Associate Professor, Pediatrics, Massachusetts General Hospital, Harvard Medical School
Internal Medicine
Dr. H S. Warren, MD
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Dr. H S. Warren practices Internal Medicine. He researched medicine at Harvard Medical School. Harvard Medical School is a top medical college. Harvard Medical School is ranked 1 in research and 12 in primary care medicine Warren is a published MD. He published 72 publications, including: 'Quantitative analysis of the effect of CD16 ligation on human NK cell proliferation.'
Publications
- Mice are not men.
- Strategies to improve drug development for sepsis.
- Intestinal alkaline phosphatase promotes gut bacterial growth by reducing the concentration of luminal nucleotide triphosphates.
- Intestinal alkaline phosphatase prevents metabolic syndrome in mice.
- Genomic responses in mouse models poorly mimic human inflammatory diseases.
- Development of a Genomic Metric That Can Be Rapidly Used to Predict Clinical Outcome in Severely Injured Trauma Patients.
- Intestinal alkaline phosphatase inhibits the proinflammatory nucleotide uridine diphosphate.
- Prevalence and Outcomes of Antimicrobial Treatment for Staphylococcus aureus Bacteremia in Outpatients with ESRD.
- Identification of Hemopexin as an Anti-inflammatory Factor That Inhibits Synergy of Hemoglobin with HMGB1 in Sterile and Infectious Inflammation.
- Case records of the Massachusetts General Hospital. Case 19-2012. A premature newborn boy with respiratory distress.
- Local peritoneal irrigation with intestinal alkaline phosphatase is protective against peritonitis in mice.
- A role for intestinal alkaline phosphatase in the maintenance of local gut immunity.
- Editorial: Control issues in sepsis: what modulates apoptosis?
- Intestinal alkaline phosphatase has beneficial effects in mouse models of chronic colitis.
- Synergistic inflammation is induced by blood degradation products with microbial Toll-like receptor agonists and is blocked by hemopexin.
- Identification of specific targets for the gut mucosal defense factor intestinal alkaline phosphatase.
- Resilience to bacterial infection: difference between species could be due to proteins in serum.
- Validation of the riboleukogram to detect ventilator-associated pneumonia after severe injury.
- Hemopexin down-regulates LPS-induced proinflammatory cytokines from macrophages.
- Editorial: Mouse models to study sepsis syndrome in humans.
- Peroxisome proliferator-activated receptor-gamma agonists, control of bacterial outgrowth, and inflammation.
- Intestinal alkaline phosphatase is a gut mucosal defense factor maintained by enteral nutrition.
- MyD88-dependent and MyD88-independent pathways in synergy, priming, and tolerance between TLR agonists.
- Passive immunization to outer membrane proteins MLP and PAL does not protect mice from sepsis.
- Toll-like receptors.
- Bacterial peptidoglycan-associated lipoprotein: a naturally occurring toll-like receptor 2 agonist that is shed into serum and has synergy with lipopolysaccharide.
- Reversing established sepsis with antagonists of endogenous high-mobility group box 1.
- Case records of the Massachusetts General Hospital. Weekly clinicopathological exercises. Case 38-2003. A 12-year-old girl with fever and coma.
- Protective efficacy of CAP18106-138-immunoglobulin G in sepsis.
- Murein lipoprotein, peptidoglycan-associated lipoprotein, and outer membrane protein A are present in purified rough and smooth lipopolysaccharides.
- Structural basis for the proinflammatory cytokine activity of high mobility group box 1.
- Risks and benefits of activated protein C treatment for severe sepsis.
- Bacterial peptidoglycan-associated lipoprotein is released into the bloodstream in gram-negative sepsis and causes inflammation and death in mice.
- Active immunization with a detoxified Escherichia coli J5 lipopolysaccharide group B meningococcal outer membrane protein complex vaccine protects animals from experim...
- Localization of endotoxin in the rat intestinal epithelium.
- A model of infected burn wounds using Escherichia coli O18:K1:H7 for the study of gram-negative bacteremia and sepsis.
