Professor of Medicine, Northwestern University Heme-Onc Section Chief, Jesse Brown VA
Hematology
Dr. Elizabeth A. Eklund, MD
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Elizabeth A. Eklund works in the field of Hematology. She went to Indiana University School of Medicine. Indiana University School of Medicine is ranked 45/47 in Research/PrimaryCare. She has 1 awards "Super Doctor". Elizabeth Eklund is also published. She has 67 publications published. The lastest publication: "PU.1, interferon regulatory factor 1, and interferon consensus sequence-binding protein cooperate to increase gp91(phox) expression.' She accepts Medicare payments and is registered with Medicare.gov.
Publications
- The Interferon Consensus Sequence Binding Protein (Icsbp/Irf8) Is Required for Termination of Emergency Granulopoiesis.
- Small molecule screen for inhibitors of expression from canonical CREB response element-containing promoters.
- Targeting novel signaling pathways for resistant Acute myeloid leukemia.
- ResVeratrol enhances the suppressive effects of arsenic trioxide on primitive leukemic progenitors.
- The Leukemia-associated Mll-Ell Oncoprotein Induces Fibroblast Growth Factor 2 (Fgf2)-dependent Cytokine Hypersensitivity in Myeloid Progenitor Cells.
- Essential Role for the Mnk Pathway in the Inhibitory Effects of Type I Interferons on Myeloproliferative Neoplasm (MPN) Precursors.
- Fas associated phosphatase 1 (Fap1) influences ?catenin activity in Myeloid progenitor cells expressing the Bcr-abl oncogene.
- Administration of nicotinamide does not increase platelet levels in mice.
- β-Catenin Activates the HOXA10 and CDX4 Genes in Myeloid Progenitor Cells.
- Fas-associated phosphatase 1 mediates Fas resistance in Myeloid progenitor cells expressing the Bcr-abl oncogene.
- Regulatory Effects of Programmed Cell Death 4 (PDCD4) Protein in Interferon (IFN)-Stimulated Gene Expression and Generation of Type I IFN Responses.
- The Leukemia-associated fusion-protein Tel-platelet derived growth factor receptor B (Tel-PdgfRB) inhibits transcriptional repression of the PTPN13 gene by the interfe...
- Inhibition of histone deacetylase 6 as a therapeutic strategy for Acute lymphocytic leukemia.
- HoxA10 Influences Protein Ubiquitination by Activating Transcription of ARIH2, the Gene Encoding Triad1.
- HoxA10 regulates transcription of the gene encoding transforming growth factor beta2 (TGFbeta2) in Myeloid cells.
- Regulatory effects of ribosomal S6 kinase 1 (RSK1) in IFNλ signaling.
- Geminin deletion from hematopoietic cells causes Anemia and thrombocytosis in mice.
- Interferon consensus sequence binding protein (ICSBP) decreases beta-catenin activity in Myeloid cells by repressing GAS2 transcription.
- Genomic analysis of Acute myeloid leukemia: potential for new prognostic indicators.
- The interferon consensus sequence binding protein (ICSBP/IRF8) activates transcription of the FANCF gene during Myeloid differentiation.
- Role of Schlafen 2 (SLFN2) in the generation of interferon alpha-induced growth inhibitory responses.
- Constitutively active SHP2 cooperates with HoxA10 overexpression to induce Acute myeloid leukemia.
- Thrombocytopenia and cancer.
- Dual regulatory roles of phosphatidylinositol 3-kinase in IFN signaling.
- The interferon consensus sequence-binding protein (ICSBP/IRF8) represses PTPN13 gene transcription in differentiating Myeloid cells.
- Constitutive activation of SHP2 protein tyrosine phosphatase inhibits ICSBP-induced transcription of the gene encoding gp91PHOX during Myeloid differentiation.
- Constitutive activation of SHP2 in mice cooperates with ICSBP deficiency to accelerate progression to Acute myeloid leukemia.
- Identification of a HoxA10 activation domain necessary for transcription of the gene encoding beta3 integrin during Myeloid differentiation.
- HoxA10 activates transcription of the gene encoding mitogen-activated protein kinase phosphatase 2 (Mkp2) in Myeloid cells.
- PU.1, interferon regulatory factor (IRF) 2, and the interferon consensus sequence-binding protein (ICSBP/IRF8) cooperate to activate NF1 transcription in differentiati...
- The role of HOX genes in malignant Myeloid disease.
- Activation of SHP2 protein-tyrosine phosphatase increases HoxA10-induced repression of the genes encoding gp91(PHOX) and p67(PHOX).
