Assistant Professor, Medicine, Case Western Reserve University School of Medicine
Internal Medicine
Dr. Ismail A. Dreshaj, MD
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Ismail Dreshaj practices Internal Medicine. Ismail Dreshaj is also a published doctor. He has 55 paper published. The most recent paper is: Selective hypoxic loading of the ventrolateral medulla inhibits cholinergic outflow to the airways. Ismail Dreshaj is registered with Medicare.
Publications
- Role of brain-derived neurotrophic factor in hyperoxia-induced enhancement of contractility and impairment of relaxation in lung parenchyma.
- Disruption of NO-cGMP signaling by neonatal hyperoxia impairs relaxation of lung parenchyma.
- The basomedial hypothalamus modulates the ventilatory response to hypoxia in neonatal rats.
- Inhibition of Na(+)/H(+) exchanger type 3 reduces duration of apnea induced by laryngeal stimulation in piglets.
- Endogenous nitric oxide modulates responses of tissue and airway resistance to vagal stimulation in piglets.
- Activation of the midbrain periaqueductal gray induces airway smooth muscle relaxation.
- Effects of hypoxia on respiratory neural output and lower esophageal sphincter pressure in piglets.
- Primary hydatid cyst of the gallbladder: a case report.
- An astrocyte toxin influences the pattern of breathing and the ventilatory response to hypercapnia in neonatal rats.
- Selective antihypertensive action of moxonidine is mediated mainly by I1-imidazoline receptors in the rostral ventrolateral medulla.
- Central chemosensitivity affects respiratory muscle responses to laryngeal stimulation in the piglet.
- Ontogeny of neurokinin-1 receptors in the porcine respiratory system.
- Circulatory heat sources for canine respiratory heat exchange.
- Effect of exposure to green coffee dust extract on airway reactivity.
- Partitioning of pulmonary resistance during constriction in the dog: effects of volume history.
- A novel mechanism of action for hypertension control: moxonidine as a selective I1-imidazoline agonist.
- Ventilatory behavior after hypoxia in C57BL/6J and A/J mice.
- Nitric oxide deficiency contributes to impairment of airway relaxation in cystic fibrosis mice.
- Differential effects of hypercapnia on expiratory phases of respiration in the piglet.
- Central GABAergic mechanisms are involved in apnea induced by SLN stimulation in piglets.
- The excitatory amino acid glutamate mediates reflexly increased tracheal blood flow and airway submucosal gland secretion.
- Recurrent hypoxic exposure and reflex responses during development in the piglet.
- Role of endogenous nitric oxide in hyperoxia-induced airway hyperreactivity in maturing rats.
- Differential cardiorespiratory control elicited by activation of ventral medullary sites in mice.
- The alpha3 subtype of the nicotinic acetylcholine receptor is expressed in airway-related neurons of the nucleus tractus solitarius, but is not essential for reflex br...
- alpha(2)-adrenergic receptors are not required for central anti-hypertensive action of moxonidine in mice.
- Involvement of glutamate in transmission of afferent constrictive inputs from the airways to the nucleus tractus solitarius in ferrets.
- Maturation of the cholinergic response of tracheal smooth muscle in the piglet.
- Effect of I1-imidazoline receptor activation on responses of hypoglossal and phrenic nerve to chemical stimulation.
- Central effects of endothelin on respiratory output during development.
- [The effect of Ca2+ blockers on the response of respiratory tract smooth muscle to bronchoconstrictor and bronchodilator agents].
- [Normal values of the diffusing capacity of the lungs in adults].
- [Effect of ethanol on tone of isolated smooth muscle of the the pulmonary artery].
- CO2-induced c-fos expression in medullary neurons during early development.
- CO2-induced prolongation of expiratory time during early development.
- Changes in respiratory timing induced by hypercapnia in maturing rats.
- Moxonidine acting centrally inhibits airway reflex responses.
- Selective hypoxic loading of the ventrolateral medulla inhibits cholinergic outflow to the airways.
- Mechanism for substance P-induced relaxation of precontracted airway smooth muscle during development.
- Interleukin-12 suppresses filaria-induced pulmonary eosinophilia, deposition of major basic protein and airway hyperresponsiveness.
- I1-imidazoline receptors and cholinergic outflow to the airways.
- The role of the medullary raphe nuclei in regulation of cholinergic outflow to the airways.
- Role of the medullary raphe nuclei in the respiratory response to CO2.
- The role of excitatory amino acids in airway reflex responses in anesthetized dogs.
- Role of endogenous NO in modulating airway contraction mediated by muscarinic receptors during development.
- Effect of hyperoxia on substance P expression and airway reactivity in the developing lung.
- Spiral nerve cuff electrode for recordings of respiratory output.
- Effect of exogenous and endogenous nitric oxide on the airway and tissue components of lung resistance in the newborn piglet.
- Maturational changes in responses of tissue and airway resistance to histamine.
- Nitric oxide and ventilatory response to hypoxia.
- Neurochemical control of tissue resistance in piglets.
- Hypoglossal and phrenic responses to central respiratory inhibition in piglets.
- Responses of lung parenchyma and airways to tachykinin peptides in piglets.
- A comparison of three single layer anastomotic techniques in the colon of the rat.
Doctors Specialties
- Internal Medicine
Accepted Insurances
Education
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University of Pristina Faculty of Medicine
Hospital
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University Hospitals Case Medical Center
Drug Facts
| NPI NUMBER | 1295751568 | |
| NPPES Provider LastName | DRESHAJ | |
| NPPES Provider FirstName | ISMAIL | |
| NPPES Provider ZIPCode | 441061716 | |
| NPPES Provider State | OH | |
| Specialty Description | Internal Medicine | |
| Total Claim Count | 14.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1295751568 |
| Last Name Of The Provider | DRESHAJ |
| First Name Of The Provider | ISMAIL |
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