Associate Professor, Internal Medicine, Ohio State University College of Medicine
Internal Medicine
Dr. Elliott D. Crouser, MD
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Elliott Crouser works in the field of Internal Medicine. He attended University of Toledo College of Medicine. He over the years received 1 awards: "CMS Meaningful Use Stage 1 Certification". Elliott D. Crouser, MD is also published. He has 56 publications published. The lastest publication: "Ischemia/reperfusion injury to the ileum does not account for the ileal VO2-DO2 alterations induced by endotoxin.' He is registered with Medicare and accepts Medicare payments.
Publications
- Mathematical model of Sarcoidosis.
- Current best practice in the management of Pulmonary and systemic sarcoidosis.
- Improved detection of cardiac Sarcoidosis using magnetic resonance with myocardial t2 mapping.
- Nonischemic myocardial changes detected by cardiac magnetic resonance in critical care patients with sepsis.
- Nicotine treatment improves Toll-like receptor 2 and Toll-like receptor 9 responsiveness in active Pulmonary sarcoidosis.
- Sustained reduction of central line-associated bloodstream infections in an intensive care unit using a top-down and bottom-up approach.
- Quantitative computerized two-point correlation analysis of Lung CT scans correlates with pulmonary function in pulmonary sarcoidosis.
- At-risk but viable myocardium in a large animal model of non ST-segment elevation acute coronary syndrome: cardiovascular magnetic resonance with ex vivo validation.
- Mitochondrial Transcription Factor A, an Endogenous Danger Signal, Promotes TNFα Release via RAGE- and TLR9-Responsive Plasmacytoid Dendritic Cells.
- Relationship of substrate level to turnover rate in fasted adult and newborn dogs.
- Herpes simplex type 2 causing fulminant hepatic Failure.
- Sepsis: links between pathogen sensing and organ damage.
- Mitochondrial mechanisms of sepsis-induced organ Failure.
- Flavopiridol causes early mitochondrial damage in Chronic lymphocytic leukemia cells with impaired oxygen consumption and mobilization of intracellular calcium.
- Suppression of monocyte metabolism by septic plasma: revisiting the concept of "blood poisoning".
- Considering an infectious etiology of Sarcoidosis.
- Transformation of Pulmonary histoplasmosis to sarcoidosis: a case report.
- Carbamoyl phosphate synthase-1: a marker of mitochondrial damage and depletion in the liver during sepsis.
- A proteomic analysis of liver mitochondria during acute endotoxemia.
- Calcineurin regulates myocardial function during acute endotoxemia.
- Mitochondrial iPLA2 activity modulates the release of cytochrome c from mitochondria and influences the permeability transition.
- Respiratory failure during critical illness: are mitochondria to blame?
- Mitochondrial dysfunction in septic shock and multiple organ dysfunction syndrome.
- Is cell death a prerequisite for cardiac dysfunction during sepsis?
- Redefining the roles of apoptosis pathways during sepsis.
- Toxicity of Food Drug and Cosmetic Blue No. 1 dye in critically ill patients.
- Therapeutic benefits of antioxidants during sepsis: is protection against oxidant-mediated tissue damage only half the story?
- Abnormal permeability of inner and outer mitochondrial membranes contributes independently to mitochondrial dysfunction in the liver during acute endotoxemia.
- Quantitation of cytochrome c release from rat liver mitochondria.
- Cyclosporin A ameliorates mitochondrial ultrastructural injury in the ileum during acute endotoxemia.
- Pore formation and uncoupling initiate a Ca2+-independent degradation of mitochondrial phospholipids.
- Endotoxin-induced mitochondrial damage correlates with impaired Respiratory activity.
- Warming up to hypothermia for treatment of severe sepsis*.
- Differential expression of microRNA and predicted targets in Pulmonary sarcoidosis.
- Intestinal epithelium is more susceptible to cytopathic injury and altered permeability than the Lung epithelium in the context of acute sepsis.
- An in silico modeling approach to understanding the dynamics of Sarcoidosis.
- Mitochondrial dysfunction during sepsis: still more questions than answers.
- Mitochondrial mechanisms are at the 'heart' of novel ischemia-reperfusion therapies.
- Formyl peptide receptor ligands promote wound closure in Lung epithelial cells.
- Peroxisome proliferator-activated receptors gamma coactivator-1alpha: master regulator of mitochondrial biogenesis and survival during critical illness?
- The CD4+ lymphopenic Sarcoidosis phenotype is highly responsive to anti-tumor necrosis factor-{alpha} therapy.
- Altered immune surveillance: a common link between Cancer and sepsis?
- Hypothesis: sarcoidosis is a STAT1-mediated Disease.
- Monocyte activation by necrotic cells is promoted by mitochondrial proteins and formyl peptide receptors.
- Stem cell therapy: A "magic bullet" for cardiomyopathies?
- Zinc deficiency increases organ damage and mortality in a murine model of polymicrobial sepsis.
- Gene expression profiling identifies MMP-12 and ADAMDEC1 as potential pathogenic mediators of Pulmonary sarcoidosis.
- Endotoxin-induced ileal mucosal injury and nitric oxide dysregulation are temporally dissociated.
- Ileal VO(2)-O(2) alterations induced by endotoxin correlate with severity of mitochondrial injury.
- Ischemia/reperfusion injury to the ileum does not account for the ileal VO2-DO2 alterations induced by endotoxin.
- Endotoxin-induced ileal Vo2-Do2 alterations do not correlate with the severity of ileal injury.
- Metabolic consequences of sepsis. Correlation with altered intracellular calcium homeostasis.
- Acid aspiration results in ileal injury without altering ileal V(O2)-D(O2) relationships.
- Gastrointestinal tract dysfunction in critical illness: pathophysiology and interaction with acute Lung injury in adult respiratory distress syndrome/multiple organ dy...
- Digital imaging analysis for the study of endotoxin-induced mitochondrial ultrastructure injury.
Schools
Medical College Of Ohio
Oh State University Hospital
Procedures Preformed
- Bronchoscopy
- Thoracentesis
- Ventilation Management
Conditions Treated
- Acidosis
- Asthma
- Atelectasis
- Bronchiectasis
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Doctors Specialties
- Critical Care Medicine
- Pulmonology
- Internal Medicine
Accepted Insurances
Awards
- CMS Meaningful Use Stage 1 Certification
Education
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Ohio State University Hospital -
University of Toledo College of Medicine
Hospital
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Ohio State University Wexner Medical Center
Drug Facts
| NPI NUMBER | 1578576922 | |
| NPPES Provider LastName | CROUSER | |
| NPPES Provider FirstName | ELLIOTT | |
| NPPES Provider ZIPCode | 432213502 | |
| NPPES Provider State | OH | |
| Specialty Description | Pulmonary Disease | |
| Total Claim Count | 279.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1578576922 |
| Last Name Of The Provider | CROUSER |
| First Name Of The Provider | ELLIOTT |
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