- Leukemia-associated, constitutively active mutants of SHP2 protein tyrosine phosphatase inhibit NF1 transcriptional activation by the interferon consensus sequence bin...
- The role of HOX genes in Myeloid leukemogenesis.
- HoxA10 represses transcription of the gene encoding p67phox in phagocytic cells.
- Differential regulation of the p70 S6 kinase pathway by interferon alpha (IFNalpha) and imatinib mesylate (STI571) in chronic myelogenous Leukemia cells.
- HOXA9 activates transcription of the gene encoding gp91Phox during Myeloid differentiation.
- JAK2 is necessary and sufficient for interferon-gamma-induced transcription of the gene encoding gp91PHOX.
- The interferon consensus sequence-binding protein activates transcription of the gene encoding neurofibromin 1.
- HoxA10 represses gene transcription in undifferentiated Myeloid cells by interaction with histone deacetylase 2.
- SHP1 protein-tyrosine phosphatase regulates HoxA10 DNA binding and transcriptional repression activity in undifferentiated Myeloid cells.
- Hyposialylation of integrins stimulates the activity of Myeloid fibronectin receptors.
- Regulation of CDX4 gene transcription by HoxA9, HoxA10, the Mll-Ell oncogene and Shp2 during leukemogenesis.
- IUPAC collaborative trial study of a method to detect genetically modified soy beans and maize in dried powder.
- Purification and characterization of a lipid thiobis ester from human neutrophil cytosol that reversibly deactivates the O2- -generating NADPH oxidase.
- Biosynthesis of the proteoglycan decorin -- identification of intermediates in galactosaminoglycan assembly.
- Dermatan is a better substrate for 4-O-sulfation than chondroitin: implications in the generation of 4-O-sulfated, L-iduronate-rich galactosaminoglycans.
- Leadership training in UAFs supported by the Division of Maternal and Child Health.
- Cloning of a cDNA encoding a human DNA-binding protein similar to ribosomal protein S1.
- Resolution of a low molecular weight G protein in neutrophil cytosol required for NADPH oxidase activation and reconstitution by recombinant Krev-1 protein.
- A neutrophil GTP-binding protein that regulates cell free NADPH oxidase activation is located in the cytosolic fraction.
- Mutations in the promoter region of the gene for gp91-phox in X-linked Chronic granulomatous disease with decreased expression of cytochrome b558.
- Characterization of a gp91-phox promoter element that is required for interferon gamma-induced transcription.
- Characterization of three promoter elements and cognate DNA binding protein(s) necessary for IFN-gamma induction of gp91-phox transcription.
- Kruppel-like factor 4 regulates laminin alpha 3A expression in mammary epithelial cells.
- Tyrosine phosphorylation of HoxA10 decreases DNA binding and transcriptional repression during interferon gamma -induced differentiation of myeloid Leukemia cell lines.
- SHP1 protein-tyrosine phosphatase inhibits gp91PHOX and p67PHOX expression by inhibiting interaction of PU.1, IRF1, interferon consensus sequence-binding protein, and ...
- Identification and characterization of TF1(phox), a DNA-binding protein that increases expression of gp91(phox) in PLB985 myeloid Leukemia cells.
- Recruitment of CREB-binding protein by PU.1, IFN-regulatory factor-1, and the IFN consensus sequence-binding protein is necessary for IFN-gamma-induced p67phox and gp9...
- PU.1, interferon regulatory factor 1, and interferon consensus sequence-binding protein cooperate to increase gp91(phox) expression.
- The role of Hox proteins in leukemogenesis: insights into key regulatory events in hematopoiesis.
- HoxA10 activates CDX4 transcription and Cdx4 activates HOXA10 transcription in Myeloid cells.
- Activin subunit and receptor expression in normal and cleft human fetal palate tissues.
- Very high inhibin A concentration attributed to heterophilic antibody interference.
- Screening for microRNAs in Myelodysplastic syndromes.
- Correlation between IDH2 mutations and disease status in Acute myeloid leukemia.
Schools
Rush University
Clarian Indiana University Hospital
Mayo Grad School MedMayo Fndn
Rush Presby St Luke39s M C
Conditions Treated
- Acute Leukemia
- Anemia
- Chronic Myeloid Leukemia (CML)
- Cryoglobulinemia
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Doctors Specialties
- Hematology
Accepted Insurances
Awards
- Super Doctor
Education
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Indiana University School of Medicine -
Mayo School of Graduate Medical Education -
Rush University Medical Center -
Rush Medical College
Hospital
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Northwestern Memorial Hospital -
Jesse Brown Veterans Affairs Medical Center
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1598861205 |
| Last Name Of The Provider | EKLUND |
| First Name Of The Provider | ELIZABETH |
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