Professor, Medicine, University of Colorado School of Medicine
Allergy & Immunology
Dr. Henry N. Claman, MD
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Henry Claman works in the field of Allergy & Immunology. He graduated from NYU School of Medicine. Doctor Henry N. Claman, MD is a published MD. He published 100 publications, including: 'Diphtheria toxin A gene-mediated HIV-1 protection of cord blood-derived T cells in the SCID-hu mouse model.'
Publications
- David w. Talmage, 1919-2014.
- Restoration of suppressed immunity in burned mice with cimetidine.
- Experimental production of granulomatous pneumonitis. Comparison of immunological and morphological sequelae with particulate and soluble antigens administered via the...
- Characterization of the immunosuppressive effect of burned tissue in an animal model.
- Diphtheria toxin A gene-mediated HIV-1 protection of cord blood-derived T cells in the SCID-hu mouse model.
- To our students. Medicine and the two brains.
- Dyspnea and pulmonary infiltrates in a 53-year-old woman with common variable immunodeficiency.
- Creativity and illness: Christopher Smart and Samuel Johnson.
- Delayed Hypersensitivity.
- Minor antigen graft-versus-host reactions revealed in irradiated spleen and popliteal lymph node assays.
- Unresponsiveness in hapten-specific cytotoxic lymphocytes. III. Influence of H-2 and non-H-2 gene loci on the in vitro trinitrophenyl-specific CTL response following e...
- The induction of alloreactive cytotoxic responses in vivo are inhibited by pretreatment with M is-disparate cells.
- Induction of tolerance to 1-fluoro-2,4-dinitrobenzene contact sensitivity with hapten-modified lymphoid cells. II. Selective tolerance in F1 mice of T cell subsets rec...
- Unresponsiveness in hapten-specific cytotoxic lymphocytes. II. Ability of various TNP-congeners to induce unresponsiveness after repeated treatments beginning at birth.
- T cell signals in tolerance.
- Vasculitis in chronic urticaria.
- Control of experimental contact sensitivity.
- The induction of cell-mediated immunity and tolerance to insulin with insulin coupled to syngeneic lymphoid cells.
- Amyloid-associated muscle pseudohypertrophy.
- The induction of tolerance to DNFB contact sensitivity using hapten modified lymphoid cells. I. Cellular requirements for rapid induction of tolerance.
- Transplantation of enriched CD34-positive autologous marrow into breast cancer patients following high-dose chemotherapy: influence of CD34-positive peripheral-blood p...
- Increased ICAM-1 expression in the early stages of murine chronic graft-versus-host disease.
- Antinuclear antibodies and breast implants.
- Mast cell immunohistochemistry: non-immunological immunostaining mediated by non-specific F(ab'')2-mast cell secretory granule interaction.
- Corticosteroids as immunomodulators.
- Antinuclear antibodies in apparently healthy women with breast implants.
- Autoimmunity after silicone breast implants.
- Emin Pasha; confusion in the jungle.
- Dr. WILLIAM GILBERT--England''s first great scientist.
- Progressive myocardial damage following recovery from diphtheria. A case showing development of complete heart block.
- THYMECTOMY: PROLONGATION OF IMMUNOLOGICAL TOLERANCE IN THE ADULT MOUSE.
- DECLINE OF ANTIBODY AND IMPAIRED ANAMNESIS FOLLOWING X-RAY.
- TOLERANCE TO A PROTEIN ANTIGEN IN ADULT MICE AND THE EFFECT OF NONSPECIFIC FACTORS.
- KARL JASPERS-MODERN EXISTENTIALIST.
- CASE RECORDS OF THE MASSACHUSETTS GENERAL HOSPITAL. CASE 44-1964.
- THYMUS AND X-RADIATION IN THE TERMINATION OF ACQUIRED IMMUNOLOGICAL TOLERANCE IN THE ADULT MOUSE.
- QUANTITATIVE MEASUREMENT OF HUMAN GAMMA-2, BETA-2A, AND BETA-2M SERUM IMMUNOGLOBULINS.
- THE EFFECT OF ENDOTOXIN ON THE THYMUS OF YOUNG MICE.
- IMMUNOGLOBULIN COMPONENTS OF "GAMMA" GLOBULIN IN HUMAN SERUM.
- Tolerance and contact sensitivity to DNFB in mice. IV. Desensitization as a manifestation of increased proliferation of sensitized cells.
- Tolerance and contact sensitivity to DNFB in mice. V. Induction of tolerance with DNP compounds and with free and membrane-associated DNFB.
- The biology of the immune response.
- Histamine release from mouse and rat mast cells cultured with supernatants from chronic murine graft-vs-host splenocytes.
- Endothelial and fibroblastic activation in scleroderma. The myth of the "uninvolved skin".
- Morphological evidence for chronic mast cell activation after prolonged exposure with supernatants from chronic graft-versus-host splenocytes.
- Graft-versus-host disease and animal models for scleroderma.
- IL-3, IL-4, and IL-6 enhance IFN-gamma-dependent bone marrow natural suppressor activity.
- Dermal mast cell degranulation in systemic sclerosis.
- More about mast cells and fibrosis in porphyria cutanea tarda.
- Immunoglobulin dysregulation in murine graft-vs-host disease: a hyper-IgE syndrome.
