Professor of Medicine, University Iowa College of Medicine

Rheumatology

Dr. Elizabeth H. Field works in the field of Rheumatology. She studied medicine at Penn State College of Medicine. She received 1 awards: "Elected Member". Elizabeth Field is a published doctor as well. She has 39 publications published. The latest was: CD4 regulatory cells in immune tolerance. Elizabeth Field accepts Medicare payments and is listed with Medicare.gov.

Publications

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• Dendritic Cells Control CD4(+)CD25(+) Treg Cell Suppressor Function In Vitro through Juxtacrine Delivery of IL-2.
• Natural Tregs, CD4+CD25+ inhibitory hybridomas, and their cell contact dependent suppression.
• Downstream of tyrosine kinases-1 and Src homology 2-containing inositol 5'-phosphatase are required for regulation of CD4+CD25+ T cell development.
• TLR-9 activation of marginal zone B cells in lupus mice regulates immunity through increased IL-10 production.
• Total lymphoid irradiation nonmyeloablative preconditioning enriches for IL-4-producing CD4+-TNK cells and skews differentiation of immunocompetent donor CD4+ cells.
• Requirements for developing mixed-chimerism in nonmyeloablative total-lymphoid irradiated mice: role of IL-4 and immunoredirection.
• Isolation and sequence of L3T4 complementary DNA clones: expression in T cells and brain.
• Long-term followup of rheumatoid arthritis patients treated with total lymphoid irradiation.
• Sustained improvement of intractable rheumatoid arthritis after total lymphoid irradiation.
• Treatment of lupus nephritis with total lymphoid irradiation. Observations during a 12-79-month followup.
• The gene encoding the mouse T cell differentiation antigen L3T4 is located on chromosome 6.
• Lupus nephritis after total lymphoid irradiation: persistent improvement and reduction of steroid therapy.
• Transient rheumatoid factor positivity in acute human parvovirus B19 infection.
• Reduced in vitro immune responses of purified human Leu-3 (helper/induced phenotype) cells after total lymphoid irradiation.
• CD4 regulatory cells in immune tolerance.
• Early-life co-administration of cockroach allergen and endotoxin augments pulmonary and systemic responses.
• Age influences recovery of systemic and mucosal immune responses following acute depletion of CD4 T cells.
• Effect of anti-CD4 on CD4 subsets. I. Anti-CD4 preferentially deletes resting, naive CD4 cells and spares activated CD4 cells.
• Prevention of Th1 response is critical for tolerance.
• Tolerance, mixed chimerism and protection against graft-versus-host disease after total lymphoid irradiation.
• Enhanced type 2 and diminished type 1 cytokines in neonatal tolerance.
• CD4 Th2 cells do not functionally suppress CTL generation in neonatal tolerant mice.
• Expansion of memory Th2 cells over Th1 cells in neonatal primed mice.
• Nondeletional mechanisms of tolerance in total-lymphoid irradiation-induced bone marrow chimeras.
• Mechanisms of total lymphoid irradiation-induced immunosuppression. II. Failure of con A-stimulated splenocytes from TLI-treated mice to express IL-2 and IL-2 receptor...
• Transferrin receptor synthesis is an early event in B cell activation.
• The immunosuppressive mechanism of total lymphoid irradiation. I. The effect on IL-2 production and IL-2 receptor expression.
• Evidence for excessive Th2 CD4+ subset activity in vivo.
• Blocking of mixed lymphocyte reaction by spleen cells from total lymphoid-irradiated mice involves interruption of the IL-2 pathway.
• Kinetics of anti-CD4-induced T helper cell depletion and inhibition of function. Activation of T cells by the CD3 pathway inhibits anti-CD4-mediated T cell elimination...
• Balancing the immune system for tolerance: a case for regulatory CD4 cells.
• CD4+CD25+ regulatory cells in acquired MHC tolerance.
• Association between enhanced Th2/Th1 cytokine profile and donor T-cell chimerism following total lymphoid irradiation.
• The role of interleukin-4 in the induction phase of allogeneic neonatal tolerance.
• Alloantigen priming after total lymphoid irradiation alters alloimmune cytokine responses.
• Altered IFN-gamma and IL-4 pattern lymphokine secretion in mice partially depleted of CD4 T cells by anti-CD4 monoclonal antibody.
• Mechanisms of tolerance induction: splenocytes from total lymphoid irradiated mice inhibit the IL-2 pathway in TCR-activated CD4 cells.
• CD4+CD25+ cells regulate CD8 cell anergy in neonatal tolerant mice.

Schools

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• Pennsylvania State University College of Medicine

• Hershey McPenn St University

• Stanford University

Doctors Specialties

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• Internal Medicine
• Rheumatology

Accepted Insurances

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Awards

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• Elected Member

Education

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• Stanford University
• Penn State University
• Penn State College of Medicine

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