Professor of Neurological Surgery
Neurosurgery
Doctor Dzung H. Dinh, MD works in the field of Neurosurgery. He studied medicine at University of Iowa Carver College of Medicine. University of Iowa Carver College of Medicine is ranked 29/16 in Research/PrimaryCare. He has 9 awards "CMS Meaningful Use Stage 1 Certification", "Americas Top Physicians", "Residency Teaching Award for Teaching Excellence, Neurological Surgery", "Phi Beta Kappa", "Phi Kappa Phi", "General Science Scholarship", "National Honor Society", "Fellow (FACS)" and "Fellow (FAANS)". Dzung H. Dinh, MD is also published. He has 101 publications published. The lastest publication: "Mesenchymal stem cells in the treatment of spinal cord injuries: A review.' Dzung Dinh accepts Medicare payments and is registered with Medicare.gov.
Publications
- Mesenchymal stem cells in the treatment of Spinal cord injuries: A review.
- Myelopathy in patients under chronic steroid therapy: remember spinal epidural lipomatosis.
- Radiation-induced hypomethylation triggers urokinase plasminogen activator transcription in meningioma cells.
- Role of MMP-2 in the regulation of IL-6/Stat3 survival signaling via interaction with ?5?1 integrin in glioma.
- Apoptosis Induced by Knockdown of uPAR and MMP-9 is Mediated by Inactivation of EGFR/STAT3 Signaling in Medulloblastoma.
- Blockade of Sox4 mediated DNA Repair by SPARC Enhances Radioresponse in Medulloblastoma.
- Cord blood stem cells reverts glioma stem cell EMT by down regulating transcriptional activation of Sox2 and Twist1.
- Metabolic remodeling precedes mitochondrial outer membrane permeabilization in human glioma xenograft cells.
- MMP-9 induces CD44 cleavage and CD44 mediated cell migration in glioblastoma xenograft cells.
- uPAR and cathepsin B downregulation induces apoptosis by targeting calcineurin A to BAD via Bcl-2 in glioma.
- Transcriptional Repression of Mad-Max Complex by Human Umbilical Cord Blood Stem Cells Downregulates ERK in Glioblastoma.
- Chk2-mediated G2/M cell cycle arrest maintains radiation resistance in malignant meningioma cells.
- MMP-9 silencing regulates hTERT expression via ?1 integrin-mediated FAK signaling and induces senescence in glioma xenograft cells.
- Gadd45a sensitizes Medulloblastoma cells to irradiation and suppresses MMP-9-mediated EMT.
- Mechanism of p27 upregulation induced by downregulation of cathepsin B and uPAR in glioma.
- SPARC mediates Src-induced disruption of actin cytoskeleton via inactivation of small GTPases Rho-Rac-Cdc42.
- p53- and Bax-Mediated Apoptosis in Injured Rat Spinal Cord.
- SPARC Stimulates Neuronal Differentiation of Medulloblastoma Cells via the Notch1/Signal Transducer and Activator of Transcription 3 (STAT3) Pathway.
- Human umbilical Cord blood-derived mesenchymal stem cells upregulate myelin basic protein in shiverer mice.
- siRNA-mediated downregulation of MMP-9 and uPAR in combination with radiation induces G2/M cell-cycle arrest in Medulloblastoma.
- MMP-2 siRNA Inhibits Radiation-Enhanced Invasiveness in Glioma Cells.
- Regulation of DNA Repair Mechanism in Human Glioma Xenograft Cells both In Vitro and In Vivo in Nude Mice.
- Downregulation of Focal Adhesion Kinase (FAK) by Cord blood stem cells inhibits angiogenesis in glioblastoma.
- Urokinase plasminogen activator receptor and/or matrix metalloproteinase-9 inhibition induces apoptosis signaling through lipid rafts in glioblastoma xenograft cells.
- Human umbilical Cord blood stem cells show PDGF-D-dependent glioma cell tropism in vitro and in vivo.
