Professor of Medicine, Brigham and Women's Hospital
Cardiology
Dr. James K. Liao, MD
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James Liao works in the field of Cardiology. James K. Liao, MD is a published doctor. He published 98 publications, including: 'Abrogation of Airway Hyperresponsiveness but not Inflammation by Rho kinase Insufficiency.' Dr. James K. Liao accepts Medicare payments and is listed with Medicare.gov.
Publications
- Abrogation of Airway Hyperresponsiveness but not Inflammation by Rho kinase Insufficiency.
- Metabolic regulation of cancer cell side population by glucose through activation of the Akt pathway.
- Rock1 & 2 perform overlapping and unique roles in angiogenesis and angiosarcoma tumor progression.
- Folliculin, the Product of the Birt-Hogg-Dube Tumor Suppressor Gene, Interacts with the Adherens Junction Protein p0071 to Regulate Cell-Cell Adhesion.
- A mouse model of diet-induced obesity and insulin resistance.
- Mitohormesis: another pleiotropic effect of statins?
- ROCK inhibition prevents fetal serum-induced alteration in structure and function of organ-cultured mesenteric Artery.
- Squalene synthase inhibitor lapaquistat acetate: could anything be better than statins?
- Pitavastatin-induced angiogenesis and arteriogenesis is mediated by Notch1 in a murine hindlimb ischemia model without induction of VEGF.
- Striatal neurons expressing full-length mutant huntingtin exhibit decreased N-cadherin and altered neuritogenesis.
- Notch1 in bone marrow-derived cells mediates cardiac repair after Myocardial infarction.
- Fingolimod provides long-term protection in rodent models of cerebral ischemia.
- NF-κB and innate immunity in ischemic stroke.
- Phosphorylation of IRF4 by ROCK2 regulates IL-17 and IL-21 production and the development of autoimmunity in mice.
- RhoA/ROCK signaling is essential for multiple aspects of VEGF-mediated angiogenesis.
- Noninvasive method for assessing the human circadian clock using hair follicle cells.
- Notch signaling regulates endothelial progenitor cell activity during recovery from arterial injury in hypercholesterolemic mice.
- Rac1 and connective tissue growth factor. The missing link between Atrial remodeling and the pathogenesis of Atrial fibrillation?
- Genetically elevated C-reactive protein and ischemic vascular Disease.
- gamma-Secretase inhibitor reduces diet-induced atherosclerosis in apolipoprotein E-deficient mice.
- Smooth muscle Notch1 mediates neointimal formation after vascular injury.
- Comparison of effects of rosuvastatin (10 mg) versus atorvastatin (40 mg) on rho kinase activity in caucasian men with a previous atherosclerotic event.
- Evidence for statin pleiotropy in humans: differential effects of statins and ezetimibe on rho-associated coiled-coil containing protein kinase activity, endothelial f...
- Rac1 is a critical mediator of endothelium-derived neurotrophic activity.
- Obesity increases vascular senescence and susceptibility to ischemic injury through Chronic activation of Akt and mTOR.
- Increased vascular senescence and impaired endothelial progenitor cell function mediated by mutation of circadian gene Per2.
- Deficiency of ROCK1 in bone marrow-derived cells protects against atherosclerosis in LDLR-/- mice.
- Tumor necrosis factor-alpha downregulates endothelial nitric oxide synthase mRNA stability via translation elongation factor 1-alpha 1.
- Regulation of endothelial nitric oxide synthase and postnatal angiogenesis by Rac1.
- Effect of torcetrapib in patients at high risk for Coronary events.
- HDL cholesterol, very low levels of LDL cholesterol, and cardiovascular events.
- Fine-tuning the angiogenic response to vascular endothelial growth factor.
- Additive effects of statin and dipyridamole on cerebral blood flow and stroke protection.
- ROCK1 mediates leukocyte recruitment and neointima formation following vascular injury.
- Dynamic regulation of endothelial NOS mediated by competitive interaction with alpha-actinin-4 and calmodulin.
- Linking Notch signaling to ischemic stroke.
- Translational therapeutics of dipyridamole.
- Identification of ROCK1 as an upstream activator of the JIP-3 to JNK signaling axis in response to UVB damage.
- A method for measuring Rho kinase activity in tissues and cells.
- Does it matter whether or not a lipid-lowering agent inhibits Rho kinase?
- Primary prevention of cardiovascular Disease with pravastatin.
- Effect of rosiglitazone on the frequency of diabetes.
- Effect of ramipril on the incidence of diabetes.
- Rho kinase (ROCK) inhibitors.
- Inhibition of apoptosis-regulated signaling kinase-1 and prevention of congestive Heart failure by estrogen.
- Rho-kinase inhibition Acutely augments blood flow in focal cerebral ischemia via endothelial mechanisms.
- Increased Rho kinase activity in a Taiwanese population with metabolic Syndrome.
- Phosphatidylinositol 3-kinase/protein kinase Akt negatively regulates plasminogen activator inhibitor type 1 expression in vascular endothelial cells.
- Secondary prevention of stroke and transient ischemic Attack: is more platelet inhibition the answer?
- Roles of rho-associated kinase and oxidative stress in the pathogenesis of Aortic stiffness.
- Safety and efficacy of statins in Asians.
- Critical role of endothelial Notch1 signaling in postnatal angiogenesis.
