National Jewish Health
Pulmonology
Dr. Gregory P. Downey, MD
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Gregory Downey is an expert in the field of Pulmonology. He has 2 awards "Member" and "Elected Member". Dr. Gregory P. Downey is a published physician. He published 71 publications, including: 'Divergent Functions of Toll-Like Receptors during Bacterial Lung Infections.' He is registered with Medicare and accepts Medicare payments.
Publications
- Divergent Functions of Toll-Like Receptors during Bacterial Lung Infections.
- Investigating the role of NOD-Like Receptors in Bacterial Lung Infection.
- Protein Tyrosine Phosphatase α Mediates Profibrotic Signaling in Lung Fibroblasts through TGF-β Responsiveness.
- TNF-α Accelerates the Resolution of Established Pulmonary Fibrosis in Mice by Targeting Pro-Fibrotic Lung Macrophages.
- Serum VEGF-D concentration as a biomarker of lymphangioleiomyomatosis severity and treatment response: a prospective analysis of the Multicenter International Lymphang...
- Neutrophil intercellular communication in acute Lung injury. Emerging roles of microparticles and gap junctions.
- The Fibroproliferative Response in ARDS: Mechanisms and Clinical Significance.
- Neutrophil Intercellular Communication in Acute Lung Injury: Emerging Roles of Microparticles and Gap Junctions.
- Role of ?-catenin-regulated CCN matricellular proteins in epithelial repair after inflammatory Lung injury.
- Detrimental role of the airway mucin Muc5ac during ventilator-induced Lung injury.
- HER receptor signaling in acute Lung injury.
- Burkholderia cenocepacia disrupts host cell actin cytoskeleton by inactivating Rac and Cdc42.
- On, around, and through: neutrophil-endothelial interactions in innate immunity.
- Resolving the scar of Pulmonary fibrosis.
- Neutrophil transmigration triggers repair of the Lung epithelium via {beta}-catenin signaling.
- Matrix Metalloproteinase 3 Is a Mediator of Pulmonary Fibrosis.
- Age and Gender Dimorphisms Contribute to the Severity of Bleomycin-Induced Lung Injury and Fibrosis.
- Focal adhesions and Ras are functionally and spatially integrated to mediate IL-1 activation of ERK.
- Importance of protein-tyrosine phosphatase-alpha catalytic domains for interactions with SHP-2 and interleukin-1-induced matrix metalloproteinase-3 expression.
- It's much more than just pretty pictures.
- Remote ischemic preconditioning decreases adhesion and selectively modifies functional responses of human neutrophils.
- Leukocyte elastase induces Lung epithelial apoptosis via a PAR-1-, NF-kappaB-, and p53-dependent pathway.
- Dysfunctional cystic fibrosis transmembrane conductance regulator inhibits phagocytosis of apoptotic cells with proinflammatory consequences.
- Protein-tyrosine phosphatase-alpha and Src functionally link focal adhesions to the endoplasmic reticulum to mediate interleukin-1-induced Ca2+ signaling.
- Human neutrophil peptides and phagocytic deficiency in bronchiectatic Lungs.
- Transepithelial migration of neutrophils: mechanisms and implications for acute Lung injury.
- Protease-activated receptor (Par)1 alters bioelectric properties of distal Lung epithelia without compromising barrier function.
- Use of fluorescent probes to detect lipid signaling intermediates in macrophages.
- Tyrosine phosphatase PTPalpha regulates focal adhesion remodeling through Rac1 activation.
- Role of caveolin-1 in regulation of inflammation: different strokes for different folks.
- Role of PAR2 in murine Pulmonary pseudomonal infection.
- CD44-mediated phagocytosis induces inside-out activation of complement receptor-3 in murine macrophages.
- Molecular pathogenesis of lymphangioleiomyomatosis: lessons learned from orphans.
- Reactive oxygen and nitrogen species as signaling molecules regulating neutrophil function.
- Cystic fibrosis mouse models.
- Up-regulation of functional CXCR4 expression on human lymphocytes in sepsis.