- Upregulation of PTEN in glioma cells by Cord blood mesenchymal stem cells inhibits migration via downregulation of the PI3K/Akt pathway.
- Downregulation of uPAR and cathepsin B induces apoptosis via regulation of Bcl-2 and Bax and inhibition of the PI3K/Akt pathway in Gliomas.
- Co-depletion of cathepsin B and uPAR induces G0/G1 arrest in glioma via FOXO3a mediated p27 upregulation.
- Cord blood stem cell-mediated induction of apoptosis in glioma downregulates X-linked inhibitor of apoptosis protein (XIAP).
- MMP-9, uPAR and cathepsin B silencing downregulate integrins in human glioma xenograft cells in vitro and in vivo in nude mice.
- Suppression of uPAR retards radiation-induced invasion and migration mediated by integrin β1/FAK signaling in Medulloblastoma.
- Suppression of uPA and uPAR attenuates angiogenin mediated angiogenesis in endothelial and glioblastoma cell lines.
- Neuronal apoptosis is inhibited by cord blood stem cells after spinal cord Injury.
- Human umbilical cord blood stem cells upregulate matrix metalloproteinase-2 in rats after spinal cord Injury.
- Stem cells downregulate the elevated levels of tissue plasminogen activator in rats after spinal cord Injury.
- Neuroprotection by Cord blood stem cells against glutamate-induced apoptosis is mediated by Akt pathway.
- Umbilical cord blood stem cell mediated downregulation of fas improves functional recovery of rats after spinal cord Injury.
- Mesenchymal stem cells from rat bone marrow downregulate caspase-3-mediated apoptotic pathway after spinal cord Injury in rats.
- Intraperitoneal injection of a hairpin RNA-expressing plasmid targeting urokinase-type plasminogen activator (uPA) receptor and uPA retards angiogenesis and inhibits i...
- Restoration of tissue factor pathway inhibitor-2 in a human glioblastoma cell line triggers caspase-mediated pathway and apoptosis.
- Transfection with anti-p65 intrabody suppresses invasion and angiogenesis in glioma cells by blocking nuclear factor-kappaB transcriptional activity.
- Axonal remyelination by cord blood stem cells after spinal cord Injury.
- RNA interference-mediated simultaneous down-regulation of urokinase-type plasminogen activator receptor and cathepsin B induces caspase-8-mediated apoptosis in SNB19 h...
- Recombinant adeno-associated virus (rAAV) expressing TFPI-2 inhibits invasion, angiogenesis and Tumor growth in a human glioblastoma cell line.
- Specific interference of urokinase-type plasminogen activator receptor and matrix metalloproteinase-9 gene expression induced by double-stranded RNA results in decreas...
- Adenovirus-mediated expression of antisense urokinase plasminogen activator receptor and antisense cathepsin B inhibits Tumor growth, invasion, and angiogenesis in gli...
- Downregulation of uPA, uPAR and MMP-9 using small, interfering, hairpin RNA (siRNA) inhibits glioma cell invasion, angiogenesis and Tumor growth.
- Glial cell-induced endothelial morphogenesis is inhibited by interfering with extracellular signal-regulated kinase signaling.
- Synergistic down-regulation of urokinase plasminogen activator receptor and matrix metalloproteinase-9 in SNB19 glioblastoma cells efficiently inhibits glioma cell inv...
- Simultaneous downregulation of uPAR and MMP-9 induces overexpression of the FADD-associated protein RIP and activates caspase 9-mediated apoptosis in Gliomas.
- Induction of apoptosis in glioma cells requires cell-to-cell contact with human umbilical Cord blood stem cells.
- Suppression of uPA and uPAR blocks radiation-induced MCP-1 mediated recruitment of endothelial cells in meningioma.
- Specific knockdown of uPA/uPAR attenuates invasion in glioblastoma cells and xenografts by inhibition of cleavage and trafficking of Notch -1 receptor.
