VA Staff Neurologist and UCLA Professor
Neurology
Dr. Denson G. Fujikawa, MD
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Denson Fujikawa works in the field of Neurology. He attended Keck School of Medicine of USC. Keck School of Medicine of USC is ranked 31/72 in Research/PrimaryCare. Denson Fujikawa is a published doctor and has 43 publications published. The lastest was: 'An electroencephalographic study comparing maximum blink rates in schizophrenic and nonschizophrenic psychiatric patients and nonpsychiatric control subjects.'
Publications
- Methamphetamine-induced neuronal necrosis: the role of electrographic seizure Discharges.
- Chronic epileptic encephalopathy in adult patients with bilaterally synchronous frequent and/or prolonged subclinical epileptiform Discharges.
- Nuclear translocation of mitochondrial cytochrome c, lysosomal cathepsins B and D, and three other death-promoting proteins within the first 60 minutes of generalized ...
- Mild as well as severe insults produce necrotic, not apoptotic, cells: evidence from 60-min seizures.
- Caspase-dependent programmed cell death pathways are not activated in generalized seizure-induced neuronal death.
- Prolonged seizures and cellular injury: understanding the connection.
- Neuronal death in mesial temporal Sclerosis: separating morphology from mechanism.
- Hypoxic neuronal necrosis: protein synthesis-independent activation of a cell death program.
- Caspase-3 is not activated in seizure-induced neuronal necrosis with internucleosomal DNA cleavage.
- Nonconvulsive status epilepticus with frontal features: quantitating severity of subclinical epileptiform Discharges provides a marker for treatment efficacy, recurren...
- Effects of AMPA-receptor and voltage-sensitive sodium channel blockade on high potassium-induced glutamate release and neuronal death in vivo.
- Apoptosis: ignoring morphology and focusing on biochemical mechanisms will not eliminate confusion.
- Chronically Impaired Frontal Lobe Function from Subclinical Epileptiform Discharges.
- Confusion between neuronal apoptosis and activation of programmed cell death mechanisms in acute necrotic insults.
- Seizure-induced neuronal necrosis: implications for programmed cell death mechanisms.
- Status epilepticus-induced neuronal loss in humans without systemic complications or Epilepsy.
- Kainic acid-induced seizures produce necrotic, not apoptotic, neurons with internucleosomal DNA cleavage: implications for programmed cell death mechanisms.
- Lithium-pilocarpine-induced status epilepticus produces necrotic neurons with internucleosomal DNA fragmentation in adult rats.
- An electroencephalographic study comparing maximum blink rates in schizophrenic and nonschizophrenic psychiatric patients and nonpsychiatric control subjects.
- Effects of N-methyl-D-aspartate-receptor blockade on high-potassium-induced neuronal death and glutamate release.
- In vivo elevation of extracellular potassium in the rat amygdala increases extracellular glutamate and aspartate and damages neurons.
- The temporal evolution of neuronal damage from pilocarpine-induced status epilepticus.
- Anticonvulsants for eclampsia.
- Neuroprotective effect of ketamine administered after status epilepticus onset.
- The competitive NMDA receptor antagonist CGP 40116 protects against status epilepticus-induced neuronal damage.
- Pathophysiological mechanisms of Brain damage from status epilepticus.
- Neuropathological changes during generalized seizures in newborn monkeys.
- Single-dose phenytoin infusion.
- Clinical characterization of Alzheimer's Disease: reliability of 'age at onset' and a new descriptor, 'age at shift'.
- Local Cerebral glucose utilization during status epilepticus in newborn primates.
- Cerebral glucose metabolic patterns in Alzheimer's disease. Effect of gender and age at dementia onset.
- Posthypoxic treatment with MK-801 reduces hypoxic-ischemic damage in the neonatal rat.
- Generalized seizures deplete Brain energy reserves in normoxemic newborn monkeys.
- Low [3H]cytochalasin B binding in the Cerebral cortex of newborn rat.
- Cerebral hypoxia-ischemia in immature rats: methodological considerations.
- The effects of adrenergic, opioid and pancreatic polypeptidergic compounds on feeding and other behaviors in neonatal leghorn chicks.
- Preferential blood flow to Brainstem during generalized seizures in the newborn marmoset monkey.
- Metabolic anatomy of generalized bicuculline seizures in the newborn marmoset monkey.
- Brain protein metabolism in epilepsy.
- Phaeohyphomycosis of Brain: granulomatous encephalitis caused by Drechslera spicifera.
- Letter: Treatment by vertebral manipulation.
- Nonconvulsive status epilepticus: Epilepsy Research Foundation workshop reports.
Schools
University of Southern California Keck School of Medicine
Conditions Treated
- Autonomic Disorders
- Brain Aneurysm
- Cerebral Artery Thrombosis
- Convulsions
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Doctors Specialties
- Neurology
Accepted Insurances
Education
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Harbor - UCLA Medical Center -
UCLA Medical Center -
New York Presbyterian Hospital - Columbia Campus -
Keck School of Medicine of USC
Hospital
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Ronald Reagan UCLA Medical Center
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