodium falciparum malaria in pregnancy by HIV infection.
- Phenotypes of Plasmodium falciparum from the peripheral blood of pregnant women.
- Antibodies to variant surface antigens of Plasmodium falciparum-infected erythrocytes and adhesion inhibitory antibodies are associated with placental malaria and have...
- Plasmodium falciparum-infected erythrocytes demonstrate dual specificity for adhesion to hyaluronic acid and chondroitin sulfate A and have distinct adhesive properties.
- Selective accumulation of mature asexual stages of Plasmodium falciparum-infected erythrocytes in the placenta.
- Evaluating specific adhesion of Plasmodium falciparum-infected erythrocytes to immobilised hyaluronic acid with comparison to binding of mammalian cells.
- The structural motif in chondroitin sulfate for adhesion of Plasmodium falciparum-infected erythrocytes comprises disaccharide units of 4-O-sulfated and non-sulfated N...
- Expanding the paradigms of placental malaria.
- Pathogenesis of Plasmodium falciparum malaria: the roles of parasite adhesion and antigenic variation.
- Parasite adhesion and immune evasion in placental malaria.
- New agents to combat malaria.
- Sticky rings: challenging the cytoadherence dogma.
- Inhibition of adhesion of Plasmodium falciparum-infected erythrocytes by structurally defined hyaluronic acid dodecasaccharides.
- Identification of glycosaminoglycan binding domains in Plasmodium falciparum erythrocyte membrane protein 1 of a chondroitin sulfate A-adherent parasite.
- Adhesion of Plasmodium falciparum-infected erythrocytes to hyaluronic acid in placental malaria.
- Plasmodium falciparum rosette formation is uncommon in isolates from pregnant women.
- The placenta in malaria: mechanisms of infection, disease and foetal morbidity.
- Plasmodium falciparum isolates from infected pregnant women and children are associated with distinct adhesive and antigenic properties.
- The adhesion of Plasmodium falciparum-infected erythrocytes to chondroitin sulfate A is mediated by P. falciparum erythrocyte membrane protein 1.
- 2'-Fluoropyrimidine RNA-based aptamers to the 165-amino acid form of vascular endothelial growth factor (VEGF165). Inhibition of receptor binding and VEGF-induced vasc...
- Inhibition of binding of malaria-infected erythrocytes by a tetradecasaccharide fraction from chondroitin sulfate A.
- Differential distribution of amyloid protein precursor immunoreactivity in the rat brain studied by using five different antibodies.
Schools
Indiana University School of Medicine
Doctors Specialties
- Emergency Medicine
Accepted Insurances
- Aetna
- Cofinity PPO
- Commercial Insurance Company
- Emblem Health
- GEHA
- Medicare Advantage PFFS
- HealthSmart
- Humana
- Medicaid
- Medicare MCR
- Choice Plus POS
- Navigate HMO
- Navigate POS
- Options PPO
- United Healthcare
Education
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Indiana University School of Medicine
Drug Facts
| NPI NUMBER | 1700881687 | |
| NPPES Provider LastName | BEESON | |
| NPPES Provider FirstName | JAMES | |
| NPPES Provider ZIPCode | 460164337 | |
| NPPES Provider State | IN | |
| Specialty Description | Emergency Medicine | |
| Total Claim Count | 544.0 | |
| Distinct Opioid Count | 1.0 | |
| Opioid Claim Count | 117.0 | |
| Percent Opioid Claims | 21.51 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1700881687 |
| Last Name Of The Provider | BEESON |
| First Name Of The Provider | JAMES |
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