Kampen-Norins Professor and Chair
Dermatology
Dr. Elliot J. Androphy, MD
Dr. Elliot J. Androphy practices Dermatology. He went to medical school at University of Rochester School of Medicine and Dentistry. He received 1 awards: "Fellow (FAAD)". Elliot Androphy is a published doctor as well. He has 101 publications published. The latest was: The bovine papillomavirus type 1 E6 oncoprotein sensitizes cells to tumor necrosis factor alpha-induced apoptosis. He accepts all Medicare patients.
Publications
- Levels of the E2 interacting protein TopBP1 modulate papillomavirus maintenance stage replication.
- Autophagy dysregulation in Cell culture and animals models of spinal muscular atrophy.
- Enhancement of SMN protein levels in a mouse model of spinal muscular atrophy using novel drug-like compounds.
- Acetylation of conserved lysines in bovine papillomavirus E2 by p300.
- Genomic instability: Ada3 and HPV E6-acetyltransferase connections?
- Chloroquine Promotes Apoptosis in Melanoma Cells by Inhibiting BH3 domain Mediated PUMA Degradation.
- Therapeutic strategies for the treatment of spinal muscular atrophy.
- PIASy-mediated Tip60 sumoylation regulates p53-induced autophagy.
- Identification of Novel Compounds That Increase SMN Protein Levels Using an Improved SMN2 Reporter Cell Assay.
- Differential regulation of the SMN2 gene by individual HDAC proteins.
- Discovery, Synthesis and Biological Evaluation of Novel SMN Protein Modulators.
- Valproate and bone loss: iTRAQ proteomics show that valproate reduces collagens and osteonectin in SMA Cells.
- Myogenic microRNA expression requires ATP-dependent chromatin remodeling enzyme function.
- Topography of bovine papillomavirus E2 protein on the viral genome during the Cell cycle.
- Tax1BP1 interacts with papillomavirus E2 and regulates E2-dependent transcription and stability.
- hAda3 degradation by papillomavirus type 16 E6 correlates with abrogation of the p14ARF-p53 pathway and efficient immortalization of human mammary epithelial Cells.
- Binding of human papillomavirus type 16 E6 to E6AP is not required for activation of hTERT.
- Interaction of papillomavirus E2 protein with the Brm chromatin remodeling complex leads to enhanced transcriptional activation.
- Mitotic kinesin-like protein 2 binds and colocalizes with papillomavirus E2 during mitosis.
- ChlR1 is required for loading papillomavirus E2 onto mitotic chromosomes and viral genome maintenance.
- Splicing of a critical exon of human Survival Motor Neuron is regulated by a unique silencer element located in the last intron.
- In vivo selection reveals combinatorial controls that define a critical exon in the spinal muscular atrophy genes.
- Human papillomavirus type 16 E6 amino acid 83 variants enhance E6-mediated MAPK signaling and differentially regulate tumorigenesis by notch signaling and oncogenic Ras.
- An extended inhibitory context causes skipping of exon 7 of SMN2 in spinal muscular atrophy.
- Immortalization of human mammary epithelial Cells is associated with inactivation of the p14ARF-p53 pathway.
- The regulation and regulatory activities of alternative splicing of the SMN gene.
- A transgene carrying an A2G missense mutation in the SMN gene modulates phenotypic severity in mice with severe (type I) spinal muscular atrophy.
- Genetic analysis of high-risk e6 in episomal maintenance of human papillomavirus genomes in primary human keratinocytes.
- Bovine papillomavirus type 1 E6-induced sensitization to apoptosis is distinct from its transforming activity.
- A direct interaction between the survival motor neuron protein and p53 and its relationship to spinal muscular atrophy.
- The BPV-1 E2 DNA-contact helix cysteine is required for transcriptional activation but not replication in mammalian Cells.
- Genetic analysis of the bovine papillomavirus E2 transcriptional activation domain.
- Transcriptional activation function is not required for stimulation of DNA replication by bovine papillomavirus type 1 E2.
