Professor, Internal Medicine, University of Kentucky College of Medicine
Pulmonology
Dr. Gerald S. Supinski, MD
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Gerald Supinski is an expert in the field of Pulmonology. He researched medicine at University of Pittsburgh School of Medicine. Gerald Supinski is a published physician. He published 101 publications, including: 'Diaphragmatic lipid peroxidation in chronically loaded rats.' Gerald Supinski accepts Medicare.
Publications
- Calcium-dependent phospholipase A2 Modulates Infection Induced Diaphragm Dysfunction.
- Muscle-Specific Calpastatin Overexpression Prevents Diaphragm Weakness in Cecal Ligation Puncture Induced Sepsis.
- β-Hydroxy-β-methylbutyrate (HMB) prevents sepsis-induced diaphragm dysfunction in mice.
- Prevention and Treatment of ICU-Acquired Weakness: Is There a Stimulating Answer?*.
- Rapid and Complete Recovery in Ventilator Induced Diaphragm Weakness - Problem Solved?
- Calpain activation contributes to endotoxin-induced diaphragmatic dysfunction.
- Sepsis-induced myopathy.
- The JNK MAP kinase pathway contributes to the development of endotoxin-induced diaphragm caspase activation.
- Free radical-mediated skeletal muscle dysfunction in inflammatory conditions.
- The extrinsic caspase pathway modulates endotoxin-induced diaphragm contractile dysfunction.
- Diaphragm and cardiac mitochondrial creatine kinases are impaired in sepsis.
- Caspase activation contributes to endotoxin-induced diaphragm weakness.
- Polyethylene glycol-superoxide dismutase prevents endotoxin-induced cardiac dysfunction.
- Hemin prevents cardiac and diaphragm mitochondrial dysfunction in sepsis.
- Sepsis induces diaphragm electron transport chain dysfunction and protein depletion.
- Diaphragmatic free radical generation increases in an animal model of Heart failure.
- Downregulation of diaphragm electron transport chain and glycolytic enzyme gene expression in sepsis.
- PLA(2) dependence of diaphragm mitochondrial formation of reactive oxygen species.
- Effects of Lung volume on parasternal pressure-generating capacity in dogs.
- Effects of protein kinase A inhibition on rat diaphragm force generation.
- Endotoxin administration alters the force vs. pCa relationship of skeletal muscle fibers.
- Mechanism of expiratory muscle activation during lower thoracic spinal cord stimulation.
- Dichloroacetate reduces diaphragmatic lactate formation but impairs Respiratory performance.
- Free radical-induced contractile protein dysfunction in endotoxin-induced sepsis.
- Alterations in diaphragm strength and fatiguability in congestive Heart failure.
- Effects produced by infusion of a free radical-generating solution into the diaphragm.
- Effect of free radical scavengers on diaphragmatic fatigue.
- Comparison of the effects of endotoxin on limb, Respiratory, and cardiac muscles.
- Glutathione metabolic responses to loaded breathing: variation among Respiratory muscles.
- Extracellular calcium modulates generation of reactive oxygen species by the contracting diaphragm.
- Mechanical contribution of expiratory muscles to pressure generation during spinal cord stimulation.
- Sepsis increases contraction-related generation of reactive oxygen species in the diaphragm.
- Oxypurinol administration fails to prevent free radical-mediated lipid peroxidation during loaded breathing.
- Formation of reactive oxygen species by the contracting diaphragm is PLA(2) dependent.
- Peroxynitrite induces contractile dysfunction and lipid peroxidation in the diaphragm.
- Apocynin improves diaphragmatic function after endotoxin administration.
- Pattern of expiratory muscle activation during lower thoracic spinal cord stimulation.
- Diaphragmatic lipid peroxidation in Chronically loaded rats.
- Free radical induced Respiratory muscle dysfunction.
- Effect of upper respiratory tract infection in patients with neuromuscular Disease.
- Effect of acute fasting on diaphragm strength and endurance.
- Effect of PEG-superoxide dismutase on the diaphragmatic response to endotoxin.
- Analysis of the contraction of series and parallel muscles working against elastic loads.
- Relationship between parasternal and external intercostal muscle length and load compensatory responses in dogs.
- Restrictive ventilatory defect associated with Pulmonary hypertension in mixed connective tissue disease.
- Reversibility of diaphragm fatigue by mechanical hyperperfusion.
- Inspiratory action of separate external and parasternal intercostal muscle contraction.
- Effect of N-acetylcysteine on diaphragm fatigue.
- Effect of codeine on the sensations elicited by loaded breathing.
- Effect of hypoxia on diaphragm blood flow, oxygen uptake, and contractility.
- Control of Respiratory muscle blood flow.
- Effect of varying load magnitude on diaphragmatic glutathione metabolism during loaded breathing.
