Associate Professor, Pathology, Yale University School of Medicine
Pathology
Dr. Gilbert Moeckel, MD
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Gilbert Moeckel works in the field of Pathology. Gilbert Moeckel is also a published doctor. He has 74 paper published. The most recent paper is: On the origins of tumor mutations in cancer genes: insights from the p53 gene.
Publications
- GM-CSF Promotes Macrophage Alternative Activation after Renal Ischemia/Reperfusion Injury.
- Chemokine receptor Cxcr4 contributes to kidney fibrosis via multiple effectors.
- Role of medullary progenitor cells in epithelial cell migration and proliferation.
- Podocyte-associated talin1 is critical for glomerular filtration barrier maintenance.
- Renalase Prevents AKI Independent of Amine Oxidase Activity.
- Met activation is required for early cytoprotection after ischemic kidney injury.
- Alloantibody and complement promote T cell-mediated cardiac allograft vasculopathy through noncanonical nuclear factor-kappaB signaling in endothelial cells.
- Hypertonic stress and cell death. Focus on "Multiple cell death pathways are independently activated by lethal hypertonicity in renal epithelial cells".
- Mineralocorticoid receptor phosphorylation regulates ligand binding and renal response to volume depletion and hyperkalemia.
- Aldosterone stimulates fibronectin synthesis in renal fibroblasts through mineralocorticoid receptor-dependent and independent mechanisms.
- Early B-cell factor 1 is an essential transcription factor for postnatal glomerular maturation.
- Babesiosis-induced acute kidney injury with prominent urinary macrophages.
- Macrophage-specific deletion of transforming growth factor-β1 does not prevent renal fibrosis after severe ischemia-reperfusion or obstructive injury.
- Acute kidney injury in a patient with sarcoidosis: hypercalciuria and hypercalcemia leading to calcium phosphate deposition.
- NALP3-mediated inflammation is a principal cause of progressive renal failure in oxalate nephropathy.
- Recessive mutations in DGKE cause atypical hemolytic-uremic syndrome.
- A 7-year-old boy with renal insufficiency and proteinuria after stem cell transplant for T-cell acute lymphoblastic leukemia.
- Chitinase-Like Protein Brp-39/YKL-40 Modulates the Renal Response to Ischemic Injury and Predicts Delayed Allograft Function.
- Role of dynamin, synaptojanin, and endophilin in podocyte foot processes.
- Intrarenal dopamine inhibits progression of diabetic nephropathy.
- Acute Podocyte Vascular Endothelial Growth Factor (VEGF-A) Knockdown Disrupts alpha(V)beta(3) Integrin Signaling in the Glomerulus.
- Inorganic phosphate stimulates fibronectin expression in renal fibroblasts.
- Quantification and localization of M2 macrophages in human kidneys with acute tubular injury.
- Podocyte vascular endothelial growth factor (Vegfâ‚Â₆₄) overexpression causes severe nodular glomerulosclerosis in a mouse model of type 1 diabetes.
- Reabsorption of betaine in Henle's loops of rat kidney in vivo.
- On the origins of tumor mutations in cancer genes: insights from the p53 gene.
- Molecular modeling information transfer with VRML: from small molecules to large systems in bioscience.
- Kidney-targeted liposome-mediated gene transfer in mice.
- Kinetics and osmoregulation of Na(+)-and Cl(-)-dependent betaine transporter in rat renal medulla.
- Distribution of de novo synthesized betaine in rat kidney: role of renal synthesis on medullary betaine accumulation.
- Virtual reality modeling language in chemistry.
- A new approach to analysis and display of local lipophilicity/hydrophilicity mapped on molecular surfaces.
- Bicarbonate dependency of betaine synthesis in cultured LLC-PK1 cells.
- Bicarbonate-dependent betaine synthesis in rat kidney.
- Purification of human and rat kidney aldose reductase.
- Glomerular injury is exacerbated in diabetic integrin alpha1-null mice.
- Second prize: simple method for achieving renal parenchymal hypothermia for pure laparoscopic partial nephrectomy.
