Professor, Pediatrics, University of Cincinnati College of Medicine
Pediatrics
Dr. Hector R. Wong, MD
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Hector R. Wong is an expert in the field of Pediatrics. He went to medical school at Rutgers Robert Wood Johnson Medical School. Rutgers Robert Wood Johnson Medical School has a rank of 74 in research and a 67 in primary care Hector Wong is also a published physician. He has 99 publication published. The most recent publication is: Prospective Testing and Redesign of a Temporal Biomarker Based Risk Model for Patients With Septic Shock: Implications for Septic Shock Biology.
Publications
- Prospective Testing and Redesign of a Temporal Biomarker Based Risk Model for Patients With Septic Shock: Implications for Septic Shock Biology.
- Clinical utility of CT and MRI for diagnosis of posterior reversible encephalopathy syndrome (PRES) after stem cell transplant in children and adolescents.
- A Safety and Dose Escalation Study of Intravenous Zinc Supplementation in Pediatric Critical Illness.
- Comparing the prognostic performance of ASSIST to interleukin-6 and procalcitonin in patients with severe sepsis or septic shock.
- Developing a Clinically Feasible Personalized Medicine Approach to Pediatric Septic Shock.
- Combining functional and tubular damage biomarkers improves diagnostic precision for acute kidney injury after cardiac surgery.
- Identifying critically ill patients who may benefit from adjunctive corticosteroids: not as easy as we thought*.
- Biomarkers of sepsis and their potential value in diagnosis, prognosis and treatment.
- Incorporation of Biomarkers with the Renal Angina Index for Prediction of Severe AKI in Critically Ill Children.
- Performance of interleukin-27 as a sepsis diagnostic biomarker in critically ill adults.
- The pediatric sepsis biomarker risk model: potential implications for sepsis therapy and biology.
- Corticosteroids are associated with repression of adaptive immunity gene programs in pediatric septic shock.
- Gene expression profiling in sepsis: timing, tissue, and translational considerations.
- Combined Zinc Supplementation With Proinsulin C-Peptide Treatment Decreases the Inflammatory Response and Mortality in Murine Polymicrobial Sepsis.
- Role of Biomarkers in Sepsis Care.
- Interleukin-27 as a Sepsis Diagnostic Biomarker in Critically Ill Adults.
- Derivation and validation of the renal angina index to improve the prediction of acute kidney injury in critically ill children.
- Pediatric sepsis: challenges and adjunctive therapies.
- Zinc Detection in Serum by Anodic Stripping Voltammetry on Microfabricated Bismuth Electrodes.
- Genome-wide expression profiling in pediatric septic shock.
- Plasmapheresis to treat hypertriglyceridemia in a child with diabetic ketoacidosis and pancreatitis.
- Finding new therapies for sepsis: the need for patient stratification and the use of genetic biomarkers.
- Identification of candidate serum biomarkers for severe septic shock-associated kidney injury via microarray.
- Reduced PPAR? Expression is Associated With Decreased Survival and Increased Tissue Bacterial Load in Sepsis.
- A novel role for matrix metalloproteinase-8 in sepsis.
- The Influence of Developmental Age on the Early Transcriptomic Response of Children with Septic Shock.
- The PICU perspective on monitoring hemodynamics and oxygen transport.
- Validation of a gene expression-based subclassification strategy for pediatric septic shock.
- Antecedent acute kidney injury worsens subsequent endotoxin-induced lung inflammation in a 2-hit mouse model.
- The Myeloid Transcription Factor KLF2 Regulates the Host Response to Polymicrobial Infection and Endotoxic Shock.
- The immunomodulatory effects of albumin in vitro and in vivo.
- Genetic association research: understanding its challenges and limitations.
- Toward a clinically feasible gene expression-based subclassification strategy for septic shock: proof of concept.
- Mechanisms and regulation of the gene-expression response to sepsis.
- Plasma interleukin-8 is not an effective risk stratification tool for adults with vasopressor-dependent septic shock.
- Pathophysiology and treatment of septic shock in neonates.
- Biomarker discovery and development in pediatric critical care medicine.
- The host response to sepsis and developmental impact.
- Leukocyte subset-derived genomewide expression profiles in pediatric septic shock.
- Admission chemokine (C-C motif) ligand 4 levels predict survival in pediatric septic shock.
- The United States critical illness and injury trials group: an introduction.
- Lung injury after hemorrhage is age dependent: role of peroxisome proliferator-activated receptor gamma.
- Age-related decrease in proteasome expression contributes to defective nuclear factor-kappaB activation during hepatic ischemia/reperfusion.
- Critically associating.
- Zinc homeostasis in pediatric critical illness.
- Plasma angiopoietin-2 levels increase in children following cardiopulmonary bypass.
- Induction of endotoxin tolerance enhances bacterial clearance and survival in murine polymicrobial sepsis.
- The role of endogenously produced extracellular hsp72 in mononuclear cell reprogramming.
- Interleukin-8 as a stratification tool for interventional trials involving pediatric septic shock.
- Activation of hepatocytes by extracellular heat shock protein 72.
- Serum neutrophil gelatinase-associated lipocalin (NGAL) as a marker of acute kidney injury in critically ill children with septic shock.
- PP2A regulates upstream members of the c-jun N-terminal kinase mitogen-activated protein kinase signaling pathway.
- Therapeutic application of intrapericardial tissue plasminogen activator in a 4-month-old child with complex fibropurulent pericarditis.
- Admission angiopoietin levels in children with septic shock.
