Associate Professor, Internal Medicine, Yale University School of Medicine
Neurology
Dr. Elizabeth A. Jonas, MD
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Elizabeth Jonas works in the field of Neurology. She went to medical school at NYU School of Medicine. Elizabeth Jonas is a published MD. She published 45 publications, including: 'Acute myocardial infarction with normal coronary artery: a case report and review of literature.' She accepts all Medicare patients.
Publications
- Physiological roles of the mitochondrial permeability transition pore.
- Decreased SGK1 Expression and Function Contributes to Behavioral Deficits Induced by Traumatic Stress.
- Mitochondrial membrane protein Bcl-xL, a regulator of adult neuronal growth and synaptic plasticity: multiple functions beyond apoptosis.
- The mitochondrial complex V-associated large-conductance inner membrane current is regulated by cyclosporine and dexpramipexole.
- An uncoupling channel within the c-subunit ring of the F1FO ATP synthase is the mitochondrial permeability transition pore.
- Impaired import: how huntingtin harms.
- Bcl-xL is necessary for neurite outgrowth in hippocampal neurons.
- Contributions of Bcl-xL to acute and long term changes in bioenergetics during neuronal plasticity.
- A Bcl-xL-Drp1 complex regulates synaptic vesicle membrane dynamics during endocytosis.
- Bcl-xL regulates mitochondrial energetics by stabilizing the inner membrane potential.
- Molecular participants in mitochondrial cell death channel formation during neuronal ischemia.
- Bcl-x L increases mitochondrial fission, fusion, and biomass in neurons.
- Ischemic preconditioning blocks BAD translocation, Bcl-xL cleavage, and large channel activity in mitochondria of postischemic hippocampal neurons.
- PKC-induced intracellular trafficking of Ca(V)2 precedes its rapid recruitment to the plasma membrane.
- Bcl-xL inhibitor ABT-737 reveals a dual role for Bcl-xL in synaptic transmission.
- Bcl-xL induces Drp1-dependent synapse formation in cultured hippocampal neurons.
- Hypoxia increases BK channel activity in the inner mitochondrial membrane.
- Expression of the voltage-gated sodium channel NaV1.5 in the macrophage late endosome regulates endosomal acidification.
- Zinc-dependent multi-conductance channel activity in mitochondria isolated from ischemic Brain.
- Actions of BAX on mitochondrial channel activity and on synaptic transmission.
- The role of the mitochondrial apoptosis induced channel MAC in cytochrome c release.
- Oligomeric Bax is a component of the putative cytochrome c release channel MAC, mitochondrial apoptosis-induced channel.
- Exposure to hypoxia rapidly induces mitochondrial channel activity within a living synapse.
- Proapoptotic N-truncated BCL-xL protein activates endogenous mitochondrial channels in living synaptic terminals.
- Modulation of synaptic transmission by the BCL-2 family protein BCL-xL.
- Modulation of mitochondrial function by endogenous Zn2+ pools.
- BAK alters neuronal excitability and can switch from anti- to pro-death function during postnatal development.
- Detection of cardiac events by continuous electrocardiogram monitoring during ritodrine infusion.
- The effect of atherogenic infusions of the triglyceride-rich, lipid emulsion, Lipofundin-S, on the in vitro growth characteristics of rat aortic smooth muscle cells.
- Induction of early atherosclerosis in rats using parenterally-administered lipid emulsions.
- Scombroid poisoning.
- Acute myocardial infarction with normal coronary Artery: a case report and review of literature.
- Intracellular ionized calcium ([Ca++]i) mobilization in platelets from rats receiving atherogenic lipids. Modulation by nifedipine.
- Hoofbeats and zebras: neurodegenerative disorder presenting as a "first episode" of psychosis.
- Mitochondrial factors with dual roles in death and survival.
- Regulation of potassium channels by protein kinases.
- Ca2+ influx and activation of a cation current are coupled to intracellular Ca2+ release in peptidergic neurons of Aplysia californica.
- Regulation by insulin of a unique neuronal Ca2+ pool and of neuropeptide secretion.
- Giga-ohm seals on intracellular membranes: a technique for studying intracellular ion channels in intact cells.
- BCL-xL regulates synaptic plasticity.
- Insulin receptor in Aplysia neurons: characterization, molecular cloning, and modulation of ion currents.
- Regulation of synaptic transmission by mitochondrial ion channels.
- Prolonged activation of mitochondrial conductances during synaptic transmission.
- Bcl-xL regulates metabolic efficiency of neurons through interaction with the mitochondrial F1FO ATP synthase.
Schools
New York University
Conditions Treated
- Alzheimer's Disease
- Autonomic Disorders
- Brain Aneurysm
- Carpal Tunnel Syndrome
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Doctors Specialties
- Internal Medicine
- Neurology
Accepted Insurances
Education
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Yale-New Haven Hospital -
Yale New Haven Children's Center -
NYU School of Medicine
Drug Facts
| NPI NUMBER | 1598923377 | |
| NPPES Provider LastName | JONAS | |
| NPPES Provider FirstName | ELIZABETH | |
| NPPES Provider ZIPCode | 065191304 | |
| NPPES Provider State | CT | |
| Specialty Description | Internal Medicine | |
| Total Claim Count | 376.0 | |
| Distinct Opioid Count | 3.0 | |
| Opioid Claim Count | 48.0 | |
| Percent Opioid Claims | 12.77 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1598923377 |
| Last Name Of The Provider | JONAS |
| First Name Of The Provider | ELIZABETH |
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