Professor, Medicine, Brown University Program in Medicine
Infectious Disease
Dr. Edward J. Wing, MD
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Edward Wing is an expert in the field of Infectious Disease. He graduated from Harvard Medical School. Harvard Medical School is one of the top medical schools in the US. Harvard Medical School is ranked 1/12 in Research/PrimaryCare. Edward Wing is also published. He has 100 publications published. The lastest publication: "Role for activated macrophages in resistance against Trichinella spiralis.' Doctor Edward J. Wing, MD is registered with Medicare and accepts Medicare payments.
Publications
- Multicentric Castleman disease, Kaposi sarcoma, hemophagocytic syndrome, and a novel HHV8-lymphoproliferative disorder.
- It is time to implement routine, not risk-based, HIV testing.
- A diagnostic dilemma in a patient receiving antiretroviral and antituberculosis therapy.
- Early scurvy complicating anorexia nervosa.
- Neutrophil-Kupffer cell interaction: a critical component of host defenses to systemic bacterial infections.
- Listeriosis during pregnancy: a case series and review of 222 cases.
- Listeria monocytogenes: clinical and experimental update.
- Complementary adhesion molecules promote neutrophil-Kupffer cell interaction and the elimination of bacteria taken up by the liver.
- Globalization: a new dimension for academic internal medicine.
- Quality control of home monitoring of blood glucose concentrations.
- Epidemiology of legionella pneumonia and factors associated with legionella-related mortality at a tertiary care center.
- A clinic based educational programme for children with diabetes.
- A changing pattern of cerebral palsy and its implications for the early detection of motor disorders in children.
- Loss of resistance to listeria infection in autoimmune MRL/lpr mice: protection by prostaglandin E1.
- An updated model of cell-mediated immunity--listeriosis: clinical and research aspects.
- From hot dogs to CD8+ T cells: Listeria monocytogenes.
- Innate defenses in the liver during Listeria infection.
- Development of listerial meningitis during ciprofloxacin treatment.
- Effect of colony stimulating factor on murine macrophages. Induction of antitumor activity.
- Acute starvation protects mice against Listeria monocytogenes.
- Decreased resistance to Listeria monocytogenes in mice injected with killed corynebacterium parvum: association with suppression of cell-mediated immunity.
- The use of tracheal and pulmonary aspiration to diagnose Legionella micdadei pneumonia.
- Primary meningococcal arthritis and pseudogout in an elderly woman.
- Failure of erythromycin in treatment of Legionella micdadei pneumonia.
- Dissociative effects of a novel immunomodulating agent (CP-20, 961) on host defenses of mice.
- Changes in serum colony-stimulating factor and monocytic progenitor cells during Listeria monocytogenes infection in mice.
- Analysis of colony-stimulating factors and macrophage progenitor cells in mice immunized against Listeria monocytogenes by adoptive transfer.
- Characterization of wound cytokines in the sponge matrix model.
- Secretion of colony-stimulating factors by T cell clones. Role in adoptive protection against Listeria monocytogenes.
- Neutrophil-Kupffer-cell interaction in host defenses to systemic infections.
- Fas (CD95)-dependent cell-mediated immunity to Listeria monocytogenes.
- IL-6 produced by Kupffer cells induces STAT protein activation in hepatocytes early during the course of systemic listerial infections.
- Listeria monocytogenes-infected hepatocytes are targets of major histocompatibility complex class Ib-restricted antilisterial cytotoxic T lymphocytes.
- Internalin B promotes the replication of Listeria monocytogenes in mouse hepatocytes.
- Immune CD8+ T lymphocytes lyse Listeria monocytogenes-infected hepatocytes by a classical MHC class I-restricted mechanism.
- Expression of the inlAB operon by Listeria monocytogenes is not required for entry into hepatic cells in vivo.
- Lymphokine-activated killer cells lyse Listeria-infected hepatocytes and produce elevated quantities of interferon-gamma.
- Bacteria in the bloodstream are trapped in the liver and killed by immigrating neutrophils.
- Life-threatening neutropenia secondary to piperacillin/tazobactam therapy.
- Factors associated with the development of candidemia and candidemia-related death among liver transplant recipients.
- Macrophage colony-stimulating factor and the enhanced migration of monocytes are essential in primary but not secondary host defenses to Listeria organisms.
- IFN-gamma inhibits the replication of Listeria monocytogenes in hepatocytes.
- Arginine analogues suppress antigen-specific and -nonspecific T lymphocyte proliferation.
- Hepatocytes can serve as accessory cells in the response of immune T lymphocytes to heat-killed Listeria monocytogenes.
- Reactive nitrogen intermediates suppress the primary immunologic response to Listeria.
- Rapamycin but not FK506 inhibits the proliferation of mononuclear phagocytes induced by colony-stimulating factors.
- Infectious complications with plasmapheresis in rapidly progressive glomerulonephritis.
- Activated macrophages pre-incubated in vitro enhance rather than suppress mitogen-stimulated lymphocyte transformation.
- Bacillus Calmette-Guérin (BCG) decreases resistance to Listeria monocytogenes infection in mice.
- Successful treatment of Legionella micdadei (Pittsburgh pneumonia agent) pneumonia with erythromycin.