- Rho kinase inhibition improves endothelial function in human subjects with Coronary artery disease.
- Rapid nongenomic actions of thyroid hormone.
- FHL2/SLIM3 decreases cardiomyocyte survival by inhibitory interaction with sphingosine kinase-1.
- Rho-kinase: a potential link between hypercholesterolemia and abnormal vascular smooth muscle contraction.
- Absence of TRAM restricts Toll-like receptor 4 signaling in vascular endothelial cells to the MyD88 pathway.
- Requirement of Rac1 in the development of cardiac hypertrophy.
- Transcription factor CHF1/Hey2 suppresses cardiac hypertrophy through an inhibitory interaction with GATA4.
- Physiological role of ROCKs in the cardiovascular system.
- Decreased vascular lesion formation in mice with inducible endothelial-specific expression of protein kinase Akt.
- Rho GTPases, statins, and nitric oxide.
- Decreased perivascular fibrosis but not cardiac hypertrophy in ROCK1+/- haploinsufficient mice.
- Inhibition of Rho kinase (ROCK) leads to increased cerebral blood flow and stroke protection.
- Effects of statins on 3-hydroxy-3-methylglutaryl coenzyme a reductase inhibition beyond low-density lipoprotein cholesterol.
- Rho-kinase mediates hyperglycemia-induced plasminogen activator inhibitor-1 expression in vascular endothelial cells.
- Protective effects of statins involving both eNOS and tPA in focal cerebral ischemia.
- Essential role of endothelial Notch1 in angiogenesis.
- Vascular protective effects of cytochrome p450 epoxygenase-derived eicosanoids.
- Pleiotropic effects of statins.
- Induction of angiogenesis by heat shock protein 90 mediated by protein kinase Akt and endothelial nitric oxide synthase.
- Statins and the myocardium.
- Risk of Coronary artery disease associated with polymorphism of the cytochrome P450 epoxygenase CYP2J2.
- Inhibition of Rho-kinase leads to rapid activation of phosphatidylinositol 3-kinase/protein kinase Akt and cardiovascular protection.
- Atorvastatin restores endothelial function in normocholesterolemic smokers independent of changes in low-density lipoprotein.
- Statin therapy is associated with improved patency of autogenous infrainguinal bypass grafts.
- Statin therapy: having the good without the bad.
- Simvastatin preserves cardiac function in genetically determined Cardiomyopathy.
- Isoprenoid metabolism and the pleiotropic effects of statins.
- Long-term statin use and psychological well-being.
- Short-term statin therapy improves cardiac function and symptoms in patients with idiopathic dilated Cardiomyopathy.
- Activation of the phosphatidylinositol 3-kinase/protein kinase Akt pathway mediates nitric oxide-induced endothelial cell migration and angiogenesis.
- Endothelium-dependent effects of statins.
- Acute reduction of myocardial infarct size by a hydroxymethyl glutaryl coenzyme A reductase inhibitor is mediated by endothelial nitric oxide synthase.
- Rapid effects of statins: from prophylaxis to therapy for ischemic stroke.
- Molecular basis of cell membrane estrogen receptor interaction with phosphatidylinositol 3-kinase in endothelial cells.
- Vascular protection by estrogen in ischemia-reperfusion injury requires endothelial nitric oxide synthase.
- Ventricular septal defect and Cardiomyopathy in mice lacking the transcription factor CHF1/Hey2.
- Nonnuclear actions of estrogen.
- Rapid nontranscriptional activation of endothelial nitric oxide synthase mediates increased cerebral blood flow and stroke protection by corticosteroids.
- Novel non-transcriptional mechanisms for estrogen receptor signaling in the cardiovascular system. Interaction of estrogen receptor alpha with phosphatidylinositol 3-O...
- Functional interaction of endothelial nitric oxide synthase with a voltage-dependent anion channel.
- Isoprenoids as mediators of the biological effects of statins.
- Inhibition of major histocompatibility complex class II gene transcription by nitric oxide and antioxidants.
- 1alpha,25-dihydroxyvitamin D3 induces vascular smooth muscle cell migration via activation of phosphatidylinositol 3-kinase.
- Rho-kinase mediates hypoxia-induced downregulation of endothelial nitric oxide synthase.
- Non-nuclear actions of estrogen: new targets for prevention and treatment of cardiovascular Disease.
Schools
University of California At San Francisco
Brigham and Women39s Hospital
Massachusetts General Hospital
Procedures Preformed
- Cardiac Catheterization (incl. Coronary Angiography)
- Cardiac Imaging
- Cardiac MRI (Magnetic Resonance Imaging) of Heart or Chest
- Cardiac Myocardial Perfusion Imaging
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Conditions Treated
- Angina and Acute Coronary Syndrome
- Aortic Dissection
- Aortic Valve Disease
- Arrhythmias (incl. Atrial Fibrillation)
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Doctors Specialties
- Cardiology
Accepted Insurances
Hospital
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Brigham and Women's Hospital
Drug Facts
| NPI NUMBER | 1689687873 | |
| NPPES Provider LastName | LIAO | |
| NPPES Provider FirstName | JAMES | |
| NPPES Provider ZIPCode | 606371641 | |
| NPPES Provider State | IL | |
| Specialty Description | Cardiology | |
| Total Claim Count | 39.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1689687873 |
| Last Name Of The Provider | LIAO |
| First Name Of The Provider | JAMES |
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