Physician
Internal Medicine
Dr. Harry Bluestein, MD
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Harry Bluestein works in the field of Internal Medicine. He graduated from Albert Einstein College of Medicine of Yeshiva University. He received 2 awards: "CMS Meaningful Use Stage 1 Certification" and "Elected Member". Harry Bluestein, MD has written publications. He has 68 publications published with PubMed. The lastest paper is: 'Lymphocyte alteration by procainamide: relation to drug-induced lupus erythematosus syndrome.' He is registered with Medicare and accepts Medicare payments.
Publications
- Utility of the ultrasound examination of the hand and wrist joints in the management of established rheumatoid Arthritis.
- Assessment of long-term antiarrhythmic therapy: studies on the long-term efficacy and toxicity of tocainide.
- Reduction in circulating dsDNA antibody titer after administration of LJP 394.
- Neuropsychiatric disease in Sjögren's Syndrome: anti-ribosomal P and anti-neuronal antibodies.
- Cerebrospinal fluid antibodies to neuronal Cells: association with neuropsychiatric manifestations of systemic lupus erythematosus.
- Brain-reactive lymphocytotoxic antibody in the cerebrospinal fluid of patients with systemic Lupus erythematosus: correlation with central nervous system involvement.
- The pathogenesis of central nervous system manifestations of systemic Lupus erythematosus.
- Antineuronal antibodies in systemic Lupus erythematosus.
- Altered deoxynucleoside triphosphate levels paralleling deoxyadenosine toxicity in adenosine deaminase inhibited human lymphocytes.
- The mechanism of inhibition and "reversal" of mitogen-induced lymphocyte activation in a model of adenosine deaminase deficiency.
- The mechanism of inhibition and "reversal" of mitogen-induced lymphocyte activation in a model of purine-nucleoside phosphorylase deficiency.
- Deoxyadenosine (AdR) inhibition of newly activated lymphocytes: blockade at the G0-G1 interface.
- Panning separation for monoclonal antibody-specific T-Cell subsets: effects of enzymes and metabolic inhibitors.
- Epstein-Barr virus and rheumatoid Arthritis.
- Analysis of the defects responsible for the impaired regulation of EBV-induced B cell proliferation by rheumatoid Arthritis lymphocytes. II. Role of monocytes and the ...
- Analysis of the defects responsible for the impaired regulation of Epstein-Barr virus-induced B cell proliferation by rheumatoid Arthritis lymphocytes. I. Diminished g...
- Oropharyngeal Epstein-Barr virus excretion in rheumatoid Arthritis.
- Impaired regulation of Epstein-Barr virus-induced lymphocyte proliferation in rheumatoid Arthritis is due to a T cell defect.
- Altered regulation of Epstein-Barr virus induced lymphoblast proliferation in rheumatoid Arthritis lymphoid cells.
- DNA synthesis inhibitors block protein synthesis in the human lymphocyte.
- Deoxyadenosine modulates human suppressor T Cell function and B Cell differentiation stimulated by Staphylococcus aureus protein A.
- Procainamide-lymphocyte reactions. A possible explanation for drug-induced autoimmunity.
- Antibodies reactive with central nervous system antigens.
- Specific Immune response genes of the guinea pig. II. Relationship between the poly-L-lysine gene and the genes controlling Immune responsiveness to copolymers of L-gl...
- Specific Immune response genes of the guinea pig. I. Dominant genetic control of Immune responsiveness to copolymers of L-glutamic acid and L-alanine and L-glutamic ac...
- The inability of RNA from lymph node Cells from immunized strain 2 animals to confer immune competence to strain 13 lymph node Cells in the DNP-PLL immune system.
- Croton oil induced suppression of the Immune response of guinea-pigs.
- Histocompatibility specificity.
- Specific Immune response genes of the guinea pig. 3. Linkage of the GA and GT Immune response genes to histocompatibility genotypes in inbred guinea pigs.
- Anti-TNP antibody responses in vitro: augmented primary responses of carrier-primed spleen Cells.
- Specific immune response genes of the guinea pig. V. Influence of the GA and GT immune response genes on the specificity of Cellular and humoral immune responses to a ...
- Seryl transfer ribonucleic acid synthetase from bakers'' yeast. 3. The seryladenylate-enzyme complex and its interaction with transfer ribonucleic acids.
- Alloantiserum-mediated suppression of histocompatibility-linked Ir-gene-controlled Immune responses. Suppressive effects of IgG fragments derived from alloantisera.
- Development of primary M, G, and A anti-TNP plaque-forming Cell responses in vitro.
- Alloantiserum-mediated suppression of histocompatibility-linked IR gene controlled Immune responses. I. Histocompatibility antigen requirement.