- Effect of ischemia-reperfusion on diaphragm strength and fatigability.
- Effect of free radical scavengers on endotoxin-induced Respiratory muscle dysfunction.
- Respiratory muscle rest using nasal BiPAP ventilation in patients with stable severe COPD.
- Mechanisms of Disease: are oxygen-derived free radicals involved in diaphragmatic dysfunction?
- Mechanical action of the internal intercostal muscles in dogs.
- Effects of pentobarbital anesthesia on intercostal muscle activation and shortening.
- Effect of varying inspired oxygen concentration on diaphragm glutathione metabolism during loaded breathing.
- N-acetylcysteine administration and loaded breathing.
- Effect of elastase-induced Emphysema on the force-generating ability of the diaphragm.
- Diaphragm structure and function in elastase-induced Emphysema.
- The effect of elastase-induced Emphysema on diaphragmatic muscle structure in hamsters.
- The effects of caffeine and theophylline on diaphragm contractility.
- Caffeine effect on Respiratory muscle endurance and sense of effort during loaded breathing.
- Effect of alterations in muscle fiber length on diaphragm blood flow.
- Comparative effects of theophylline and adenosine on Respiratory skeletal and smooth muscle.
- Changes in chest wall structure and elasticity in elastase-induced Emphysema.
- Functional adaptation of diaphragm to Chronic hyperinflation in emphysematous hamsters.
- Effect of inspiratory muscle fatigue on perception of effort during loaded breathing.
- Relationship of changes in diaphragmatic muscle blood flow to muscle contractile activity.
- Respiratory muscle blood flow.
- Effect of amrinone on diaphragm blood flow.
- Effect of theophylline on the velocity of diaphragmatic muscle shortening.
- Effect of diaphragmatic contraction on intramuscular pressure and vascular impedance.
- Alterations in Respiratory muscle activation in the ischemic fatigued canine diaphragm.
- Artificial ventilation by means of electrical activation of the intercostal/accessory muscles alone in anesthetized dogs.
- Effects of progressive hypoxia on parasternal, costal, and crural diaphragm activation.
- Inspiratory muscle interaction in the generation of changes in airway pressure.
- Tissue release of adenosine triphosphate degradation products during shock in dogs.
- Mechanical action of the interosseous intercostal muscles as a function of Lung volume.
- Action of the intercostal muscles on the rib cage.
- Parasternal and external intercostal muscle shortening during eupneic breathing.
- Effect of norepinephrine on diaphragm contractility and blood flow.
- Contractile and endurance properties of geniohyoid and diaphragm muscles.
- Parasternal and external intercostal responses to various Respiratory maneuvers.
- Efficacy of combined inspiratory intercostal and expiratory muscle pacing to maintain artificial ventilation.
- N-acetylcysteine administration alters the response to inspiratory loading in oxygen-supplemented rats.
- Effects of intraphrenic injection of potassium on diaphragm activation.
- Failure of vasodilator administration to increase blood flow to the fatiguing diaphragm.
- Reflex control of diaphragm activation by thoracic afferents.
- Evaluation of intercostal pacing to provide artificial ventilation in quadriplegics.
- Electrical activation of the expiratory muscles to restore cough.
- Free radicals alter maximal diaphragmatic mitochondrial oxygen consumption in endotoxin-induced sepsis.
- Modulation of release of reactive oxygen species by the contracting diaphragm.
- Effect of proteasome inhibitors on endotoxin-induced diaphragm dysfunction.
- Caspase and calpain activation both contribute to sepsis-induced diaphragmatic weakness.
- MitoQ administration prevents endotoxin-induced cardiac dysfunction.
- Double-stranded RNA-dependent protein kinase activation modulates endotoxin-induced diaphragm weakness.
- Diaphragm weakness and mechanical ventilation--what''s the critical issue?
Schools
University of Pittsburgh School of Medicine
Procedures Preformed
- Bronchoscopy
- Thoracentesis
- Ventilation Management
Conditions Treated
- Acidosis
- Alkalosis
- Atelectasis
- Bronchiectasis
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Doctors Specialties
- Critical Care Medicine
- Pulmonology
Accepted Insurances
Education
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Case Western Reserve University - University Hospitals -
University of Pittsburgh School of Medicine
Hospital
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University of Kentucky Albert B. Chandler Hospital
Drug Facts
| NPI NUMBER | 1902991565 | |
| NPPES Provider LastName | SUPINSKI | |
| NPPES Provider FirstName | GERALD | |
| NPPES Provider ZIPCode | 405360248 | |
| NPPES Provider State | KY | |
| Specialty Description | Pulmonary Disease | |
| Total Claim Count | 14.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1902991565 |
| Last Name Of The Provider | SUPINSKI |
| First Name Of The Provider | GERALD |
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