Professor, Internal Medicine, University of Michigan Medical School
Nephrology
Dr. Frank C. Brosius, MD
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Doctor Frank C. Brosius, MD is an expert in the field of Nephrology. He attended University of Kansas School of Medicine. He over the years received 2 awards: "Fellow" and "CMS Meaningful Use Stage 1 Certification". Doctor Frank C. Brosius, MD has written publications. He has 42 publications published with PubMed. The lastest publication is: 'Transcriptional networks of murine diabetic peripheral neuropathy and nephropathy: common and distinct gene expression patterns.' Frank Brosius accepts Medicare payments and is listed with Medicare.gov.
Publications
- Structural Predictors of Loss of Renal Function in American Indians with Type 2 Diabetes.
- Maintenance of GLUT4 expression in smooth muscle prevents Hypertension-induced changes in vascular reactivity.
- JAK inhibition and progressive Kidney disease.
- Podocytes, Signaling Pathways, and Vascular Factors in Diabetic Kidney Disease.
- How to find a prognostic biomarker for progressive diabetic Nephropathy.
- New targets for treatment of diabetic Nephropathy: what we have learned from animal models.
- Identification of cross-species shared transcriptional networks of diabetic Nephropathy in human and mouse glomeruli.
- Perspectives on systems biology applications in diabetic Kidney disease.
- GLUT1 ENHANCES mTOR ACTIVITY INDEPENDENTLY OF TSC2 AND AMPK.
- Susceptible mice: identifying a diabetic nephropathy Disease locus using a murine model.
- Understanding Kidney disease: toward the integration of regulatory networks across species.
- Podocyte-specific overexpression of GLUT1 surprisingly reduces mesangial matrix expansion in diabetic Nephropathy in mice.
- A new pair of SOCS for diabetic Nephropathy.
- The management of diabetic neuropathy in CKD.
- Abnormalities in signaling pathways in diabetic Nephropathy.
- GLUT1-induced cFLIP expression promotes proliferation and prevents apoptosis in vascular smooth muscle cells.
- The peroxisome proliferator-activated receptor gamma agonist pioglitazone improves cardiometabolic risk and Renal inflammation in murine lupus.
- A rapid, PPAR-gamma-dependent effect of pioglitazone on the phosphorylation of MYPT.
- Enhanced expression of Janus kinase-signal transducer and activator of transcription pathway members in human diabetic Nephropathy.
- New insights into the mechanisms of fibrosis and sclerosis in diabetic Nephropathy.
- Is the ER stressed out in diabetic Kidney disease?
- Rosiglitazone treatment reduces diabetic neuropathy in streptozotocin-treated DBA/2J mice.
- Rosiglitazone reduces Renal and plasma markers of oxidative injury and reverses urinary metabolite abnormalities in the amelioration of diabetic nephropathy.
- A GSK-3/TSC2/mTOR pathway regulates glucose uptake and GLUT1 glucose transporter expression.
- From fibrosis to sclerosis: mechanisms of glomerulosclerosis in diabetic Nephropathy.
- Preserved expression of GLUT4 prevents enhanced agonist-induced vascular reactivity and MYPT1 phosphorylation in Hypertensive mouse aorta.
- Identification of the glomerular podocyte as a target for growth hormone action.
- Hypotension, lipodystrophy, and insulin resistance in generalized PPARgamma-deficient mice rescued from embryonic lethality.
- Increased JNK phosphorylation and oxidative stress in response to increased glucose flux through increased GLUT1 expression in rat retinal endothelial cells.
- GLUT4 facilitative glucose transporter specifically and differentially contributes to agonist-induced vascular reactivity in mouse aorta.
- Diabetic Nephropathy: of mice and men.
- Effects of PPAR-gamma ligands on vascular smooth muscle marker expression in Hypertensive and normal arteries.
- Induction of antioxidant enzymes in murine podocytes precedes injury by puromycin aminonucleoside.
- Immunolocalization of the proton-coupled oligopeptide transporter PEPT2 in developing rat brain.
- Glucose transporter-1-deficient mice exhibit impaired development and deformities that are similar to diabetic embryopathy.
- Trophic factors and cytokines in early diabetic glomerulopathy.
- PI3-kinase-induced hyperreactivity in DOCA-salt Hypertension is independent of GSK-3 activity.
- Targeted disruption of the PEPT2 gene markedly reduces dipeptide uptake in choroid plexus.
- Evidence for a novel TGF-beta1-independent mechanism of fibronectin production in mesangial cells overexpressing glucose transporters.
- Enhanced glycogen synthase kinase-3beta activity mediates hypoxia-induced apoptosis of vascular smooth muscle cells and is prevented by glucose transport and Metabolism.
- Transcriptional networks of murine diabetic peripheral neuropathy and Nephropathy: common and distinct gene expression patterns.
Schools
University of Kansas School of Medicine
University Of Michigan
National Institute Of Health
Procedures Preformed
- Hemodialysis
Conditions Treated
- Acidosis
- Acute Glomerulonephritis
- Acute Kidney Failure
- Alkalosis
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Doctors Specialties
- Nephrology
Accepted Insurances
Awards
- Fellow
- CMS Meaningful Use Stage 1 Certification
Education
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Beth Israel Deaconess Medical Center -
University of Michigan Hospitals and Health Centers -
University of Kansas School of Medicine
Hospital
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University of Michigan Hospitals and Health Centers
Drug Facts
| NPI NUMBER | 1336221217 | |
| NPPES Provider LastName | BROSIUS | |
| NPPES Provider FirstName | FRANK | |
| NPPES Provider ZIPCode | 481095364 | |
| NPPES Provider State | MI | |
| Specialty Description | Nephrology | |
| Total Claim Count | 464.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1336221217 |
| Last Name Of The Provider | BROSIUS |
| First Name Of The Provider | FRANK |
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