- Sequences flanking the core DNA-binding domain of bovine papillomavirus type 1 E2 contribute to DNA-binding function.
- The survival motor neuron protein in spinal muscular atrophy.
- Mutational analysis of transcriptional activation by the bovine papillomavirus type 1 E6.
- Functional interaction of a novel Cellular protein with the papillomavirus E2 transactivation domain.
- Functional interaction of the bovine papillomavirus E2 transactivation domain with TFIIB.
- Polyomavirus large T can support DNA replication in human Cells.
- Assignment of the gene for ERC-55 (RCN2) to human chromosome band 15q22.33-->q24.1 by in situ hybridization.
- Transactivation-competent bovine papillomavirus E2 protein is specifically required for efficient repression of human papillomavirus oncogene expression and for acute ...
- SMN oligomerization defect correlates with spinal muscular atrophy severity.
- Identification of an alpha helical motif sufficient for association with papillomavirus E6.
- The domain encoded by exon 2 of the survival motor neuron protein mediates nucleic acid binding.
- 1H, 15N, and 13C NMR resonance assignments for the DNA-binding domain of the BPV-1 E2 protein.
- Crystal structure of the E2 DNA-binding domain from human papillomavirus type 16: implications for its DNA binding-site selection mechanism.
- The bovine papillomavirus type 1 E6 oncoprotein sensitizes Cells to tumor necrosis factor alpha-induced apoptosis.
- A single nucleotide in the SMN gene regulates splicing and is responsible for spinal muscular atrophy.
- A single nucleotide difference that alters splicing patterns distinguishes the SMA gene SMN1 from the copy gene SMN2.
- Identification of survival motor neuron as a transcriptional activator-binding protein.
- Multiple functions of human papillomavirus type 16 E6 contribute to the immortalization of mammary epithelial Cells.
- Human papillomavirus type 16 E6-enhanced susceptibility of L929 Cells to tumor necrosis factor alpha correlates with increased accumulation of reactive oxygen species.
- Two distinct regions of the BPV1 E1 replication protein interact with the activation domain of E2.
- Structural correlates for enhanced stability in the E2 DNA-binding domain from bovine papillomavirus.
- An exonic enhancer is required for inclusion of an essential exon in the SMA-determining gene SMN.
- AMF-1/Gps2 binds p300 and enhances its interaction with papillomavirus E2 proteins.
- Rb-independent induction of apoptosis by bovine papillomavirus type 1 E7 in response to tumor necrosis factor alpha.
- Htra2-beta 1 stimulates an exonic splicing enhancer and can restore full-length SMN expression to survival motor neuron 2 (SMN2).
- Targeting the E1 replication protein to the papillomavirus origin of replication by complex formation with the E2 transactivator.
- Papillomaviruses and interferon.
- Identification of bovine papillomavirus E1 mutants with increased transforming and transcriptional activity.
- Mutational analysis of bovine papillomavirus E6 gene.
- Human papillomavirus. Current concepts.
- Papillomavirus-associated inductions of Cellular proteins in mouse C127 Cells: correlation with the presence of open reading frame E2.
- Trans activation by the bovine papillomavirus E2 protein in Saccharomyces cerevisiae.
- The specific DNA recognition sequence of the bovine papillomavirus E2 protein is an E2-dependent enhancer.
- Bovine papilloma virus-transformed Cells contain multiple E2 proteins.
- X-linked inheritance of epidermodysplasia verruciformis. Genetic and virologic studies of a kindred.
- Identification of the protein encoded by the E6 transforming gene of bovine papillomavirus.
- Bovine papillomavirus E2 trans-activating gene product binds to specific sites in papillomavirus DNA.
- Increased ouabain-sensitive glycolysis of lymphocytes treated with phytohemagglutinin: relationship to potassium transport.
- Amino acids necessary for DNA contact and dimerization imply novel motifs in the papillomavirus E2 trans-activator.
- Mechanism of action of the papillomavirus E2 repressor: repression in the absence of DNA binding.
