Research Fellow in Surgery, Massachusetts General Hospital
General Surgery
Dr. Hugh G. Auchincloss, MD
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Hugh G. Auchincloss, MD practices General Surgery. He researched medicine at Harvard Medical School. Harvard Medical School is a top medical college. Harvard Medical School is ranked 1 in research and 12 in primary care medicine Hugh Auchincloss is a published doctor as well. He has 86 publications published. The latest was: Literature update 1998, part 2.
Publications
- Massachusetts Health Care Reform and Reduced Racial Disparities in Minimally Invasive Surgery.
- Tracheal disruption from invasive pretracheal lymphadenopathy.
- Prolonged Survival of Allogeneic Islets in Cynomolgus Monkeys After Short-Term Triple Therapy.
- Spontaneously hypertensive and Wistar Kyoto rats are genetically disparate.
- Hard graft? Future challenges in transplantation.
- Liver transplantation for primary hepatic cancer.
- A surgeon views the patients options of the treatment of carcinoma of the breast.
- Transplantation of pancreatic islets in diabetic nonhuman primates.
- Mechanisms of tolerance to allografts.
- Mechanisms of transplantation tolerance.
- A phase I trial of immunosuppression with anti-ICAM-1 (CD54) mAb in renal allograft recipients.
- Indirect recognition by helper cells can induce donor-specific cytotoxic T lymphocytes in vivo.
- Management of the renal allograft recipient: immunosuppressive protocols for long-term success.
- Second renal transplantations. Ethical issues clarified by outcome; outcome enhanced by a reliable crossmatch.
- Evidence that a "four-cell cluster" may prime cytotoxic T-cells during graft rejection.
- Class II-positive hematopoietic cells cannot mediate positive selection of CD4+ T lymphocytes in class II-deficient mice.
- The role of "indirect" recognition in initiating rejection of skin grafts from major histocompatibility complex class II-deficient mice.
- Mice lacking major histocompatibility complex class I and class II molecules.
- T cell recognition of xeno-MHC peptides during concordant xenograft rejection.
- Two levels of help for B cell alloantibody production.
- Selective use of veno-venous bypass in orthotopic liver transplantation.
- Antigen processing and presentation in transplantation.
- Studies of transplantation immunology with major histocompatibility complex knockout mice.
- CD8+ effector cells responding to residual class I antigens, with help from CD4+ cells stimulated indirectly, cause rejection of "major histocompatibility complex-defi...
- Contribution of native kidney function to total glomerular filtration rate after combined kidney-pancreas transplantation.
- Delayed rejection of soluble tumor necrosis factor receptor-secreting tumor allografts.
- The strength of cell-mediated xenograft rejection in the mouse is due to the CD4+ indirect response.
- Literature update 1997, part 3.
- Xenogeneic transplantation.
- Literature update 1998, Part 1.
- Literature update 1998, part 2.
- Direct and indirect recognition: the role of MHC antigens in graft rejection.
- Literature update 1998, part 3.
- Literature update 1998, part 4.
- Literature update 1999, part 1.
- Report of the Food and Drug Administration Subcommittee on Xenotransplantation: meeting of 3 and 4 June, 1999, Center for Biologics Evaluation and Research.
- Literature update 1999, part 2.
- The role of CD154-CD40 versus CD28-B7 costimulatory pathways in regulating allogeneic Th1 and Th2 responses in vivo.
- Literature update 1999, part 3. Xenotransplantation.
- Literature update 2000, part 1.
- Two distinct murine differentiation antigens determined by genes linked to the Ly-6 locus.
- MHC class I expression and CD8+ T cell development in TAP1/beta 2-microglobulin double mutant mice.
- Xenotransplantation.
- Effector cells must recognize antigens expressed in the graft to cause efficient skin graft rejection in SCID mice.
- Role of graft in transplantation tolerance.
- Cell-mediated xenoresponses: strong or weak?
- Trimethoprim-sulfamethoxazole compared with ciprofloxacin for the prevention of urinary tract infection in renal transplant recipients. A double-blind, randomized cont...