- Outer membrane protein A, peptidoglycan-associated lipoprotein, and murein lipoprotein are released by Escherichia coli bacteria into serum.
- Release of gram-negative outer-membrane proteins into human serum and septic rat blood and their interactions with immunoglobulin in antiserum to Escherichia coli J5.
- Antiendotoxin strategies.
- Quantitative analysis of the effect of CD16 ligation on human NK cell proliferation.
- Antiserum against Escherichia coli J5 contains antibodies reactive with outer membrane proteins of heterologous gram-negative bacteria.
- Relationship of tissue and cellular interleukin-1 and lipopolysaccharide after endotoxemia and bacteremia.
- Effect of anticoagulants on binding and neutralization of lipopolysaccharide by the peptide immunoglobulin conjugate CAP18(106-138)-immunoglobulin G in whole blood.
- Strategies for the treatment of sepsis.
- A novel peptide-IgG conjugate, CAP18(106-138)-IgG, that binds and neutralizes endotoxin and kills gram-negative bacteria.
- Analysis of the costimulatory role of IL-2 and IL-15 in initiating proliferation of resting (CD56dim) human NK cells.
- A carbohydrate structure associated with CD15 (Lewis x) on myeloid cells is a novel ligand for human CD2.
- Production of IL-5 by human NK cells and regulation of IL-5 secretion by IL-4, IL-10, and IL-12.
- Distinct granzyme expression in human CD3- CD56+ large granular- and CD3- CD56+ small high density-lymphocytes displaying non-MHC-restricted cytolytic activity.
- Spontaneous Neutrophil Migration Patterns during Sepsis after Major Burns.
- Acute inflammatory response to endotoxin in mice and humans.
- A novel binding assay to assess specificity of monoclonal antibodies.
- Analysis of factorial time-course microarrays with application to a clinical study of burn injury.
- A genomic storm in critically injured humans.
- An economical adaptation of the RosetteSep procedure for NK cell enrichment from whole blood, and its use with liquid nitrogen stored peripheral blood mononuclear cells.
- The Eighth Human Leucocyte Differentiation Antigen (HLDA8) Workshop: natural killer cell section report.
- The intracellular granzyme B inhibitor, proteinase inhibitor 9, is up-regulated during accessory cell maturation and effector cell degranulation, and its overexpressio...
- Use of the intracellular fluorescent dye CFSE to monitor lymphocyte migration and proliferation.
- Target-induced natural killer cell loss as a measure of NK cell responses.
- Human genetics: an inflammatory issue.
- KIR2DL4 (CD158d) genotype influences expression and function in NK cells.
- Identification and interpretation of longitudinal gene expression changes in trauma.
- A genomic score prognostic of outcome in trauma patients.
- Functional inhibitory human leucocyte antigen class I receptors on natural killer (NK) cells in patients with chronic NK lymphocytosis.
- CD8 T cells expressing killer Ig-like receptors and NKG2A are present in cord blood and express a more naïve phenotype than their counterparts in adult blood.
- Monitoring lymphocyte proliferation in vitro and in vivo with the intracellular fluorescent dye carboxyfluorescein diacetate succinimidyl ester.
- Phase I study of detoxified Escherichia coli J5 lipopolysaccharide (J5dLPS)/group B meningococcal outer membrane protein (OMP) complex vaccine in human subjects.
- Myocardial ischemia activates an injurious innate immune signaling via cardiac heat shock protein 60 and Toll-like receptor 4.
- Cutting edge: lectin-like transcript-1 is a ligand for the inhibitory human NKR-P1A receptor.
- Evidence that the cellular ligand for the human NK cell activation receptor NKp30 is not a heparan sulfate glycosaminoglycan.
- Gut Microbiota-Induced Immunoglobulin G Controls Systemic Infection by Symbiotic Bacteria and Pathogens.
Schools
Harvard Medical School
Doctors Specialties
- Infectious Disease Medicine
- Internal Medicine
Accepted Insurances
Education
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Beth Israel Deaconess Medical Center -
University of North Carolina Hospitals -
Harvard Medical School
Hospital
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Massachusetts General Hospital
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