- Cooperative binding of the E2 protein of bovine papillomavirus to adjacent E2-responsive sequences.
- Human papillomavirus DNA sequences in Cell lines derived from head and neck squamous Cell carcinomas.
- Transcriptional activation by the papillomavirus E6 zinc finger oncoprotein.
- Antigen presentation and T-Cell activation in epidermodysplasia verruciformis.
- Proteins encoded by the bovine papillomavirus E1 open reading frame: expression in heterologous systems and in virally transformed Cells.
- The exon 2b region of the spinal muscular atrophy protein, SMN, is involved in self-association and SIP1 binding.
- Solution structure determination and mutational analysis of the papillomavirus E6 interacting peptide of E6AP.
- AMF1 (GPS2) modulates p53 transactivation.
- An in vivo reporter system for measuring increased inclusion of exon 7 in SMN2 mRNA: potential therapy of SMA.
- Regulation of murine survival motor neuron (Smn) protein levels by modifying Smn exon 7 splicing.
- The SWI/SNF chromatin remodeling subunit BRG1 is a critical regulator of p53 necessary for proliferation of malignant Cells.
- hAda3 regulates p14ARF-induced p53 acetylation and senescence.
- Modulating role of RNA structure in alternative splicing of a critical exon in the spinal muscular atrophy genes.
- Determinants of stability for the E6 protein of papillomavirus type 16.
- Identification of inhibitors to papillomavirus type 16 E6 protein based on three-dimensional structures of interacting proteins.
- The DNA helicase ChlR1 is required for sister chromatid cohesion in mammalian Cells.
- Opposing effects of bovine papillomavirus type 1 E6 and E7 genes on Fas-mediated apoptosis.
- Indoprofen upregulates the survival motor neuron protein through a cyclooxygenase-independent mechanism.
- Human papillomavirus type 16 E6 promotes retinoblastoma protein phosphorylation and Cell cycle progression.
- The Ewing''s sarcoma protein interacts with the Tudor domain of the survival motor neuron protein.
- Signals that dictate nuclear localization of human papillomavirus type 16 oncoprotein E6 in living Cells.
- The COPI vesicle complex binds and moves with survival motor neuron within axons.
- SRp30c-dependent stimulation of survival motor neuron (SMN) exon 7 inclusion is facilitated by a direct interaction with hTra2 beta 1.
- Design and characterization of helical peptides that inhibit the E6 protein of papillomavirus.
- A mutant of human papillomavirus type 16 E6 deficient in binding alpha-helix partners displays reduced oncogenic potential in vivo.
- Cellular steady-state levels of "high risk" but not "low risk" human papillomavirus (HPV) E6 proteins are increased by inhibition of proteasome-dependent degradation i...
- Structure based identification and characterization of flavonoids that disrupt human papillomavirus-16 e6 function.
Schools
University Of Rochester
NIH
University Pittsburgh Falk Clin
Procedures Preformed
- Destruction of Benign Skin Lesion
- Destruction of Malignant Skin Lesion
- Excision of Benign Skin Lesion
- Skin Tag Removal
Conditions Treated
- Actinic Keratosis
- Atopic Dermatitis (Eczema)
- Basal Cell Carcinoma
- Benign Skin Lesion
- View All
Doctors Specialties
- Dermatology
Accepted Insurances
Awards
- Fellow (FAAD)
Education
-
National Institutes of Health Clinical Center -
University of Pittsburgh Medical Center -
University of Rochester School of Medicine and Dentistry
Hospital
-
IU Health Methodist Hospital
Drug Facts
| NPI NUMBER | 1740261478 | |
| NPPES Provider LastName | ANDROPHY | |
| NPPES Provider FirstName | ELLIOT | |
| NPPES Provider ZIPCode | 462025149 | |
| NPPES Provider State | IN | |
| Specialty Description | Dermatology | |
| Total Claim Count | 23.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1740261478 |
| Last Name Of The Provider | ANDROPHY |
| First Name Of The Provider | ELLIOT |
| View All |
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