- Evidence that multiple defects in cell-surface molecule interactions across species differences are responsible for diminished xenogeneic T cell responses.
- Cellular mechanisms of rejection.
- Comparison of GK1.5 and chimeric rat/mouse GK1.5 anti-CD4 antibodies for prolongation of skin allograft survival and suppression of alloantibody production in mice.
- Skin graft rejection by beta 2-microglobulin-deficient mice.
- The high-risk liver allograft recipient. Should allocation policy consider outcome?
- A monoclonal antibody detecting unusual Thy-1 determinants.
- Anti-idiotypes against anti-H-2 antibodies VI. Detection of shared idiotypes among monoclonal anti-H-2 antibodies.
- Antiidiotypes against anti-H-2 monoclonal antibodies. V. In vivo antiidiotype treatment induces idiotype-specific helper T cells.
- Mechanisms of tolerance in murine radiation bone marrow chimeras. I. Nonspecific suppression of alloreactivity by spleen cells from early, but not late, chimeras.
- Immunologic characterization of MHC recombinant swine: role of class I and II antigens in in vitro immune responses.
- Effect of class II antigen matching on renal allograft survival in miniature swine.
- Mechanisms of tolerance in murine radiation bone marrow chimeras. II. Absence of nonspecific suppression in mature chimeras.
- Anti-idiotypes against anti-H-2 monoclonal antibodies: structural analysis of the molecules induced by in vivo anti-idiotype treatment.
- Anti-idiotypes to monoclonal anti-H-2 antibodies. III. Syngeneic anti-idiotypes on antibodies induced by in vivo administration of xenogeneic anti-idiotypes.
- Long-term metabolic and quality of life results with pancreatic/renal transplantation in insulin-dependent diabetes mellitus.
- Contributions by Paul S. Russell toward the future application of xenografting.
- Rejection of transgenic skin expressing a xeno-class I antigen is CD4-dependent and CD8-independent.
- Xenograft rejection of class I-expressing transgenic skin is CD4-dependent and CD8-independent.
- Xenogeneic proliferation and lymphokine production are dependent on CD4+ helper T cells and self antigen-presenting cells in the mouse.
- Long-term, low-dose cyclosporine treatment of renal allograft recipients. A randomized trial.
- Xenogeneic skin graft rejection is especially dependent on CD4+ T cells.
- Murine CD8+ T cell helper function is particularly sensitive to cyclosporine suppression in vivo.
- Successful treatment of invasive fungal infection with fluconazole in organ transplant recipients.
- CD4+ lymphocytes play a dominant role in murine xenograft rejection.
- Immunosuppression by cyclosporine in vivo need not block effector T-cell function in mice.
- Procurement of a whole pancreas and liver from the same cadaveric donor.
- T-cell subsets, bm mutants, and the mechanisms of allogeneic skin graft rejection.
- Simultaneous procurement of pancreas and liver from a single cadaveric donor.
- In vivo use of monoclonal antibodies against murine T cell antigens.
- Failure to induce chimerism or tolerance in mice with monoclonal anti-T cell antibodies plus allogeneic bone marrow infusion.
- Combined kidney and pancreas transplantation in diabetics.
- Effects of in vivo administration of anti-T3 monoclonal antibody on T cell function in mice. I. Immunosuppression of transplantation responses.
- Prevention of alloantibody formation after skin grafting without prolongation of graft survival by anti-L3T4 in vivo.
- Xenogeneic transplantation. A review.
- Ureteropyelostomy with a monofilament absorbable suture.
- The selective use of antilymphocyte serum for cyclosporine treated patients with renal allograft dysfunction.
- Defects in accessory molecule and other cell surface molecule interactions are responsible for weak mouse helper T-cell responses to xenoantigens.
- Symptomatic cytomegalovirus disease in the cytomegalovirus antibody seropositive renal transplant recipient treated with OKT3.
Schools
Harvard Medical School
Doctors Specialties
- General Surgery
- Other
Accepted Insurances
Education
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Massachusetts General Hospital -
Harvard Medical School
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