Assistant Professor of Medicine, Beth Israel Deaconess Medical Center
Internal Medicine
Dr. Gerburg M. Wulf, MD
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Gerburg Wulf is an expert in the field of Internal Medicine. Gerburg Wulf is a published doctor and has 40 publications published. The lastest was: 'BEACOPP, a new dose-escalated and accelerated regimen, is at least as effective as COPP/ABVD in patients with advanced-stage Hodgkin's lymphoma: interim report from a ...' She is registered with Medicare and accepts Medicare payments.
Publications
- The Rab2A GTPase Promotes Breast Cancer Stem Cells and Tumorigenesis via Erk Signaling Activation.
- Depletion of a Putatively Druggable Class of Phosphatidylinositol Kinases Inhibits Growth of p53-Null Tumors.
- PKM2 Isoform-Specific Deletion Reveals a Differential Requirement for Pyruvate Kinase in Tumor Cells.
- Closing escape routes: inhibition of IL-8 signaling enhances the anti-tumor efficacy of PI3K inhibitors.
- Combining a PI3K Inhibitor with a PARP Inhibitor Provides an Effective Therapy for BRCA1-Related Breast Cancer.
- The amplifier effect: how Pin1 empowers mutant p53.
- Phase I/II Study of Trastuzumab in Combination With Everolimus (RAD001) in Patients With HER2-Overexpressing Metastatic Breast Cancer Who Progressed on Trastuzumab-Bas...
- Loss of BRCA1 leads to an increase in epidermal growth factor receptor expression in mammary epithelial cells, and epidermal growth factor receptor inhibition prevents...
- Altered proliferation and differentiation properties of primary mammary epithelial cells from BRCA1 mutation carriers.
- Role of Pin1 in the regulation of p53 stability and p21 transactivation, and cell cycle checkpoints in response to DNA damage.
- Addition of verapamil to overcome drug resistance in multiple myeloma: preliminary clinical observations in 10 patients.
- Multidrug resistance phenotype in patients with chronic lymphocytic leukemia as detected by immunofluorescence (FACS) and northern blot Analysis.
- Thrombosis in a patient with combined homozygosity for the factor V Leiden mutation and a mutation in the 3'-untranslated region of the prothrombin gene.
- Telomeric protein Pin2/TRF1 induces mitotic entry and apoptosis in cells with short telomeres and is down-regulated in human Breast tumors.
- Images in clinical medicine. Waldenström's Macroglobulinemia.
- Inhibition of hematopoiesis by competitive binding of transcription factor PU.1.
- Inhibition of hematopoietic development from embryonic stem cells by antisense vav RNA.
- BEACOPP, a new dose-escalated and accelerated regimen, is at least as effective as COPP/ABVD in patients with advanced-stage Hodgkin's Lymphoma: interim report from a ...
- Phosphorylation-specific prolyl isomerization: is there an underlying theme?
- Pin1 regulates turnover and subcellular localization of beta-catenin by inhibiting its interaction with APC.
- cDNA cloning of a human mRNA preferentially expressed in hematopoietic cells and with homology to a GDP-dissociation inhibitor for the rho GTP-binding proteins.
- Pinning down HER2-ER crosstalk in SMRT regulation.
- Cell-to-cell variability in PI3K protein level regulates PI3K-AKT pathway activity in cell populations.
- Essential role of Pin1 in the regulation of TRF1 stability and telomere maintenance.
- The prolyl isomerase Pin1 restores the function of Alzheimer-associated phosphorylated tau protein.
- Activation of beta-catenin signaling in prostate Cancer by peptidyl-prolyl isomerase Pin1-mediated abrogation of the androgen receptor-beta-catenin interaction.
- PIN1 is an E2F target gene essential for Neu/Ras-induced transformation of mammary epithelial cells.
- Phosphorylation-dependent prolyl isomerization: a novel signaling regulatory mechanism.
- Regulation of NF-kappaB signaling by Pin1-dependent prolyl isomerization and ubiquitin-mediated proteolysis of p65/RelA.
- Prolyl isomerase Pin1: a catalyst for oncogenesis and a potential therapeutic target in Cancer.
- Targeting carcinogenesis: a role for the prolyl isomerase Pin1?
- The prolyl isomerase Pin1 is a novel prognostic marker in human prostate Cancer.
- The prolyl isomerase Pin1 is a regulator of p53 in genotoxic response.
- The prolyl isomerase Pin1 regulates amyloid precursor protein processing and amyloid-beta proDuction.
- Pin1 regulates centrosome duplication, and its overexpression induces centrosome amplification, chromosome instability, and oncogenesis.
- Modeling breast Cancer in vivo and ex vivo reveals an essential role of Pin1 in tumorigenesis.
- Pin1 is overexpressed in breast Cancer and cooperates with Ras signaling in increasing the transcriptional activity of c-Jun towards cyclin D1.
- The prolyl isomerase Pin1 in breast development and Cancer.
- Prolyl isomerase Pin1 is highly expressed in Her2-positive breast Cancer and regulates erbB2 protein stability.
Schools
Westfalische WilhelmsUniversitaet Medizinische Fakultat
St Elizabeth Med Center
Beth Israel Deaconess Med Center
Procedures Preformed
- Thoracentesis
Conditions Treated
- Anal and Rectal Cancer
- Anemia
- Antiphospholipid Syndrome (APS)
- Bile Duct Cancer
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Doctors Specialties
- Hematology
- Medical Oncology
- Oncology
- Internal Medicine
Accepted Insurances
Education
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Beth Israel Deaconess Medical Center -
St Elizabeth's Medical Center -
Westfalischen Landes University Faculty of Medicine
Hospital
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Beth Israel Deaconess Medical Center
Drug Facts
| NPI NUMBER | 1174570402 | |
| NPPES Provider LastName | WULF | |
| NPPES Provider FirstName | GERBURG | |
| NPPES Provider ZIPCode | 02115 | |
| NPPES Provider State | MA | |
| Specialty Description | Hematology | |
| Total Claim Count | 124.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1174570402 |
| Last Name Of The Provider | WULF |
| First Name Of The Provider | GERBURG |
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