- MMP-2 downregulation mediates differential regulation of cell death via ErbB-2 in glioma xenografts.
- Radiation-inducible silencing of uPA and uPAR in vitro and in vivo in meningioma.
- RNAi-mediated downregulation of MMP-2 activates the extrinsic apoptotic pathway in human glioma xenograft cells.
- Doxorubicin-mediated apoptosis in glioma cells requires NFAT3.
- uPA/uPAR downregulation inhibits radiation-induced migration, invasion and angiogenesis in IOMM-Lee meningioma cells and decreases Tumor growth in vivo.
- RNAi-mediated downregulation of radiation-induced MMP-9 leads to apoptosis via activation of ERK and Akt in IOMM-Lee cells.
- Sense p16 and antisense uPAR bicistronic construct inhibits angiogenesis and induces glioma cell death.
- RNAi-mediated abrogation of cathepsin B and MMP-9 gene expression in a malignant meningioma cell line leads to decreased Tumor growth, invasion and angiogenesis.
- Downregulation of matrix metalloproteinase-2 (MMP-2) utilizing adenovirus-mediated transfer of small interfering RNA (siRNA) in a novel Spinal metastatic melanoma model.
- RNA interference-mediated targeting of urokinase plasminogen activator receptor and matrix metalloproteinase-9 gene expression in the IOMM-lee malignant meningioma cel...
- Down-regulation of uPAR and uPA activates caspase-mediated apoptosis and inhibits the PI3K/AKT pathway.
- SPARC-induced migration of glioblastoma cell lines via uPA-uPAR signaling and activation of small GTPase RhoA.
- RNAi-mediated inhibition of cathepsin B and uPAR leads to decreased cell invasion, angiogenesis and tumor growth in Gliomas.
- Down-regulation of uPAR and cathepsin B retards cofilin dephosphorylation.
- RNAi-mediated downregulation of urokinase plasminogen activator and its receptor in human meningioma cells inhibits Tumor invasion and growth.
- Restoration of tissue factor pathway inhibitor inhibits invasion and Tumor growth in vitro and in vivo in a malignant meningioma cell line.
- Downregulation of uPA inhibits migration and PI3k/Akt signaling in glioblastoma cells.
- Physiological and chemical inducers of tissue factor pathway inhibitor-2 in human glioma cells.
- Triterpenoids from Glycine max decrease invasiveness and induce caspase-mediated cell death in human SNB19 glioma cells.
- Promoter methylation and silencing of the tissue factor pathway inhibitor-2 (TFPI-2), a gene encoding an inhibitor of matrix metalloproteinases in human glioma cells.
- Expression of antisense uPAR and antisense uPA from a bicistronic adenoviral construct inhibits glioma cell invasion, Tumor growth, and angiogenesis.
- Blockade of cathepsin B expression in human glioblastoma cells is associated with suppression of angiogenesis.
- Activation of caspase-9 with irradiation inhibits invasion and angiogenesis in SNB19 human glioma cells.
- Inhibition of cathepsin B and MMP-9 gene expression in glioblastoma cell line via RNA interference reduces Tumor cell invasion, Tumor growth and angiogenesis.
- Suppression of glioma invasion and growth by adenovirus-mediated delivery of a bicistronic construct containing antisense uPAR and sense p16 gene sequences.
- Minimal and inducible regulation of tissue factor pathway inhibitor-2 in human Gliomas.
- Downregulation of MMP-9 in ERK-mutated stable transfectants inhibits glioma invasion in vitro.
- Modulation of invasive properties of human glioblastoma cells stably expressing amino-terminal fragment of urokinase-type plasminogen activator.
- Adenovirus-mediated expression of antisense MMP-9 in glioma cells inhibits Tumor growth and invasion.
- Modulation of cystatin C expression impairs the invasive and Tumorigenic potential of human glioblastoma cells.
