Professor, Medicine, Brown University Program in Medicine
Infectious Disease
Dr. Edward E. Wing, MD
|
|
Give Review
Doctor Edward E. Wing, MD works in the field of Infectious Disease. He graduated from Harvard Medical School. Harvard Medical School is a one of the best medicals schools. Harvard Medical School is ranked 1 in research and 12 in primary care medicine Dr. Edward E. Wing has written publications. He has 100 publications published with PubMed. The lastest paper is: 'Role for activated macrophages in resistance against Trichinella spiralis.'
Publications
- Multicentric Castleman disease, Kaposi sarcoma, hemophagocytic Syndrome, and a novel HHV8-lymphoproliferative disorder.
- It is time to implement routine, not risk-based, HIV testing.
- A diagnostic dilemma in a patient receiving antiretroviral and antituberculosis therapy.
- Early scurvy Complicating anorexia nervosa.
- Neutrophil-Kupffer Cell interaction: a critical component of host defenses to systemic bacterial infections.
- Listeriosis during Pregnancy: a case series and review of 222 cases.
- Listeria monocytogenes: clinical and experimental update.
- Complementary adhesion molecules promote neutrophil-Kupffer Cell interaction and the elimination of bacteria taken up by the liver.
- Globalization: a new dimension for academic internal medicine.
- Quality control of home monitoring of blood glucose concentrations.
- Epidemiology of legionella pneumonia and factors Associated with legionella-related mortality at a tertiary care center.
- A clinic based educational programme for children With diabetes.
- A changing pattern of cerebral palsy and its implications for the early detection of motor Disorders in children.
- Loss of resistance to listeria infection in autoimmune MRL/lpr mice: protection by prostaglandin E1.
- An updated model of Cell-mediated immunity--listeriosis: clinical and research aspects.
- From hot dogs to CD8+ T Cells: Listeria monocytogenes.
- Innate defenses in the liver during Listeria infection.
- Development of listerial meningitis during ciprofloxacin treatment.
- Effect of colony stimulating factor on murine macrophages. Induction of antitumor activity.
- Acute starvation protects mice against Listeria monocytogenes.
- Decreased resistance to Listeria monocytogenes in mice injected With killed corynebacterium parvum: association With suppression of cell-mediated immunity.
- The use of tracheal and pulmonary aspiration to diagnose Legionella micdadei pneumonia.
- Primary meningococcal arthritis and pseudogout in an elderly woman.
- Failure of erythromycin in treatment of Legionella micdadei pneumonia.
- Dissociative effects of a novel immunomodulating agent (CP-20, 961) on host defenses of mice.
- Changes in serum colony-stimulating factor and monocytic progenitor Cells during Listeria monocytogenes infection in mice.
- Analysis of colony-stimulating factors and macrophage progenitor Cells in mice immunized against Listeria monocytogenes by adoptive transfer.
- Characterization of wound cytokines in the sponge matrix model.
- Secretion of colony-stimulating factors by T Cell clones. Role in adoptive protection against Listeria monocytogenes.
- Neutrophil-Kupffer-Cell interaction in host defenses to systemic infections.
- Fas (CD95)-dependent Cell-mediated immunity to Listeria monocytogenes.
- IL-6 produced by Kupffer Cells induces STAT protein activation in hepatocytes early during the course of systemic listerial infections.
- Listeria monocytogenes-infected hepatocytes are targets of major histocompatibility complex class Ib-restricted antilisterial cytotoxic T lymphocytes.
- Internalin B promotes the replication of Listeria monocytogenes in mouse hepatocytes.
- Immune CD8+ T lymphocytes lyse Listeria monocytogenes-infected hepatocytes by a classical MHC class I-restricted mechanism.
- Expression of the inlAB operon by Listeria monocytogenes is not required for entry into hepatic Cells in vivo.
- Lymphokine-activated killer Cells lyse Listeria-infected hepatocytes and produce elevated quantities of interferon-gamma.
- Bacteria in the bloodstream are trapped in the liver and killed by immigrating neutrophils.
- Life-threatening neutropenia secondary to piperacillin/tazobactam therapy.
- Factors Associated with the development of candidemia and candidemia-related death among liver transplant recipients.
- Macrophage colony-stimulating factor and the enhanced migration of monocytes are essential in primary but not secondary host defenses to Listeria organisms.
- IFN-gamma inhibits the replication of Listeria monocytogenes in hepatocytes.
- Arginine analogues suppress antigen-specific and -nonspecific T lymphocyte proliferation.
- Hepatocytes can serve as accessory Cells in the response of immune T lymphocytes to heat-killed Listeria monocytogenes.
- Reactive nitrogen intermediates suppress the primary immunologic response to Listeria.
- Rapamycin but not FK506 inhibits the proliferation of mononuclear phagocytes induced by colony-stimulating factors.
- Infectious complications With plasmapheresis in rapidly progressive glomerulonephritis.
- Activated macrophages pre-incubated in vitro enhance rather than suppress mitogen-stimulated lymphocyte transformation.
- Bacillus Calmette-Guérin (BCG) decreases resistance to Listeria monocytogenes infection in mice.
- Successful treatment of Legionella micdadei (Pittsburgh pneumonia agent) pneumonia With erythromycin.
- Yersinia enterocolitica lung infection.
- Stimulation of a Human macrophage-like cell line (U-937) to inhibit multiplication of an intracellular pathogen.