- Role of integrin alpha1beta1 in the regulation of renal medullary osmolyte concentration.
- Expression of mediators of renal injury in the remnant kidney of ROP mice is attenuated by cyclooxygenase-2 inhibition.
- Polymorphisms in human organic anion-transporting polypeptide 1A2 (OATP1A2): implications for altered drug disposition and central nervous system drug entry.
- COX2 activity promotes organic osmolyte accumulation and adaptation of renal medullary interstitial cells to hypertonic stress.
- Organic osmolytes betaine, sorbitol and inositol are potent inhibitors of erythrocyte membrane ATPases.
- Cyclooxygenase-2 inhibitor blocks expression of mediators of renal injury in a model of diabetes and hypertension.
- Puromycin induces reversible proteinuric injury in transgenic mice expressing cyclooxygenase-2 in podocytes.
- Overexpression of cyclooxygenase-2 predisposes to podocyte injury.
- Adaptation of the nematode Caenorhabditis elegans to extreme osmotic stress.
- Resolution of renal inflammation: a new role for NF-kappaB1 (p50) in inflammatory kidney diseases.
- Placental insufficiency associated with loss of Cited1 causes renal medullary dysplasia.
- Beta1 integrin expression by podocytes is required to maintain glomerular structural integrity.
- Deletion of the Met receptor in the collecting duct decreases renal repair following ureteral obstruction.
- Distinct roles for basal and induced COX-2 in podocyte injury.
- An 11-year-old African-American girl with systemic lupus erythematosus and ANCA-negative renal vasculitis.
- Resolution of proteinuria in a patient with focal segmental glomerulosclerosis following BiPAP initiation for obesity hypoventilation syndrome.
- Podocyte COX-2 exacerbates diabetic nephropathy by increasing podocyte (pro)renin receptor expression.
- SGLT2 deletion improves glucose homeostasis and preserves pancreatic beta-cell function.
- Hypertonicity-induced mitochondrial membrane permeability in renal medullary interstitial cells: protective role of osmolytes.
- Hematuria and decreased kidney function as initial signs of acute B-cell lymphoblastic leukemia.
- Nephrotoxicity from chemotherapeutic agents: clinical manifestations, pathobiology, and prevention/therapy.
- Late-onset omeprazole-associated acute interstitial nephritis.
- Identification and regulation of reticulon 4B (Nogo-B) in renal tubular epithelial cells.
- TGF-beta receptor deletion in the renal collecting system exacerbates fibrosis.
- Anti-phospholipid antibody syndrome in the kidney.
- A sealed bladder cuff technique during laparoscopic nephroureterectomy utilizing the LigaSure electrosurgical device: laboratory and clinical experience.
- Urea promotes TonEBP expression and cellular adaptation in extreme hypertonicity.
- Fibrocystin/polyductin modulates renal tubular formation by regulating polycystin-2 expression and function.
- NFATc1 identifies a population of proximal tubule cell progenitors.
- Apoptosis of the thick ascending limb results in acute kidney injury.
- Importance of functional EGF receptors in recovery from acute nephrotoxic injury.
- A novel gene encoding a TIG multiple domain protein is a positional candidate for autosomal recessive polycystic kidney disease.
- A woman with chronic hepatitis C infection and nephrotic syndrome who developed multiple renal lesions after interferon alfa therapy.
- Lack of integrin alpha1beta1 leads to severe glomerulosclerosis after glomerular injury.
- PKHD1 protein encoded by the gene for autosomal recessive polycystic kidney disease associates with basal bodies and primary cilia in renal epithelial cells.
- Proteolytic processing of laminin-5 by MT1-MMP in tissues and its effects on epithelial cell morphology.
Doctors Specialties
- Pathology
Accepted Insurances
- Medicare MCR
- Choice Plus POS
- Navigate HMO
- Navigate POS
- Options PPO
- United Healthcare
Education
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University of Arizona -
Ludwig Maximilian University of Munich Faculty of Medicine
Hospital
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Yale-New Haven Hospital
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1801970900 |
| Last Name Of The Provider | MOECKEL |
| First Name Of The Provider | GILBERT |
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