Assistant Professor, Medicine and Pediatrics, Indiana University School of Medicine
Endocrinology
Dr. Erik A. Imel, MD
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Erik Imel works in the field of Endocrinology. He attended Indiana University School of Medicine. Indiana University School of Medicine is ranked 45/47 in Research/PrimaryCare. Erik A. Imel is also a published doctor. He has 27 paper published. The most recent paper is: Serum fibroblast growth factor 23, serum iron and bone mineral density in premenopausal women. He accepts Medicare.
Publications
- Genome-wide association study of serum iron phenoTypes in premenopausal women of European descent.
- Disparities in Osteoporosis treatments.
- Pharmacokinetics and pharmacodynamics of a human monoclonal anti-FGF23 antibody (KRN23) in the first multiple ascending dose trial treating adults with X-linked hypop...
- Proportion of osteoporotic women remaining at risk for fracture despite adherence to oral bisphosphonates.
- Hyperphosphatemic Familial Tumoral Calcinosis: Genetic Models of Deficient FGF23 Action.
- Iron and fibroblast Growth factor 23 in X-linked hypophosphatemia.
- The changing face of hypophosphatemic Disorders in the FGF-23 era.
- Fluorosis Because of Prolonged Voriconazole Therapy in a Teenager With Acute Myelogenous Leukemia.
- Iron Modifies Plasma FGF23 Differently in Autosomal Dominant Hypophosphatemic Rickets and Healthy Humans.
- A clinician's guide to X-linked hypophosphatemia.
- Establishment of sandwich ELISA for soluble alpha-Klotho measurement: Age-dependent change of soluble alpha-Klotho levels in healthy subjects.
- Treatment of X-linked hypophosphatemia with calcitriol and phosphate increases circulating fibroblast Growth factor 23 concentrations.
- Clinical variability of familial tumoral calcinosis caused by novel GALNT3 mutations.
- Phosphaturic mesenchymal tumor, mixed connective tissue variant, of the mandible: report of a case and review of the literature.
- Genetics of familial tumoral calcinosis.
- Mutational survey of the PHEX gene in patients with X-linked hypophosphatemic rickets.
- A homozygous missense mutation in human KLOTHO causes severe tumoral calcinosis.
- Novel GALNT3 mutations causing hyperostosis-hyperphosphatemia syndrome result in low intact fibroblast Growth factor 23 concentrations.
- FGF23 concentrations vary with Disease status in autosomal dominant hypophosphatemic rickets.
- Tumoral calcinosis presenting with eyelid calcifications due to novel missense mutations in the glycosyl transferase domain of the GALNT3 gene.
- Intronic deletions in the SLC34A3 gene cause hereditary hypophosphatemic rickets with hypercalciuria.
- Sensitivity of fibroblast Growth factor 23 measurements in tumor-induced osteomalacia.
- Fibroblast growth factor 23: roles in health and Disease.
- Miscellaneous non-inflammatory musculoskeletal conditions. Hyperphosphatemic familial tumoral calcinosis (FGF23, GALNT3 and αKlotho).
- Fibrous dysplasia, phosphate wasting and fibroblast Growth factor 23.
- Serum fibroblast Growth factor 23, serum iron and bone mineral density in premenopausal women.
Schools
University Of Cincinnati College Of Medicine
University Of Rochester
Indiana University School Of Med
Conditions Treated
- Abnormal Thyroid
- Adrenal Insufficiency
- Adrenogenital Disorders
- Calcium Metabolism Disorders
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Doctors Specialties
- Pediatric Endocrinology
- Endocrinology
Accepted Insurances
Education
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Indiana University School of Medicine -
University of Rochester -
University of Cincinnati College of Medicine
Hospital
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Riley Hospital for Children at Indiana University Health
Drug Facts
| NPI NUMBER | 1326178971 | |
| NPPES Provider LastName | IMEL | |
| NPPES Provider FirstName | ERIK | |
| NPPES Provider ZIPCode | 462025149 | |
| NPPES Provider State | IN | |
| Specialty Description | Internal Medicine | |
| Total Claim Count | 199.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1326178971 |
| Last Name Of The Provider | IMEL |
| First Name Of The Provider | ERIK |
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