- Hot water systems as sources of Legionella pneumophila in hospital and nonhospital plumbing fixtures.
- Fasting-enhanced immune effector mechanisms in obese subjects.
- Inappropriate use of cimetidine in hospitalized patients.
- Disseminated listeriosis presenting as acute hepatitis. Case reports and review of hepatic involvement in listeriosis.
- Effect of acute nutritional deprivation on immune function in mice. II. Response to sublethal radiation.
- Toxoplasma gondii: decreased resistance to intraCellular bacteria in mice.
- Effect of acute nutritional deprivation on host defenses against Listeria monocytogenes--macrophage function.
- Effect of acute nutritional deprivation on immune function in mice. I. Macrophages.
- A comparative study of Legionella micdadei and other nosocomial acquired pneumonia.
- Second serogroup of Legionella hackeliae isolated from a patient With pneumonia.
- Cost savings Associated with use of gentamicin versus tobramycin.
- Effect of Listeria monocytogenes infection on serum levels of colony-stimulating factor and number of progenitor Cells in immune and nonimmune mice.
- 11th serogroup of Legionella pneumophila isolated from a patient With fatal pneumonia.
- Tobramycin and ticarcillin as empiric therapy for febrile patients With granulocytopenia: a reevaluation.
- Macrophage colony-stimulating factor enhances the expression of Fc receptors on murine peritoneal macrophages.
- Peritoneal macrophages exposed to purified macrophage colony-stimulating factor (M-CSF) suppress mitogen- and antigen-stimulated lymphocyte proliferation.
- Effect of acute nutritional deprivation on macrophage colony-stimulating factor and macrophage progenitor Cells in mice.
- Prospective study of pneumonia: unexpected incidence of legionellosis.
- Isolated cardiac aspergillosis after bone marrow transplantation.
- Acute starvation in mice reduces the number of T Cells and suppresses the development of T-Cell-mediated immunity.
- Antigen-specific production of colony-stimulating factors by Listeria monocytogenes-immune, L3T4-positive Cells.
- Fatal bacterial endocarditis as a complication of permanent indwelling catheters. Report of two cases.
- In vitro production of colony-stimulating factors by Listeria-immune spleen Cells.
- Stimulatory effects of purified macrophage colony-stimulating factor on murine resident peritoneal macrophages.
- Macrophage colony-stimulating factor (M-CSF) enhances the capacity of murine macrophages to secrete oxygen reduction products.
- The effect of recombinant human granulocyte macrophage colony-stimulating factor on neutrophil activation in patients With refractory carcinoma.
- Demonstration of a blood-bone marrow barrier to macrophage colony-stimulating factor.
- Recombinant Human granulocyte/macrophage colony-stimulating factor enhances monocyte cytotoxicity and secretion of tumor necrosis factor alpha and interferon in cancer...
- Cloned L3T4+ T lymphocytes protect mice against Listeria monocytogenes by secreting IFN-gamma.
- Legionella infection in transplant patients.
- Biological effects of recombinant Human GM-CSF.
- Accessory function of Kupffer Cells in the antigen-specific blastogenic response of an L3T4+ T-lymphocyte clone to Listeria monocytogenes.
- The role of colony-stimulating factors in host defenses.
- Production of colony-stimulating factors during pneumonia caused by Chlamydia trachomatis.
- Evaluation of the influence of FK 506, rapamycin, and cyclosporine on processing and presentation of particulate antigen by macrophages: assessment of a drug "carry-ov...
- Tumor necrosis factor, macrophage colony-stimulating factor, and interleukin 1 production Within sponge matrix allografts.
- Effector function of hepatocytes and Kupffer Cells in the resolution of systemic bacterial infections.
- Primary listerial infections are exacerbated in mice administered neutralizing antibody to macrophage colony-stimulating factor.
- Solitary obstructing plasmacytoma of the colon.
- Dissociation of effector functions in populations of activated macrophages.
- Pseudomonas maltophilia causing heroin-Associated infective endocarditis.
- Infectious endocarditis caused by Rothia dentocariosa.
- Studies of macrophage function during Trichinella spiralis infection in mice.
- Cell-mediated immunity and its role in resistance to infection.
- Use of quantitative bacteriologic techniques to diagnose catheter-related sepsis.
- Role for activated macrophages in resistance against Trichinella spiralis.
- Studies on the regulation of lymphocyte reactivity by Normal and activated macrophages.
Schools
SUNY Dwnstate M C Coll Med
Columbia UniversityHarlem Hospital Center
Johns Hopkins University
Procedures Preformed
- C-Section
- Colposcopy
- Contraceptive Management
- Destruction or Excision of Lesion of Cervix (incl. LEEP)
- View All
Conditions Treated
- Cervicitis
- Ectopic Pregnancy
- Encounters for High-Risk Pregnancies
- Encounters for Normal Pregnancies
- View All
Doctors Specialties
- Infectious Disease
Accepted Insurances
Education
-
Brigham and Women's Hospital -
Harvard Medical School
Hospital
-
Rhode Island Hospital
Drug Facts
| NPI NUMBER | 1598792525 | |
| NPPES Provider LastName | WING | |
| NPPES Provider FirstName | EDWARD | |
| NPPES Provider ZIPCode | 234353764 | |
| NPPES Provider State | VA | |
| Specialty Description | Obstetrics/Gynecology | |
| Total Claim Count | 19.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1598792525 |
| Last Name Of The Provider | WING |
| First Name Of The Provider | EDWARD |
| View All |