Assistant Professor, Internal Medicine, Washington University School of Medicine
Nephrology
Dr. George Jarad, MD
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Dr. George Jarad works in the field of Nephrology. George Jarad is also a published doctor. He has 22 publication published. The most recent publication is: Forced expression of laminin {beta}1 in podocytes prevents nephrotic syndrome in mice lacking laminin {beta}2, a model for Pierson syndrome. He accepts Medicare.
Publications
- Forced expression of laminin {beta}1 in podocytes prevents nephrotic syndrome in mice lacking laminin {beta}2, a model for Pierson syndrome.
- Dystroglycan does not contribute significantly to Kidney development or function, in health or after injury.
- Activation of NFAT signaling in podocytes causes glomerulosclerosis.
- A new function for parietal epithelial cells: a second glomerular barrier.
- Update on the glomerular filtration barrier.
- Albuminuria, wherefore art thou?
- The Pax3-Cre transgene exhibits a rostrocaudal gradient of expression in the skeletal muscle lineage.
- Laminins promote postsynaptic maturation by an autocrine mechanism at the neuromuscular junction.
- Podocyte-specific deletion of dicer alters cytoskeletal dynamics and causes glomerular Disease.
- Beta1 integrin expression by podocytes is required to maintain glomerular structural integrity.
- Disruption of glomerular basement membrane charge through podocyte-specific mutation of agrin does not alter glomerular permselectivity.
- Proteinuria precedes podocyte abnormalities inLamb2-/- mice, implicating the glomerular basement membrane as an albumin barrier.
- Beta8 integrin binds Rho GDP dissociation inhibitor-1 and activates Rac1 to inhibit mesangial cell myofibroblast differentiation.
- Podocyte-specific deletion of integrin-linked kinase results in severe glomerular basement membrane alterations and progressive glomerulosclerosis.
- Transgenic isolation of skeletal muscle and Kidney defects in laminin beta2 mutant mice: implications for Pierson syndrome.
- Renal phenotype is exacerbated in Os and lpr double mutant mice.
- The NHE1 Na+/H+ exchanger recruits ezrin/radixin/moesin proteins to regulate Akt-dependent cell survival.
- Nucleic acid-based techniques for post-transcriptional regulation of molecular targets.
- Renal tubular epithelial cell apoptosis is associated With caspase cleavage of the NHE1 Na+/H+ exchanger.
- Fas activation induces Renal tubular epithelial cell beta 8 integrin expression and function in the absence of apoptosis.
- Apoptosis and JNK activation are differentially regulated by Fas expression level in Renal tubular epithelial cells.
Procedures Preformed
- Hemodialysis
- Peritoneal Dialysis
Conditions Treated
- Acidosis
- Acute Glomerulonephritis
- Acute Kidney Failure
- Calcium Metabolism Disorders
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Doctors Specialties
- Nephrology
Accepted Insurances
Education
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Case Western Reserve University - MetroHealth -
Damascus University Faculty of Medicine
Hospital
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Barnes-Jewish Hospital
Drug Facts
| NPI NUMBER | 1942409529 | |
| NPPES Provider LastName | JARAD | |
| NPPES Provider FirstName | GEORGE | |
| NPPES Provider ZIPCode | 631101032 | |
| NPPES Provider State | MO | |
| Specialty Description | Nephrology | |
| Total Claim Count | 658.0 | |
| Distinct Opioid Count | 1.0 | |
| Opioid Claim Count | 13.0 | |
| Percent Opioid Claims | 1.98 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1942409529 |
| Last Name Of The Provider | JARAD |
| First Name Of The Provider | GEORGE |
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