Professor, Pediatrics and Communicable Diseases, University of Michigan Medical School
Neurology
Dr. Faye S. Silverstein, MD
Faye Silverstein works in the field of Neurology. Faye Silverstein is a published doctor as well. He has 98 publications published. The latest was: Excitotoxic brain injury stimulates expression of the chemokine receptor CCR5 in neonatal rats.
Publications
- Efficacy Outcome Selection in the Therapeutic Hypothermia After Pediatric Cardiac Arrest Trials.
- Rationale, Timeline, Study Design, and Protocol Overview of the Therapeutic Hypothermia After Pediatric Cardiac Arrest Trials.
- Bumetanide augments the neuroprotective efficacy of phenobarbital plus hypothermia in a neonatal hypoxia-ischemia model.
- Phenobarbital augments hypothermic neuroprotection.
- Do seizures contribute to neonatal hypoxic-ischemic brain injury?
- Off-label use of antiepileptic drugs for the treatment of neonatal seizures.
- Improving the treatment of neonatal seizures: National Institute of Neurological Disorders and Stroke workshop report.
- Neonatal seizures: multicenter variability in current treatment practices.
- Replacing neocortical neurons after stroke.
- Plasticity of neurons and glia following neonatal hypoxic-ischemic brain injury in rats.
- Hypoxic-ischemic injury stimulates subventricular zone proliferation and neurogenesis in the neonatal rat.
- Neonatal hypoxic-ischemic injury increases forebrain subventricular zone neurogenesis in the mouse.
- Topiramate extends the therapeutic window for hypothermia-mediated neuroprotection after stroke in neonatal rats.
- New oligodendrocytes are generated after neonatal hypoxic-ischemic brain injury in rodents.
- Complement activation contributes to hypoxic-ischemic brain injury in neonatal rats.
- Developmental changes in the expression of chemokine receptor CCR1 in the rat cerebellum.
- Hypoxic-ischemic injury induces macrophage inflammatory protein-1alpha expression in immature rat brain.
- Hypoxic-ischemic oligodendroglial injury in neonatal rat brain.
- Excitatory amino acids contribute to the pathogenesis of perinatal hypoxic-ischemic brain injury.
- The microsomal calcium-ATPase inhibitor thapsigargin is a neurotoxin in perinatal rodent brain.
- Excitotoxic brain injury suppresses striatal high-affinity glutamate uptake in perinatal rats.
- The glutamate analogue quisqualic acid is neurotoxic in striatum and hippocampus of immature rat brain.
- Neurotoxicity of N-methyl-D-aspartate is markedly enhanced in developing rat central nervous system.
- A model of methotrexate encephalopathy: neurotransmitter and pathologic abnormalities.
- Flunarizine limits hypoxia-ischemia induced morphologic injury in immature rat brain.
- Muscarinic cholinergic receptors in human infant forebrain: [3H]quinuclidinyl benzilate binding in homogenates and quantitative autoradiography in sections.
- Hypoxia-ischemia stimulates hippocampal glutamate efflux in perinatal rat brain: an in vivo microdialysis study.
- Transient hypoxia alters striatal catecholamine metabolism in immature brain: an in vivo microdialysis study.
- Effects of perinatal stroke on striatal amino acid efflux in rats studied with in vivo microdialysis.
- Effect of depolarization on striatal amino acid efflux in perinatal rats: an in vivo microdialysis study.
- Effects of hypoxia-ischemia and MK-801 treatment on the binding of a phencyclidine analogue in the developing rat brain.
- Hypoglycemia alters striatal amino acid efflux in perinatal rats: an in vivo microdialysis study.
- Magnesium reduces N-methyl-D-aspartate (NMDA)-mediated brain injury in perinatal rats.
- Systemic administration of MK-801 protects against N-methyl-D-aspartate- and quisqualate-mediated neurotoxicity in perinatal rats.
- MK-801 pretreatment enhances N-methyl-D-aspartate-mediated brain injury and increases brain N-methyl-D-aspartate recognition site binding in rats.
- Cerebrospinal fluid biogenic amine metabolites in children during treatment for acute lymphocytic leukemia.
- Effects of intraventricular methotrexate on cerebrospinal fluid monoamine metabolites in rhesus monkeys.
- Purine and monoamine metabolites in cerebrospinal fluid.
- Cerebrospinal fluid cyclic nucleotide alterations in the Lesch-Nyhan syndrome.
- Lesch-Nyhan syndrome: CSF neurotransmitter abnormalities.
- Neuroprotective effects of MK-801, TCP, PCP and CPP against N-methyl-D-aspartate induced neurotoxicity in an in vivo perinatal rat model.
- HA-966 (1-hydroxy-3-aminopyrrolidone-2) selectively reduces N-methyl-D-aspartate (NMDA)-mediated brain damage.
- Quantitative assessment of neuroprotection against NMDA-induced brain injury.
- Effect of glycine and glycine receptor antagonists on NMDA-induced brain injury.
- MK-801 protects the neonatal brain from hypoxic-ischemic damage.
- Perinatal hypoxic-ischemic brain injury enhances quisqualic acid-stimulated phosphoinositide turnover.
- New insights into mechanisms of neuronal damage in the developing brain.
- Perinatal hypoxia-ischemia disrupts striatal high-affinity [3H]glutamate uptake into synaptosomes.
- Evidence for transient perinatal glutamatergic innervation of globus pallidus.
- Hypoxia-ischemia produces focal disruption of glutamate receptors in developing brain.
- Risks of neuroleptic drugs in children.