- Expression of matrix metalloproteinases, their inhibitors, and urokinase plasminogen activator in human Meningiomas.
- Downregulation of urokinase-type plasminogen activator receptor (uPAR) induces caspase-mediated cell death in human glioblastoma cells.
- Glioma cells deficient in urokinase plaminogen activator receptor expression are susceptible to Tumor necrosis factor-alpha-related apoptosis-inducing ligand-induced a...
- Down-regulation of integrin alpha(v)beta(3) expression and integrin-mediated signaling in glioma cells by adenovirus-mediated transfer of antisense urokinase-type plas...
- A novel function of tissue factor pathway inhibitor-2 (TFPI-2) in human glioma invasion.
- Adenovirus-mediated antisense urokinase-type plasminogen activator receptor gene transfer reduces tumor cell invasion and metastasis in non-small cell lung Cancer cell...
- Down-regulation of cathepsin B expression impairs the invasive and Tumorigenic potential of human glioblastoma cells.
- Adenovirus-mediated transfer of siRNA against MMP-2 mRNA results in impaired invasion and Tumor-induced angiogenesis, induces apoptosis in vitro and inhibits Tumor gro...
- Modulation of endothelial cell morphogenesis in vitro by MMP-9 during glial-endothelial cell interactions.
- Cathepsin B and uPAR knockdown inhibits Tumor-induced angiogenesis by modulating VEGF expression in glioma.
- A novel role of tissue factor pathway inhibitor-2 in apoptosis of malignant human Gliomas.
- MMP-2 mediates mesenchymal stem cell tropism towards Medulloblastoma tumors.
- Cathepsin B facilitates autophagy-mediated apoptosis in SPARC overexpressed primitive neuroectodermal Tumor cells.
- Stable transfection of urokinase-type plasminogen activator antisense construct modulates invasion of human glioblastoma cells.
- Elevated levels of cathepsin B in human glioblastoma cell lines.
- The role of MMP-9 in the anti-angiogenic effect of secreted protein acidic and rich in cysteine.
- Chiari I malformation with traumatic syringomyelia and spontaneous resolution: case report and literature review.
Schools
Roy J Lucille a Carver College Of Medicine At University Of Iowa
Parkland Memorial Hospital
St Francis HospU Ill
Procedures Preformed
- CerebroSpinal Fluid (CSF) Shunt - Insertion, Repair or Removal
- Craniectomy, Craniotomy, Surgery of Skull Base, Neuroendoscopy
- Disc Replacement
- Dural Repair or Other Spinal Spinal Cord Repair
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Conditions Treated
- Brain and Nervous System Cancer (incl. Gliomas, Astrocytoma, Schwannoma, Medulloblastoma, Chordoma)
- Brain Aneurysm
- Brain Cancer
- Cerebral Artery Thrombosis
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Doctors Specialties
Accepted Insurances
Awards
- CMS Meaningful Use Stage 1 Certification
- America’s Top Physicians
- Residency Teaching Award for Teaching Excellence, Neurological Surgery
- Phi Beta Kappa
- Phi Kappa Phi
- General Science Scholarship
- National Honor Society
- Fellow (FACS)
- Fellow (FAANS)
Education
-
University of Iowa Carver College of Medicine
Drug Facts
NPI NUMBER |
|
1679513642 |
NPPES Provider LastName |
|
DINH |
NPPES Provider FirstName |
|
DZUNG |
NPPES Provider ZIPCode |
|
61605 |
NPPES Provider State |
|
IL |
Specialty Description |
|
Neurosurgery |
Total Claim Count |
|
333.0 |
Distinct Opioid Count |
|
2.0 |
Opioid Claim Count |
|
103.0 |
Percent Opioid Claims |
|
30.93 |
Helpful Reviews
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Anonymous Review
Medicare Facts
National Provider Identifier [NPI] |
1679513642 |
Last Name Of The Provider |
DINH |
First Name Of The Provider |
DZUNG |
View All |
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