- Yersinia enterocolitica lung infection.
- Stimulation of a human macrophage-like cell line (U-937) to inhibit multiplication of an intracellular pathogen.
- Hot water systems as sources of Legionella pneumophila in hospital and nonhospital plumbing fixtures.
- Fasting-enhanced immune effector mechanisms in obese subjects.
- Inappropriate use of cimetidine in hospitalized patients.
- Disseminated listeriosis presenting as acute hepatitis. Case reports and review of hepatic involvement in listeriosis.
- Effect of acute nutritional deprivation on immune function in mice. II. Response to sublethal radiation.
- Toxoplasma gondii: decreased resistance to intracellular bacteria in mice.
- Effect of acute nutritional deprivation on host defenses against Listeria monocytogenes--macrophage function.
- Effect of acute nutritional deprivation on immune function in mice. I. Macrophages.
- A comparative study of Legionella micdadei and other nosocomial acquired pneumonia.
- Second serogroup of Legionella hackeliae isolated from a patient with pneumonia.
- Cost savings associated with use of gentamicin versus tobramycin.
- Effect of Listeria monocytogenes infection on serum levels of colony-stimulating factor and number of progenitor cells in immune and nonimmune mice.
- 11th serogroup of Legionella pneumophila isolated from a patient with fatal pneumonia.
- Tobramycin and ticarcillin as empiric therapy for febrile patients with granulocytopenia: a reevaluation.
- Macrophage colony-stimulating factor enhances the expression of Fc receptors on murine peritoneal macrophages.
- Peritoneal macrophages exposed to purified macrophage colony-stimulating factor (M-CSF) suppress mitogen- and antigen-stimulated lymphocyte proliferation.
- Effect of acute nutritional deprivation on macrophage colony-stimulating factor and macrophage progenitor cells in mice.
- Prospective study of pneumonia: unexpected incidence of legionellosis.
- Isolated cardiac aspergillosis after bone marrow transplantation.
- Acute starvation in mice reduces the number of T cells and suppresses the development of T-cell-mediated immunity.
- Antigen-specific production of colony-stimulating factors by Listeria monocytogenes-immune, L3T4-positive cells.
- Fatal bacterial endocarditis as a complication of permanent indwelling catheters. Report of two cases.
- In vitro production of colony-stimulating factors by Listeria-immune spleen cells.
- Stimulatory effects of purified macrophage colony-stimulating factor on murine resident peritoneal macrophages.
- Macrophage colony-stimulating factor (M-CSF) enhances the capacity of murine macrophages to secrete oxygen reduction products.
- The effect of recombinant human granulocyte macrophage colony-stimulating factor on neutrophil activation in patients with refractory carcinoma.
- Demonstration of a blood-bone marrow barrier to macrophage colony-stimulating factor.
- Recombinant human granulocyte/macrophage colony-stimulating factor enhances monocyte cytotoxicity and secretion of tumor necrosis factor alpha and interferon in cancer...
- Cloned L3T4+ T lymphocytes protect mice against Listeria monocytogenes by secreting IFN-gamma.
- Legionella infection in transplant patients.
- Biological effects of recombinant human GM-CSF.
- Accessory function of Kupffer cells in the antigen-specific blastogenic response of an L3T4+ T-lymphocyte clone to Listeria monocytogenes.
- The role of colony-stimulating factors in host defenses.
- Production of colony-stimulating factors during pneumonia caused by Chlamydia trachomatis.
- Evaluation of the influence of FK 506, rapamycin, and cyclosporine on processing and presentation of particulate antigen by macrophages: assessment of a drug "carry-ov...
- Tumor necrosis factor, macrophage colony-stimulating factor, and interleukin 1 production within sponge matrix allografts.
- Effector function of hepatocytes and Kupffer cells in the resolution of systemic bacterial infections.
- Primary listerial infections are exacerbated in mice administered neutralizing antibody to macrophage colony-stimulating factor.
- Solitary obstructing plasmacytoma of the colon.
- Dissociation of effector functions in populations of activated macrophages.
- Pseudomonas maltophilia causing heroin-associated infective endocarditis.
- Infectious endocarditis caused by Rothia dentocariosa.
- Studies of macrophage function during Trichinella spiralis infection in mice.
- Cell-mediated immunity and its role in resistance to infection.
- Use of quantitative bacteriologic techniques to diagnose catheter-related sepsis.
- Role for activated macrophages in resistance against Trichinella spiralis.
- Studies on the regulation of lymphocyte reactivity by normal and activated macrophages.
Schools
Harvard Medical School
Peter Bent Brigham Hospital
Stanford
Doctors Specialties
- Infectious Disease Medicine
- Infectious Disease
Accepted Insurances
Education
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Brigham and Women's Hospital -
Harvard Medical School
Hospital
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Rhode Island Hospital
Drug Facts
| NPI NUMBER | 1578503694 | |
| NPPES Provider LastName | WING | |
| NPPES Provider FirstName | EDWARD | |
| NPPES Provider ZIPCode | 029062853 | |
| NPPES Provider State | RI | |
| Specialty Description | Internal Medicine | |
| Total Claim Count | 465.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1578503694 |
| Last Name Of The Provider | WING |
| First Name Of The Provider | EDWARD |
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