- Glutamate recognition sites in human fetal brain.
- Hematological monitoring during therapy with carbamazepine in children.
- Adverse behavioral reactions in children treated with carbamazepine (Tegretol).
- The mitochondrial inhibitor malonate enhances NMDA toxicity in the neonatal rat striatum.
- The glutamate uptake inhibitor L-trans-2,4-pyrrolidine dicarboxylate is neurotoxic in neonatal rat brain.
- Inhibition of nitric oxide synthase activity attenuates striatal malonate lesions in rats.
- gp120 and neurotoxicity in vivo.
- gp120, an HIV-1 protein, increases susceptibility to hypoglycemic and ischemic brain injury in perinatal rats.
- Cerebral hypoxia-ischemia stimulates cytokine gene expression in perinatal rats.
- Postvaricella basal ganglia infarction in children.
- Acute disruption of cytochrome oxidase activity in brain in a perinatal rat stroke model.
- Resistance to nitroprusside neurotoxicity in perinatal rat brain.
- Hypoxic-ischemic brain injury stimulates glial fibrillary acidic protein mRNA and protein expression in neonatal rats.
- Potentiation of N-methyl-D-aspartate-mediated brain injury by a human immunodeficiency virus-1-derived peptide in perinatal rodents.
- Excitotoxic injury stimulates glial fibrillary acidic protein mRNA expression in perinatal rat brain.
- Hypoxic-ischemic injury acutely disrupts microtubule-associated protein 2 immunostaining in neonatal rat brain.
- Hypoxic-ischemic brain injury induces an acute microglial reaction in perinatal rats.
- Intracerebral NMDA injection stimulates production of interleukin-1 beta in perinatal rat brain.
- Adenovirus-mediated over-expression of interleukin-1 receptor antagonist reduces susceptibility to excitotoxic brain injury in perinatal rats.
- The platelet-activating factor antagonist BN 52021 attenuates hypoxic-ischemic brain injury in the immature rat.
- gp120, a human immunodeficiency virus-1 coat protein, augments excitotoxic hippocampal injury in perinatal rats.
- Neutrophils and reactive oxygen intermediates mediate glucan-induced pulmonary granuloma formation through the local induction of monocyte chemoattractant protein-1.
- Cytokines and perinatal brain injury.
- Hypoxic-ischemic injury induces monocyte chemoattractant protein-1 expression in neonatal rat brain.
- Role of glutamate receptor-mediated excitotoxicity in bilirubin-induced brain injury in the Gunn rat model.
- Can inhibition of apoptosis rescue ischemic brain?
- Excitotoxic injury induces monocyte chemoattractant protein-1 expression in neonatal rat brain.
- Excitotoxic brain injury stimulates expression of the chemokine receptor CCR5 in neonatal rats.
- Tat, a human immunodeficiency virus-1-derived protein, augments excitotoxic hippocampal injury in neonatal rats.
- Expanding the scope of child neurology for the 21st century.
- Mice deficient in interleukin-1 converting enzyme are resistant to neonatal hypoxic-ischemic brain damage.
- Inhibition of TNF-alpha can attenuate or exacerbate excitotoxic injury in neonatal rat brain.
- Experimental gliosarcoma induces chemokine receptor expression in rat brain.
- Acute excitotoxic injury induces expression of monocyte chemoattractant protein-1 and its receptor, CCR2, in neonatal rat brain.
- Monocyte chemoattractant protein-1 is a mediator of acute excitotoxic injury in neonatal rat brain.
- Hypoxic-ischemic injury results in acute disruption of myelin gene expression and death of oligodendroglial precursors in neonatal mice.
- Attenuation of hypoxia-ischemia-induced monocyte chemoattractant protein-1 expression in brain of neonatal mice deficient in interleukin-1 converting enzyme.
- Successful use of rt-PA in pediatric stroke.
- AMPA-Induced suppression of oligodendroglial gene expression in neonatal rat brain.
- In-hospital versus out-of-hospital pediatric cardiac arrest: a multicenter cohort study.
- Hypothermia and other treatment options for neonatal encephalopathy: an executive summary of the Eunice Kennedy Shriver NICHD workshop.
- Impact of indolent inflammation on neonatal hypoxic-ischemic brain injury in mice.
- Microglial expression of chemokine receptor CCR5 during rat forebrain development and after perinatal hypoxia-ischemia.
- Pathogenesis of hypoxic-ischemic brain injury in a perinatal rodent model.
- Cerebrospinal fluid monoamine metabolites in patients with infantile spasms.
- Effects of hypoxia-ischemia on monoamine metabolism in the immature brain.
Doctors Specialties
- Pediatrics
- Neurology
Accepted Insurances
- Choice Plus POS II
- Amerigroup (Wellpoint)
- Blue Card PPO
- Blue Cross Blue Shield of Illinois PPO
- Group Employees PPO
- Cigna HMO
- Cigna PPO
- Great West Healthcare-Cigna PPO
- Open Access Plus
- Cofinity PPO
- Commercial Insurance Company
- First Health (Coventry Health Care) PPO
- Health Alliance
- Preferred PPO
- Choice Care PPO
- Medicaid
- Medicare MCR
- MultiPlan PPO
- PHCS Network PPO
- NGS CoreSource
- Paramount
- Priority PPO
- Tricare
- Choice Plus POS
- Options PPO
- United Healthcare
Education
-
University of Michigan Hospitals and Health Centers -
McGill University Faculty of Medicine
Hospital
-
University of Michigan Hospitals and Health Centers
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