- Cellular requirements for development of primary anti-hapten antibody responses in vitro.
- Neuropsychiatric manifestations of systemic Lupus erythematosus.
- Deoxy-ATP accumulation in adenosine deaminase-inhibited human B and T lymphocytes.
- Platelet glycoproteins Ia, Ic, and IIa are physicochemically indistinguishable from the very late activation antigens adhesion-related proteins of lymphocytes and othe...
- Anatomic and genetic considerations in the pathogenesis of Ankylosing spondylitis.
- Immunopathogenesis of the neuropsychiatric manifestations of systemic Lupus erythematosus.
- Regulation of the growth of Epstein-Barr virus-infected B Cells: temporal profile of the in vitro development of three distinct cytotoxic Cells.
- Use of the monoclonal antibody 12F1 to characterize the differentiation antigen VLA-2.
- Vancomycin-induced vasculitis.
- Regulation of the growth of Epstein-Barr virus-infected B Cells. I. Growth regression by E rosetting Cells from VCA-positive donors is a combined effect of autologous ...
- Generation of human cytomegalovirus-specific cytotoxic T-lymphocytes in a short-term culture.
- Effects of tilomisole, indomethacin and levamisole on regulation of Epstein Barr virus-induced B Cell proliferation by peripheral blood mononuclear Cells from normal i...
- Antigenic polymorphism of human very late activation protein-2 (platelet glycoprotein Ia-IIa). Platelet alloantigen Hca.
- Polymorphism of lymphocyte function-associated antigen-1 demonstrated by a Lupus patient's alloantiserum.
- Very late activation antigens (VLA) are human leukocyte-neuronal crossreactive Cell surface antigens.
- Neuropsychologic deficits and antineuronal antibodies in pediatric systemic Lupus erythematosus.
- BEN, a novel surface molecule of the immunoglobulin superfamily on avian hemopoietic progenitor Cells shared with neural Cells.
- An antigen expressed by avian neuronal Cells is also expressed by activated T lymphocytes.
- Hypocomplementemia in procainamide-associated systemic Lupus erythematosus.
- Alloantiserum-mediated supperssion of Ir gene controlled antigen-induced lymphocyte activation: effect on uridine and leucine incorporation.
- Lymphocytotoxic antibody activity in cryoprecipitates from serum of patients with SLE.
- Lymphocyte reactivity of antisera to cryoproteins in systemic Lupus erythematosus.
- Heterogeneous neurocytotoxic antibodies in systemic Lupus erythematosus.
- Accumulation of deoxyribonucleotides as a possible mediator of immunosuppression in hereditary deficiency of adenosine deaminase.
- Antigen recognition by primed T lymphocytes in vitro. I. Separation of recognitive and proliferative phases of antigen-induced T Cell activation.
- Antibody-mediated suppression of bone marrow colony formation in vitro.
- Neurocytotoxic antibodies in serum of patients with systemic Lupus erythematosus.
- Lymphocyte alteration by procainamide: relation to drug-induced lupus erythematosus Syndrome.
- Antibody-mediated suppression of antibody-dependent cytotoxic effector Cell function.
- Neoantigen of the membrane attack complex of human complement: Occurrence on peripheral blood leukocytes from patients with systemic Lupus erythematosus.
- Brain-reactive lymphocytotoxic antibodies in the serum of patients with systemic Lupus erythematosus.
- Improving knowledge in palliative medicine with a required hospice rotation for third-year medical students.
Schools
Albert Einstein College of Medicine of Yeshiva University
Georgetown University Hospital
Procedures Preformed
- Joint Drainage
- Myofascial Trigger Point Injection
- Steroid Injection
Conditions Treated
- Amyloidosis
- Ankylosing Spondylitis
- Antiphospholipid Syndrome (APS)
- Arthritis
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Doctors Specialties
- Rheumatology
- Internal Medicine
Accepted Insurances
Awards
- CMS Meaningful Use Stage 1 Certification
- Elected Member
Education
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Georgetown University Hospital -
Albert Einstein College of Medicine of Yeshiva University
Drug Facts
| NPI NUMBER | 1295793024 | |
| NPPES Provider LastName | BLUESTEIN | |
| NPPES Provider FirstName | HARRY | |
| NPPES Provider ZIPCode | 921039000 | |
| NPPES Provider State | CA | |
| Specialty Description | Rheumatology | |
| Total Claim Count | 824.0 | |
| Distinct Opioid Count | 1.0 | |
| Opioid Claim Count | 11.0 | |
| Percent Opioid Claims | 1.33 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1295793024 |
| Last Name Of The Provider | BLUESTEIN |
| First Name Of The Provider | HARRY |
| View All |
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