- Diverse cardioprotective signaling mechanisms of peroxisome proliferator-activated receptor-gamma ligands, 15-deoxy-Delta12,14-prostaglandin J2 and ciglitazone, in rep...
- Hsp72 induces inflammation and regulates cytokine production in airway epithelium through a TLR4- and NF-kappaB-dependent mechanism.
- Genome-level longitudinal expression of signaling pathways and gene networks in pediatric septic shock.
- The green tea polyphenol epigallocatechin-3-gallate improves systemic hemodynamics and survival in rodent models of polymicrobial sepsis.
- Role of heat shock protein 70 in hepatic ischemia-reperfusion injury in mice.
- Hepatocyte NF-kappaB activation is hepatoprotective during ischemia-reperfusion injury and is augmented by ischemic hypothermia.
- Heat shock response and acute lung injury.
- Extracellular heat shock protein-70 induces endotoxin tolerance in THP-1 cells.
- Glutamine's protection against cellular injury is dependent on heat shock factor-1.
- Adaptation and increased susceptibility to infection associated with constitutive expression of misfolded SP-C.
- Translation.
- Heat shock-mediated regulation of MKP-1.
- Extracellular hsp70 levels in children with septic shock.
- Contribution of MKP-1 regulation of p38 to endotoxin tolerance.
- Heat shock inhibition of lipopolysaccharide-mediated tumor necrosis factor expression is associated with nuclear induction of MKP-1 and inhibition of mitogen-activated...
- Theaflavin, a black tea extract, is a novel anti-inflammatory compound.
- Epigallocatechin-3-gallate, a green tea-derived polyphenol, inhibits IL-1 beta-dependent proinflammatory signal transduction in cultured respiratory epithelial cells.
- Differential regulation of activator protein-1 and heat shock factor-1 in myocardial ischemia and reperfusion injury: role of poly(ADP-ribose) polymerase-1.
- Short-term modulation of interleukin-1beta signaling by hyperoxia: uncoupling of IkappaB kinase activation and NF-kappaB-dependent gene expression.
- Proteasome inhibitors induce inhibitory kappa B (I kappa B) kinase activation, I kappa B alpha degradation, and nuclear factor kappa B activation in HT-29 cells.
- Epigallocatechin, a green tea polyphenol, attenuates myocardial ischemia reperfusion injury in rats.
- Parthenolide improves systemic hemodynamics and decreases tissue leukosequestration in rats with polymicrobial sepsis.
- Molecular genetics in the pediatric intensive care unit.
- Intracellular delivery of HSP70 using HIV-1 Tat protein transduction domain.
- Nuclear factor-kappaB as a therapeutic target in critical care medicine.
- Ablation of the heat shock factor-1 increases susceptibility to hyperoxia-mediated cellular injury.
- The A2A receptor mediates an endogenous regulatory pathway of cytokine expression in THP-1 cells.
- Phosphatase inhibition leads to activation of IkappaB kinase in murine macrophages.
- A green tea-derived polyphenol, epigallocatechin-3-gallate, inhibits IkappaB kinase activation and IL-8 gene expression in respiratory epithelium.
- Role of gene polymorphisms in sepsis.
- The stress response decreases NF-kappaB activation in liver of endotoxemic mice.
- Temporal and mechanistic effects of heat shock on LPS-mediated degradation of IkappaBalpha in macrophages.
- Yet another potential role for nitric oxide in the pathophysiology of septic shock.
- Parthenolide, an inhibitor of the nuclear factor-kappaB pathway, ameliorates cardiovascular derangement and outcome in endotoxic shock in rodents.
- Proteasome inhibitors induce heat shock response and increase IL-6 expression in human intestinal epithelial cells.
- Hyperoxia prolongs tumor necrosis factor-alpha-mediated activation of NF-kappaB: role of IkappaB kinase.
- Heat shock inhibits activation of NF-kappaB in the absence of heat shock factor-1.
- Absence of inducible nitric oxide synthase modulates early reperfusion-induced NF-kappaB and AP-1 activation and enhances myocardial damage.
- Heat shock inhibits tnf-induced ICAM-1 expression in human endothelial cells via I kappa kinase inhibition.
- Sesquiterpene lactone parthenolide, an inhibitor of IkappaB kinase complex and nuclear factor-kappaB, exerts beneficial effects in myocardial reperfusion injury.
- Interactions between the heat shock response and the nuclear factor-kappa B signaling pathway.
- Inhaled nitric oxide increases endothelin-1 levels: a potential cause of rebound pulmonary hypertension.
- Interactions between the heat shock response and the nuclear factor-kappaB signaling pathway.
- ARDS. The future.
Schools
UmdnjRobert Wood Johnson Medical School
Chldn39s National Med Center
Pittsburgh Chldn39s Hospital
Doctors Specialties
- Pediatric Critical Care Medicine
- Pediatrics
Accepted Insurances
- Amerigroup (Wellpoint)
- CareSource
- Cigna HMO
- Cigna PPO
- Great West Healthcare-Cigna PPO
- Open Access Plus
- Cofinity PPO
- Coventry Health America PPO
- First Health (Coventry Health Care) PPO
- Medical Mutual of Ohio
- Medicare MCR
- Molina
- Choice Plus POS
- Navigate HMO
- Navigate POS
- Options PPO
- United Healthcare
Education
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University of Pittsburgh Medical Center -
Children's National Medical Center - George Washington University -
Rutgers Robert Wood Johnson Medical School
Hospital
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Cincinnati Children's Hospital Medical Center
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