- Murine hepatitis virus strain 1 produces a clinically relevant model of severe acute Respiratory syndrome in A/J mice.
- Phosphorylation of SHP-2 regulates interactions between the endoplasmic reticulum and focal adhesions to restrict interleukin-1-induced Ca2+ signaling.
- Regenerative medicine and the developing world.
- CD44 is a phagocytic receptor.
- Tyrosine phosphatase SHP-2 regulates IL-1 signaling in fibroblasts through focal adhesions.
- Human neutrophil peptides induce interleukin-8 production through the P2Y6 signaling pathway.
- Abnormalities in the Pulmonary innate immune system in cystic fibrosis.
- Cyclic nucleotides modulate genioglossus and hypoglossal responses to excitatory inputs in rats.
- Neutrophil granule contents in the pathogenesis of Lung injury.
- Tyrosine phosphatase MEG2 modulates murine development and platelet and lymphocyte activation through secretory vesicle function.
- Proteinase-activated receptor-1 mediates elastase-induced apoptosis of human Lung epithelial cells.
- Mitochondrial function is a critical determinant of IL-1-induced ERK activation.
- Cytosolic phospholipase A2-alpha is necessary for platelet-activating factor biosynthesis, efficient neutrophil-mediated bacterial killing, and the innate immune respo...
- SHP-2 modulates interleukin-1-induced Ca2+ flux and ERK activation via phosphorylation of phospholipase Cgamma1.
- Rac1 is the small GTPase responsible for regulating the neutrophil chemotaxis compass.
- Lung inflammation as a therapeutic target in cystic fibrosis.
- The remote ischemic preconditioning stimulus modifies inflammatory gene expression in humans.
- Leukocyte elastase induces epithelial apoptosis: role of mitochondial permeability changes and Akt.
- Measurements of phagocytosis and phagosomal maturation.
- Factors affecting attainment of paid employment after Lung transplantation.
- Airway inflammation and Infection in congenital bilateral absence of the vas deferens.
- Biological markers in diagnosing, monitoring, and treating Asthma: a focus on noninvasive measurements.
- Oxidative stress and acute Lung injury.
- IL-1 induced release of Ca2+ from internal stores is dependent on cell-matrix interactions and regulates ERK activation.
- The protein tyrosine phosphatase SHP-2 regulates interleukin-1-induced ERK activation in fibroblasts.
- L-selectin stimulation enhances functional expression of surface CXCR4 in lymphocytes: implications for cellular activation during adhesion and migration.
- Rac1 deletion in mouse neutrophils has selective effects on neutrophil functions.
- Transfection of Lung cells in vitro and in vivo: effect of antioxidants and intraliposomal bFGF.
- Proteases and Lung injury.
- Accommodation after Lung xenografting from hamster to rat.
- Conventional mechanical ventilation is associated with bronchoalveolar lavage-induced activation of polymorphonuclear leukocytes: a possible mechanism to explain the s...
- Translocation of the tetraspanin CD63 in association with human eosinophil mediator release.
- A simplified model for en bloc double Lung xenotransplantation from hamster to rat.
- Protein-tyrosine phosphatase MEG2 is expressed by human neutrophils. Localization to the phagosome and activation by polyphosphoinositides.
Procedures Preformed
- Bronchoscopy
Conditions Treated
- Asthma
- Atelectasis
- Bronchiectasis
- Bronchiolitis
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Doctors Specialties
- Critical Care Medicine
- Pulmonology
Accepted Insurances
Awards
- Member
- Elected Member
Education
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University of Colorado -
Beth Israel Deaconess Medical Center -
University of Manitoba
Hospital
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National Jewish Health
Drug Facts
| NPI NUMBER | 1164598116 | |
| NPPES Provider LastName | DOWNEY | |
| NPPES Provider FirstName | GREGORY | |
| NPPES Provider ZIPCode | 802062761 | |
| NPPES Provider State | CO | |
| Specialty Description | Pulmonary Disease | |
| Total Claim Count | 127.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1164598116 |
| Last Name Of The Provider | DOWNEY |
| First Name Of The Provider | GREGORY |
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