Director; Associate Professor, Medicine, Endocrinology; Medicine, Albert Einstein College of Medicine
Endocrinology
Dr. Irwin J. Kurland, MD
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Irwin Kurland is an expert in the field of Endocrinology. He attended Keck School of Medicine of USC. Keck School of Medicine of USC has a rank of 31 in research and a 72 in primary care Irwin Kurland is a published physician. He published 43 publications, including: 'Regulation of rat 6-phosphofructo-2-kinase/fructose-2,6-bisphosphatase. Role of the NH2-terminal region.'
Publications
- Intestinal microbiota-derived metabolomic blood plasma markers for prior radiation injury.
- The brighter (and evolutionarily older) face of the metabolic syndrome: evidence from Trypanosoma cruzi infection in CD-1 mice.
- Role of the tumor suppressor IQGAP2 in metabolic homeostasis: Possible link between diabetes and cancer.
- Advanced mass spectrometry-based multi-omics technologies for exploring the pathogenesis of hepatocellular carcinoma.
- Glucose-Induced Metabolic Memory in Schwann Cells: Prevention by PPAR Agonists.
- Application of combined omics platforms to accelerate biomedical discovery in diabesity.
- Peripheral Effects of FAAH Deficiency on Fuel and Energy Homeostasis: Role of Dysregulated Lysine Acetylation.
- UCP2 regulates energy metabolism and differentiation potential of human pluripotent stem cells.
- The Fasted/Fed Mouse Metabolic Acetylome : N6-Acetylation Differences Suggest Acetylation Coordinates Organ-Specific Fuel Switching.
- Advantages of tandem LC-MS for the rapid assessment of tissue-specific metabolic complexity using a pentafluorophenylpropyl stationary phase.
- Enhanced energy expenditure, glucose utilization, and insulin sensitivity in VAMP8 null mice.
- Advantages of dynamic "closed loop" stable isotope flux phenotyping over static "open loop" clamps in detecting silent genetic and dietary phenotypes.
- Fyn-dependent regulation of energy expenditure and body weight is mediated by tyrosine phosphorylation of LKB1.
- Insulin resistance in striated muscle-specific integrin receptor beta1-deficient mice.
- Integrative metabolic regulation of peripheral tissue fatty acid oxidation by the SRC kinase family member Fyn.
- Decreased hepatic futile cycling compensates for increased glucose disposal in the Pten heterodeficient mouse.
- Lipin deficiency impairs diurnal metabolic fuel switching.
- NR4A orphan nuclear receptors are transcriptional regulators of hepatic glucose metabolism.
- Role of endosomal trafficking dynamics on the regulation of hepatic insulin receptor activity: models for Fao cells.
- Peroxisomal proliferator-activated receptor alpha deficiency diminishes insulin-responsiveness of gluconeogenic/glycolytic/pentose gene expression and substrate cycle ...
- Determination of a glucose-dependent futile recycling rate constant from an intraperitoneal glucose tolerance test.
- Peroxisome proliferator-activated receptor alpha (PPARalpha) influences substrate utilization for hepatic glucose production.
- Integrated control of hepatic lipogenesis versus glucose production requires FoxO transcription factors.
- The histone deacetylase Sirt6 regulates glucose homeostasis via Hif1alpha.
- PIM3 proto-oncogene kinase is a common transcriptional target of divergent EWS/ETS oncoproteins.
- Expression of mammalian liver glycolytic/gluconeogenic enzymes in Escherichia coli: recovery of active enzyme is strain and temperature dependent.
- Evolutionary reengineering of the phosphofructokinase active site: ARG-104 does not stabilize the transition state in 6-phosphofructo-2-kinase.
- Loss of [13C]glycerol carbon via the pentose cycle. Implications for gluconeogenesis measurement by mass isotoper distribution analysis.
- N- and C-termini modulate the effects of pH and phosphorylation on hepatic 6-phosphofructo-2-kinase/fructose-2,6-bisphosphatase.
- Regulation of rat 6-phosphofructo-2-kinase/fructose-2,6-bisphosphatase. Role of the NH2-terminal region.
- Lack of evidence for a role of Cys-138 as a base catalyst in the skeletal muscle 6-phosphofructo-2-kinase reaction.
- Evidence for NH2- and COOH-terminal interactions in rat 6-phosphofructo-2-kinase/fructose-2,6-bisphosphatase.
- Compensation in pancreatic beta-cell function in subjects with glucokinase mutations.
- 6-Phosphofructo-2-kinase/fructose-2,6-bisphosphatase: a metabolic signaling enzyme.
- Covalent control of 6-phosphofructo-2-kinase/fructose-2,6-bisphosphatase: insights into autoregulation of a bifunctional enzyme.
- A minimal model of liver glycogen metabolism; feasibility for predicting flux rates.
- Indirect versus direct routes of hepatic glycogen synthesis.
- Rat liver 6-phosphofructo-2-kinase/fructose-2,6-bisphosphatase. Properties of phospho- and dephospho- forms and of two mutants in which Ser32 has been changed by site-...
- Site-directed mutagenesis in rat liver 6-phosphofructo-2-kinase. Mutation at the fructose 6-phosphate binding site affects phosphate activation.
- Birefringent prism couplers for thin-film optical waveguides.
- MKR mice have increased dynamic glucose disposal despite metabolic inflexibility, and hepatic and peripheral insulin insensitivity.
- Isotopic Ratio Outlier Analysis of the S. cerevisiae Metabolome Using Accurate Mass Gas Chromatography/Time-of-Flight Mass Spectrometry: A New Method for Discovery.
Schools
University of Southern California Keck School of Medicine
University Cincinnati
SUNY
Doctors Specialties
- Endocrinology
Accepted Insurances
Education
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SUNY Stony Brook University Hospital & Medical Center -
University Hospital - University of Cincinnati College of Medicine -
Keck School of Medicine of USC
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