Physician

Family Medicine

David J. Steger, MD practices Family Medicine. He graduated from Saint Louis University School of Medicine. Saint Louis University School of Medicine has a rank of 67 in research and a 57 in primary care David J. Steger, MD is also published. He has 35 publications published. The lastest publication: "Activation domain-specific and general transcription stimulation by native histone acetyltransferase complexes.'

Publications

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• Histone deacetylase 3 is an epigenomic brake in macrophage alternative activation.
• Repressor transcription factor 7-like 1 promotes adipogenic competency in precursor cells.
• Propagation of adipogenic signals through an epigenomic transition state.
• Cell-specific determinants of peroxisome proliferator-activated receptor gamma function in adipocytes and macrophages.
• Re-expression of GATA2 cooperates with peroxisome proliferator-activated receptor-gamma depletion to revert the adipocyte phenotype.
• Adipocyte-specific expression of murine resistin is mediated by synergism between peroxisome proliferator-activated receptor gamma and CCAAT/enhancer-binding proteins.
• Retinol saturase promotes adipogenesis and is downregulated in obesity.
• PPARgamma and C/EBP factors orchestrate adipocyte biology via adjacent binding on a genome-wide scale.
• Selective partial agonism of liver X receptor alpha is related to differential corepressor recruitment.
• DOT1L/KMT4 recruitment and H3K79 methylation are ubiquitously coupled with gene transcription in mammalian cells.
• Identification and characterization of a selective peroxisome proliferator-activated receptor beta/delta (NR1C2) antagonist.
• Genetic analysis of chromatin remodeling using budding yeast as a model.
• Regulation of chromatin remodeling by inositol polyphosphates.
• Chromatin decouples promoter threshold from dynamic range.
• Adipogenic hotspots: where the action is.
• The chromo domain protein chd1p from budding yeast is an ATP-dependent chromatin-modifying factor.
• Distribution of acetylated histones resulting from Gal4-VP16 recruitment of SAGA and NuA4 complexes.
• Remodeling chromatin structures for transcription: what happens to the histones?
• Activation domain-specific and general transcription stimulation by native histone acetyltransferase complexes.
• Stable co-occupancy of transcription factors and histones at the HIV-1 enhancer.
• Transcriptional activators direct histone acetyltransferase complexes to nucleosomes.
• Purified histone acetyltransferase complexes stimulate HIV-1 transcription from preassembled nucleosomal arrays.
• A novel H2A/H4 nucleosomal histone acetyltransferase in Tetrahymena thermophila.
• Analysis of transcription factor-mediated remodeling of nucleosomal arrays in a purified system.
• Coordinate control of the alpha- and beta-subunit genes of human chorionic gonadotropin by trophoblast-specific element-binding protein.
• Activation domain-mediated targeting of the SWI/SNF complex to promoters stimulates transcription from nucleosome arrays.
• A subset of TAF(II)s are integral components of the SAGA complex required for nucleosome acetylation and transcriptional stimulation.
• Regulation of transcription by multisubunit complexes that alter nucleosome structure.
• GATA-binding proteins regulate the human gonadotropin alpha-subunit gene in the placenta and pituitary gland.
• Evolution of placenta-specific gene expression: comparison of the equine and human gonadotropin alpha-subunit genes.
• AP-2 family members regulate basal and cAMP-induced expression of human chorionic gonadotropin.
• Analysis of nucleosome disruption by ATP-driven chromatin remodeling complexes.
• Transcriptional analysis of purified histone acetyltransferase complexes.
• The Nuclear Receptor Rev-erbα Regulates Adipose Tissue-Specific Fibroblast Growth Factor 21 (FGF21) Signaling.

Schools

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• Columbia University College Of Physicians and Surgeons

• Martin Army Hospital

Doctors Specialties

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• Family Medicine

Accepted Insurances

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Education

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• Saint Louis University School of Medicine

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