Professor, Medicine, UCSD School of Medicine
Allergy & Immunology
Dr. David H. Broide, MD
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David Broide practices Allergy & Immunology. He received 2 awards: "CMS Meaningful Use Stage 1 Certification" and "Fellow (FAAAAI)". Doctor David H. Broide, MD is a published doctor. He has 100 papers published. The most current publication "miR-232724 clusters control effector T cell differentiation and function." He is registered with Medicare.
Publications
- Group 2 innate lymphocytes (ILC2s) are enriched in active eosinophilic esophagitis.
- Long-term assessment of esophageal remodeling in patients with pediatric eosinophilic esophagitis treated with topical corticosteroids.
- Increased ILC2s in the eosinophilic nasal polyp endotype are associated with corticosteroid responsiveness.
- Id2 and Id3 maintain the regulatory T cell pool to suppress inflammatory Disease.
- Association between specific timothy grass antigens and changes in TH1- and TH2-cell responses following specific immunotherapy.
- Synthetic di-sulfated iduronic acid attenuates Asthmatic response by blocking T-cell recruitment to inflammatory sites.
- TGF-β1-induced phospholamban expression alters esophageal smooth muscle cell contraction in patients with eosinophilic esophagitis.
- ORMDL3 Transgenic Mice Have Increased Airway Remodeling and Airway Responsiveness Characteristic of Asthma.
- Smad3 Deficient Mice Have Reduced Esophageal Fibrosis and Angiogenesis in a Mouse Model of Egg Induced Eosinophilic Esophagitis.
- Allergen challenge in Allergic rhinitis rapidly induces increased peripheral blood type 2 innate lymphoid cells that express CD84.
- Targeting AMCase reduces esophageal eosinophilic inflammation and remodeling in a mouse model of egg induced eosinophilic esophagitis.
- Prostaglandin D2 regulates human type 2 innate lymphoid cell chemotaxis.
- Sialyltransferase ST3Gal-III Regulates Siglec-F Ligand Formation and Eosinophilic Lung Inflammation in Mice.
- Lung type 2 innate lymphoid cells express cysteinyl leukotriene receptor 1, which regulates TH2 cytokine production.
- Myeloid cell HIF-1α regulates Asthma airway resistance and eosinophil function.
- Anti-IL-5 therapy reduces mast cell and IL-9 cell numbers in pediatric patients with eosinophilic esophagitis.
- Lung-resident tissue macrophages generate Foxp3+ regulatory T cells and promote airway tolerance.
- Previously undescribed grass pollen antigens are the major inducers of T helper 2 cytokine-producing T cells in Allergic individuals.
- A strategy to determine HLA class II restriction broadly covering the DR, DP, and DQ allelic variants most Commonly expressed in the general population.
- Hypoxia potentiates allergen induction of HIF-1?, chemokines, airway inflammation, TGF-?1, and airway remodeling in a mouse model.
- The identification of potentially pathogenic and therapeutic epitopes from Common human allergens.
- ORMDL3 is an inducible lung epithelial gene regulating metalloproteases, chemokines, OAS, and ATF6.
- STAT6 Regulates Natural Helper Cell Proliferation during Lung Inflammation Initiated by Alternaria.
- Histamine-releasing factor has a proinflammatory role in mouse models of asthma and Allergy.
- Siglec-f inhibition reduces esophageal eosinophilia and angiogenesis in a mouse model of eosinophilic esophagitis.
- The tumor necrosis factor family member LIGHT is a target for Asthmatic airway remodeling.
- Chronic OVA allergen challenged TNF p55/p75 receptor deficient mice have reduced airway remodeling.
- Advances in mechanisms of asthma, Allergy, and immunology in 2010.
- Mast cells infiltrate the esophageal smooth muscle in patients with eosinophilic esophagitis, express TGF-β1, and increase esophageal smooth muscle contraction.
- Adiponectin-deficient mice are protected against tobacco-induced inflammation and increased emphysema.
- Persistent airway inflammation and emphysema progression on CT scan in ex-smokers observed for 4 years.
- Allergic rhinitis: Pathophysiology.
- Chronic allergen challenge induces bronchial mast cell accumulation in BALB/c but not C57BL/6 mice and is independent of IL-9.
