Assistant Professor, Medicine, Indiana University School of Medicine
Infectious Disease
Dr. Diane M. Janowicz, MD
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Diane Janowicz works in the field of Infectious Disease. She graduated from Indiana University School of Medicine. Indiana University School of Medicine is ranked 45 in research and 47 in primary care medicine Diane Janowicz is a published doctor. She published 23 publications, including: 'Outer membrane protein P4 is not required for virulence in the human challenge model of Haemophilus ducreyi infection.' She is registered with Medicare and accepts Medicare payments.
Publications
- A (p)ppGpp null mutant of Haemophilus ducreyi is partially attenuated in humans due to multiple conflicting phenotypes.
- Permeases of the sap transporter are required for cathelicidin resistance and virulence of Haemophilus ducreyi in humans.
- Sialylation of Lipooligosaccharides Is Dispensable for the Virulence of Haemophilus ducreyi in Humans.
- Expression of the Flp Proteins by Haemophilus ducreyi is necessary for virulence in human volunteers.
- A Haemophilus ducreyi CpxR Deletion Mutant Is Virulent in Human Volunteers.
- Activation of the CpxRA system by deletion of cpxA impairs the ability of Haemophilus ducreyi to infect humans.
- Role played by CD4+FOXP3+ regulatory T Cells in suppression of host responses to Haemophilus ducreyi during experimental infection of human volunteers.
- Haemophilus ducreyi SapA contributes to cathelicidin resistance and virulence in humans.
- Host-pathogen interplay of Haemophilus ducreyi.
- Mechanism of human natural killer cell activation by Haemophilus ducreyi.
- Inactivation of the Haemophilus ducreyi luxS gene affects the virulence of this pathogen in human subjects.
- Experimental infection of human volunteers with Haemophilus ducreyi: fifteen years of clinical data and experience.
- A fibrinogen-binding lipoprotein contributes to the virulence of Haemophilus ducreyi in humans.
- Dysregulated immune profiles for skin and dendritic cells are associated with increased host susceptibility to Haemophilus ducreyi infection in human volunteers.
- Experimental infection with Haemophilus ducreyi in persons who are infected with HIV does not cause local or augment systemic viral replication.
- Expression of Haemophilus ducreyi collagen binding outer membrane protein NcaA is required for virulence in swine and human challenge models of chancroid.
- Expression of the LspA1 and LspA2 proteins by Haemophilus ducreyi is required for virulence in human volunteers.
- Outer membrane protein P4 is not required for virulence in the human challenge model of Haemophilus ducreyi infection.
- A DltA mutant of Haemophilus ducreyi Is partially attenuated in its ability to cause pustules in human volunteers.
- Expression of OmpP2A and OmpP2B is not required for pustule formation by Haemophilus ducreyi in human volunteers.
- Successful treatment of CMV ventriculitis immune reconstitution syndrome.
- Identification of Haemophilus ducreyi genes expressed during human infection.
Schools
Indiana University School of Medicine
Doctors Specialties
- Infectious Disease Medicine
- Infectious Disease
Accepted Insurances
Education
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Indiana University School of Medicine
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1881604874 |
| Last Name Of The Provider | JANOWICZ |
| First Name Of The Provider | DIANE |
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