- Mast cells and fibrosis. The relevance to scleroderma.
- Antibody-mediated regulation of T cell responses. I. Characterization of a monoclonal antibody which specifically regulates contact hypersensitivity to DNFB in BALB/c ...
- Immunologic dysfunction in scleroderma: evidence for increased mast cell releasability and HLA-DR positivity in the dermis.
- Hepatic homing of mononuclear inflammatory cells isolated during murine chronic graft-vs-host disease.
- Mast cells in murine graft-versus-host disease: a model of immunologically induced fibrosis.
- Ultrastructural evidence for mast cell activation in a case of neurofibromatosis.
- Natural suppressor cells.
- Murine natural suppressor cells in the newborn, in bone marrow, and after cyclophosphamide. Genetic variations and dependence on IFN-gamma.
- Immune mechanisms: T cells and B cells.
- Mast cell activity in normal human skin.
- On scleroderma. Mast cells, endothelial cells, and fibroblasts.
- Hepatic lesions in murine chronic graft-versus-host disease to minor histocompatibility antigens. A reproducible model of nonsuppurative destructive cholangitis.
- "Signal theory" in cellular immunology: collaboration between T- and B-lymphocytes in the immune response.
- Anti-immunoglobulin stimulation of murine lymphocytes. I. Age dependency of the proliferative response.
- Tolerance to contact hypersensitivity.
- Immune suppression with supraoptimal doses of antigen in contact sensitivity. I. Demonstration of suppressor cells and their sensitivity to cyclophosphamide.
- The induction of cell-mediated immunity and tolerance with protein antigens coupled to syngeneic lymphoid cells.
- Active suppression of 1-fluoro-2,4-dinitrobenzene-immune T cells. Requirement of an auxiliary T cell induced by antigen.
- Regulation of cell mediated immunity by antibodies: possible role of anti-receptor antibodies in the regulation of contact sensitivity to DNFB in mice.
- Human necrotizing vasculitis: immunoglobulins and complement in vessel walls of cutaneous lesions and normal skin.
- Age-related changes in cell-mediated immunity in BALB/C mice.
- H-2 restriction of suppressor T-cell induction by hapten-modified lymphoid cells in tolerance to 1-fluoro-2,4-dinitrobenzene contact sensitization.
- The induction of hapten-specific T cell tolerance using hapten-modified lymphoid membranes. II. Relative roles of suppressor T cells and clone inhibition in the tolera...
- Tolerance: two pathways of negative immunoregulation in contact sensitivity to DNFB.
- A splenic requirement for the generation of suppressor T cells.
- Genetic restrictions for the induction of suppressor T cells by hapten-modified lymphoid cells in tolerance to 1-fluoro-2,4-dinitrobenzene contact sensitivity. Role of...
- Suppressor T cell mechanisms in contact sensitivity. I. Efferent blockade by syninduced suppressor T cells.
- Suppressor T cell mechanisms in contact sensitivity. II. Afferent blockade by alloinduced suppressor T cells.
- Hypothesis. T-cell tolerance--one signal?
- Efficient induction of immediate tolerance to contact sensitivity by hapten-modified spleen cells requires Ia+ cells compatible with recipient.
- Binding of ovalbumin to mouse spleen cells with and without carbodiimide.
- How corticosteroids work.
- Suppressor cells in tolerance to contact sensitivity active against hapten-syngeneic and hapten-allogeneic determinants.
- Urticarial vasculitis.
- Leukocytoclastic vasculitis: sequential appearance of immunoreactants and cellular changes in serial biopsies.
- Requirements for induction of T cell tolerance to DNFB: efficiency of membrane-associated DNFB.
- The induction of hapten-specific T cell tolerance by using hapten-modified lymphoid cells. I. Characteristics of tolerance induction.
- Mast cell changes in a case of rapidly progressive scleroderma-ultrastructural analysis.
- Murine graft-versus-host disease across minor barriers: immunosuppressive aspects of natural suppressor cells.
- Desensitization of contact allergy to DNFB in mice. III. Characteristics of immediate desensitization induced by haptenated spleen cells.
- Desensitization of experimental contact sensitivity.
- Natural suppressor activity in graft-vs-host spleen and normal bone marrow is augmented by IL 2 and interferon-gamma.
- Cutaneous mast cell depletion and recovery in murine graft-vs-host disease.
- Simultaneous in situ detection of IgE receptors and cytoplasmic granules in murine cutaneous mast cells.
- Modulation of suppressor cell activity and improved resistance to infection in the burned mouse.
- Suppression of cytotoxic T-cell generation by natural suppressor cells from mice with GVHD is partially reversed by indomethacin.
- Evidence that IFN-gamma is responsible for natural suppressor activity in GVHD spleen and normal bone marrow.
- Chronic graft-vs.-host disease: changes in natural suppressor cells and mast cells.
- Corticosteroids and the immune response.
Schools
New York University
Barnes Jewish Hospital
Massachusetts General Hospital
University Colo Med Center
Doctors Specialties
- Allergy
- Immunology
Accepted Insurances
Education
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University of Colorado -
Massachusetts General Hospital -
Washington University - Barnes-Jewish Hospital -
NYU School of Medicine
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