- Molecular determinants of T cell epitope recognition to the Common Timothy grass allergen.
- Innate Immunity.
- Tobacco smoke promotes lung tumorigenesis by triggering IKKbeta- and JNK1-dependent inflammation.
- Anti-Siglec-F antibody reduces allergen-induced eosinophilic inflammation and airway remodeling.
- Environmental tobacco smoke exposure does not prevent corticosteroids reducing inflammation, remodeling, and airway hyperreactivity in mice exposed to allergen.
- Anti-Siglec-F antibody inhibits oral egg allergen induced intestinal eosinophilic inflammation in a mouse model.
- PI3K gamma-deficient mice have reduced levels of allergen-induced eosinophilic inflammation and airway remodeling.
- CD4+ cells are required for Chronic eosinophilic lung inflammation but not airway remodeling.
- Adiponectin and functional adiponectin receptor 1 are expressed by airway epithelial cells in Chronic obstructive pulmonary disease.
- Immunomodulation of Allergic disease.
- Myeloid differentiation primary response gene 88 is required for the resolution of otitis media.
- Immunologic and inflammatory mechanisms that drive Asthma progression to remodeling.
- Down-regulation of caveolin-1, an inhibitor of transforming growth factor-beta signaling, in acute allergen-induced airway remodeling.
- 3-Hydroxyanthranilic acid inhibits PDK1 activation and suppresses experimental Asthma by inducing T cell apoptosis.
- Computed tomographic scan-diagnosed Chronic obstructive pulmonary disease-emphysema: eotaxin-1 is associated with bronchodilator response and extent of emphysema.
- The pathophysiology of Allergic rhinoconjunctivitis.
- Inhibition of allergen-induced airway remodeling in Smad 3-deficient mice.
- Defining the in vivo function of Siglec-F, a CD33-related Siglec expressed on mouse eosinophils.
- Coexposure to environmental tobacco smoke increases levels of allergen-induced airway remodeling in mice.
- Esophageal remodeling in pediatric eosinophilic esophagitis.
- Reduced peribronchial fibrosis in allergen-challenged MMP-9-deficient mice.
- Remodeling associated expression of matrix metalloproteinase 9 but not tissue inhibitor of metalloproteinase 1 in airway epithelium: modulation by immunostimulatory DNA.
- Immunostimulatory DNA inhibits allergen-induced peribronchial angiogenesis in mice.
- Indoleamine-2,3-dioxygenase modulation of Allergic immune responses.
- Corticosteroids prevent myofibroblast accumulation and airway remodeling in mice.
- Allergen-induced peribronchial fibrosis and mucus production mediated by IkappaB kinase beta-dependent genes in airway epithelium.
- Combination of corticosteroid therapy and allergen avoidance reverses allergen-induced airway remodeling in mice.
- DNA vaccines: an evolving approach to the treatment of Allergic disorders.
- Immunostimulatory sequences of DNA and conjugates in the treatment of Allergic rhinitis.
- Immunostimulatory DNA reverses established allergen-induced airway remodeling.
- Inhibition of experimental Asthma by indoleamine 2,3-dioxygenase.
- Immunostimulatory DNA inhibits transforming growth factor-beta expression and airway remodeling.
- Cutting edge: activation of Toll-like receptor 2 induces a Th2 immune response and promotes experimental Asthma.
- Inhibition of airway remodeling in IL-5-deficient mice.
- Accumulation of peribronchial mast cells in a mouse model of ovalbumin allergen induced Chronic airway inflammation: modulation by immunostimulatory DNA sequences.
- A murine model to study leukocyte rolling and intravascular trafficking in lung microvessels.
- Resolution of airway inflammation following ovalbumin inhalation: comparison of ISS DNA and corticosteroids.
- Constitutive overexpression of IL-5 induces extramedullary hematopoiesis in the spleen.
- Core 2 oligosaccharides mediate eosinophil and neutrophil peritoneal but not lung recruitment.
- Human Mesenchymal Stem Cells Suppress the Stretch-Induced Inflammatory miR-155 and Cytokines in Bronchial Epithelial Cells.
- Towards defining molecular determinants recognized by adaptive immunity in Allergic disease: an inventory of the available data.
