Associate Professor, Neurology, University of Alabama School of Medicine
Neurology
Dr. Erik D. Roberson, MD
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Doctor Erik D. Roberson, MD works in the field of Neurology. He graduated from Baylor College of Medicine. Erik D. Roberson is also a published doctor. He has 38 paper published. The most recent paper is: Protected-site phosphorylation of protein kinase C in hippocampal long-term potentiation. Erik Roberson is registered with Medicare.
Publications
- DNA methylation slows effects of C9orf72 mutations: An epigenetic brake on genetic inheritance.
- Tau-Mediated NMDA Receptor Impairment Underlies Dysfunction of a Selectively Vulnerable Network in a Mouse Model of Frontotemporal Dementia.
- MicroRNA-124 modulates social behavior in frontotemporal Dementia.
- Rarity of the Alzheimer Disease-Protective APP A673T Variant in the United States.
- Effects of Multiple Genetic Loci on Age at Onset in Late-Onset Alzheimer Disease: A Genome-Wide Association Study.
- Early retinal neurodegeneration and impaired Ran-mediated nuclear import of TDP-43 in progranulin-deficient FTLD.
- AlphaScreen HTS and Live-Cell Bioluminescence Resonance Energy Transfer (BRET) Assays for Identification of Tau-Fyn SH3 Interaction Inhibitors for Alzheimer Disease.
- Davunetide in patients with Progressive supranuclear palsy: a randomised, double-blind, placebo-controlled phase 2/3 trial.
- Seizure resistance without parkinsonism in aged mice after tau reduction.
- The dendritic hypothesis for Alzheimer's Disease pathophysiology.
- Dissociation of Frontotemporal Dementia-Related Deficits and Neuroinflammation in Progranulin Haploinsufficient Mice.
- The advantages of frontotemporal degeneration drug development (part 2 of frontotemporal degeneration: The next therapeutic frontier).
- Frontotemporal degeneration, the next therapeutic frontier: Molecules and animal models for frontotemporal degeneration drug development.
- Mouse models of frontotemporal Dementia.
- Usp14 Deficiency Increases Tau Phosphorylation without Altering Tau Degradation or Causing Tau-Dependent Deficits.
- Mouse models of Alzheimer's Disease.
- Geriatric Epilepsy: research and clinical directions for the future.
- Common variants at MS4A4/MS4A6E, CD2AP, CD33 and EPHA1 are associated with late-onset Alzheimer's Disease.
- Amyloid-β/Fyn-induced synaptic, network, and cognitive impairments depend on tau levels in multiple mouse models of Alzheimer's Disease.
- Step-by-step in situ hybridization method for localizing gene expression changes in the Brain.
- Quantifying biomarkers of cognitive dysfunction and neuronal network hyperexcitability in mouse models of Alzheimer's Disease: depletion of calcium-dependent proteins ...
- Contemporary approaches to Alzheimer's Disease and frontotemporal dementia.
- Beyond diagnosis: what biomarkers are teaching us about the "bio"logy of Alzheimer Disease.
- Loss of Hsp70 exacerbates pathogenesis but not levels of fibrillar aggregates in a mouse model of Huntington's Disease.
- Enkephalin elevations contribute to neuronal and behavioral impairments in a transgenic mouse model of Alzheimer's Disease.
- Aberrant excitatory neuronal activity and compensatory remodeling of inhibitory hippocampal circuits in mouse models of Alzheimer's Disease.
- Reducing endogenous tau ameliorates amyloid beta-induced deficits in an Alzheimer's Disease mouse model.
- 100 years and counting: prospects for defeating Alzheimer's Disease.
- Antiamyloidogenic and neuroprotective functions of cathepsin B: implications for Alzheimer's Disease.
- Frontotemporal lobar degeneration: demographic characteristics of 353 patients.
- PolyNeuropathy following gastric bypass surgery.
- Memory-forming chemical reactions.
- Protected-site phosphorylation of protein kinase C in hippocampal long-term potentiation.
- The mitogen-activated protein kinase cascade couples PKA and PKC to cAMP response element binding protein phosphorylation in area CA1 of hippocampus.
- Mice lacking ataxin-1 display learning deficits and decreased hippocampal paired-pulse facilitation.
- Abnormal social behaviors in mice lacking Fgf17.
- Frontotemporal dementia progresses to death faster than Alzheimer Disease.
Schools
Baylor University
Baylor College Of Med
University Ca Sf School Of Med
Procedures Preformed
- EEG (Electroencephalogram)
Conditions Treated
- Alzheimer's Disease
- Anterior Horn Disease
- Ataxia
- Brain and Nervous System Cancer (incl. Gliomas, Astrocytoma, Schwannoma, Medulloblastoma, Chordoma)
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Doctors Specialties
- Neurology
Accepted Insurances
- Aetna
- Blue Card PPO
- Dental Blue 100/200/300
- Great West Healthcare-Cigna PPO
- First Health (Coventry Health Care) PPO
- Choice Care PPO
- Medicare MCR
- MultiPlan PPO
- Choice Plus POS
- Navigate HMO
- Navigate POS
- Options PPO
Education
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UCSF -
Baylor College of Medicine
Hospital
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University of Alabama Hospital
Drug Facts
| NPI NUMBER | 1063437382 | |
| NPPES Provider LastName | ROBERSON | |
| NPPES Provider FirstName | ERIK | |
| NPPES Provider ZIPCode | 352491900 | |
| NPPES Provider State | AL | |
| Specialty Description | Neurology | |
| Total Claim Count | 394.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1063437382 |
| Last Name Of The Provider | ROBERSON |
| First Name Of The Provider | ERIK |
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