- Allergen-induced coexpression of bFGF and TGF-β1 by macrophages in a mouse model of airway remodeling: bFGF induces macrophage TGF-β1 expression in vitro.
- TNFA deletion alters apoptosis as well as caspase 3 and 4 expression during otitis media.
- Genetic immunization for Allergy immunotherapy.
- Chronic OVA allergen challenged Siglec-F deficient mice have increased mucus, remodeling, and epithelial Siglec-F ligands which are up-regulated by IL-4 and IL-13.
- Inactivation of I kappaB-kinase-beta dependent genes in airway epithelium reduces tobacco smoke induced acute airway inflammation.
- Resolution of remodeling in eosinophilic esophagitis correlates with epithelial response to topical corticosteroids.
- Toll-like receptor-9 agonist inhibits airway inflammation, remodeling and hyperreactivity in mice exposed to Chronic environmental tobacco smoke and allergen.
- Reconstitution of the mast cell population in W/Wv mice.
- Advances in mechanisms of asthma, Allergy, and immunology in 2008.
- Potent inhibitors of pro-inflammatory cytokine production produced by a marine-derived bacterium.
- Cytokines and growth factors in airway remodeling in Asthma.
- Prospects for CpG based immunotherapy in asthma and Allergy.
- An antiinflammatory role for IKKbeta through the inhibition of "classical" macrophage activation.
- Airway fibrosis and angiogenesis due to eosinophil trafficking in chronic Asthma.
- New perspectives on mechanisms underlying chronic Allergic inflammation and asthma in 2007.
- Immunotherapy with a ragweed-toll-like receptor 9 agonist vaccine for Allergic rhinitis.
- Role of mast cells in otitis media.
- Induction and inhibition of the Th2 phenotype spread: implications for childhood Asthma.
- Immunomodulation and reversal of airway remodeling in Asthma.
- Fine structure mapping of CIAS1: identification of an ancestral haplotype and a Common FCAS mutation, L353P.
- IL-5 links adaptive and natural Immunity specific for epitopes of oxidized LDL and protects from atherosclerosis.
- New targets for Allergic rhinitis--a disease of civilization.
- Immunologic treatment of Allergic diseases.
- Fast flowing eosinophils: signals for stopping and stepping out of blood vessels.
- miR-23∼27∼24 clusters control Effector T cell differentiation and function.
Schools
University Of Cape Town Faculty Of Health Sciences
Groote Schuur Hospital
Brigham Womens Hospital
Brigham Womens Hospital Waltham
Ucsd Med Center
Procedures Preformed
- Allergy Shots
- Allergy Skin Testing
Conditions Treated
- Allergic Rhinitis
- Anaphylactic Food Allergies
- Anaphylactic Tree Nut Allergy
- Anaphylaxis
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Doctors Specialties
- Internal Medicine
- Allergy
- Immunology
Accepted Insurances
- Aetna HMO
- Choice Plus POS II
- Power Select HMO
- Blue Card PPO
- CaliforniaCare HMO
- Advantage Preferred DirectAccess Plus PPO
- Blue Cross Blue Shield of California PPO
- Pathway X Direct Access PPO
- Pathway X Tiered Direct Access
- Select Plus HMO
- Cigna
- Commercial Insurance Company
- Coventry Health Care
- Federal Employee Program (FEP)
- Medicare Advantage PFFS
- Medicaid
- Medicare MCR
- Molina
- Kaiser PHCS Network PPO
- MultiPlan PPO
- PHCS Network PPO
- Heritage Signature
- Choice Plus POS
- Navigate HMO
- Navigate POS
- Options PPO
- PacifiCare HMO
- United Healthcare
- Worker's Compensation WC
Awards
- CMS Meaningful Use Stage 1 Certification
- Fellow (FAAAAI)
Education
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University of California San Diego -
Brigham and Women's Hospital -
University of Cape Town Faculty of Medicine
Drug Facts
| NPI NUMBER | 1760422992 | |
| NPPES Provider LastName | BROIDE | |
| NPPES Provider FirstName | DAVID | |
| NPPES Provider ZIPCode | 92037 | |
| NPPES Provider State | CA | |
| Specialty Description | Allergy/Immunology | |
| Total Claim Count | 208.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1760422992 |
| Last Name Of The Provider | BROIDE |
| First Name Of The Provider | DAVID |
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