Professor, Medicine, Medical College of Wisconsin
Cardiology
Dr. David Gutterman, MD
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Dr. David Gutterman is an expert in the field of Cardiology. He studied medicine at University of North Carolina at Chapel Hill School of Medicine. University of North Carolina at Chapel Hill School of Medicine is a top medical university. University of North Carolina at Chapel Hill School of Medicine has a rank of 22 in research and a 2 in primary care He over the years received 3 awards: "Fellow (FACC)", "Senior Associate Dean for Research" and "President". Dr. David Gutterman is a published doctor and has 82 publications published. The lastest was: 'Endothelium-derived hyperpolarization and coronary vasodilation: diverse and integrated roles of epoxyeicosatrienoic acids, hydrogen peroxide and gap junctions.' David Gutterman accepts Medicare payments and is listed with Medicare.gov.
Publications
- Endothelium-derived hyperpolarization and Coronary vasodilation: diverse and integrated roles of epoxyeicosatrienoic acids, hydrogen peroxide and gap junctions.
- Acute Exertion Elicits a H2O2-Dependent Vasodilator Mechanism in the Microvasculature of Exercise-Trained but not Sedentary Adults.
- An Acute Rise in Intraluminal Pressure Shifts the Mediator of Flow-Mediated Dilation from Nitric Oxide to Hydrogen Peroxide in Human Arterioles.
- Ceramide Changes the Mediator of Flow-Induced Vasodilation from Nitric Oxide to Hydrogen Peroxide in the Human Microcirculation.
- The Vascular Renin Angiotensin System Contributes to Blunted Vasodilation Induced by Transient High Pressure in Human Adipose Microvessels.
- Exercise and vascular function: how much is too much?
- Vascular dysfunction in diabetes mellitus: large conductance calcium-activated potassium channels as part of a subsarcolemmal signaling soiree.
- In-depth proteomic analysis of human tropomyosin by top-down mass spectrometry.
- The Intermediate-conductance Calcium-activated Potassium Channel KCa3.1 Regulates Vascular Smooth Muscle Cell Proliferation via Controlling Calcium-dependent Signaling.
- Mitochondrial reactive oxygen species and vascular function: less is more.
- Ecto-5'-nucleotidase, CD73, is an endothelium-derived hyperpolarizing factor synthase.
- Diversity in Mechanisms of Endothelium-Dependent Vasodilation in Health and Disease.
- Adverse Alterations in Mitochondrial Function Contribute to Type 2 Diabetes Mellitus-Induced Endothelial Dysfunction in Humans.
- Nitric oxide synthase-dependent vasodilation of human subcutaneous arterioles correlates with noninvasive measurements of endothelial function.
- Acute Exposure to Low Glucose Rapidly Induces Endothelial Dysfunction and Mitochondrial Oxidative Stress: Role for AMP Kinase.
- H2O2-Induced Dilation in Human Coronary Arterioles: Role of Protein Kinase G Dimerization and Large-Conductance Ca2+-Activated K+ Channel Activation.
- Activation of endothelial TRPV4 mediates flow-induced dilation in human Coronary arterioles: role of Ca2+ entry and mitochondrial ROS signaling.
- Regulation of the human Coronary microcirculation.
- Human microvascular dysfunction and apoptotic injury induced by AL amyloidosis light chain proteins.
- Soluble epoxide hydrolase contamination of specific catalase preparations inhibits epoxyeicosatrienoic acid vasodilation of rat renal arterioles.
- Folic Acid Supplementation Improves Vascular Function in Professional Dancers With Endothelial Dysfunction.
- ROS induced ROS release in vascular biology: redox-redox signaling.
- Regulation of Endothelial Function by Mitochondrial Reactive Oxygen Species.
- Resistance and aerobic exercise protects against Acute endothelial impairment induced by a single exposure to hypertension during exertion.
- H2O2 is the transferrable factor mediating flow-induced dilation in human Coronary arterioles.
- Association between the female athlete triad and endothelial dysfunction in dancers.
- Transient receptor potential channel activation and endothelium-dependent dilation in the systemic circulation.
- Cigarette smoking impairs Na+-K+-ATPase activity in the human Coronary microcirculation.
- Measuring FMD in the brachial Artery: how important is QRS gating?
- Resurrecting hope for antioxidant treatment of cardiovascular Disease: focus on mitochondria.
- Altered mitochondrial membrane potential, mass, and morphology in the mononuclear cells of humans with type 2 diabetes.
- Folic acid supplementation improves vascular function in amenorrheic runners.
- TRPV4-mediated endothelial Ca2+ influx and vasodilation in response to shear stress.
- Prevalence of the female athlete triad in high school athletes and sedentary students.
- Bradykinin-induced dilation of human Coronary arterioles requires NADPH oxidase-derived reactive oxygen species.
- Transient receptor potential vanilloid type 4-deficient mice exhibit impaired endothelium-dependent relaxation induced by acetylcholine in vitro and in vivo.
- Effects of limb electrode placement on the 12- and 16-lead electrocardiogram.
- The intermediate-conductance calcium-activated potassium channel KCa3.1 contributes to atherogenesis in mice and humans.
- Endothelial cytoskeletal elements are critical for flow-mediated dilation in human Coronary arterioles.
- Age-related attenuation of isoflurane preconditioning in human Atrial cardiomyocytes: roles for mitochondrial respiration and sarcolemmal adenosine triphosphate-sensit...
- Benefit of low-fat over low-carbohydrate diet on endothelial health in obesity.
- Hydrogen peroxide inhibits cytochrome p450 epoxygenases: interaction between two endothelium-derived hyperpolarizing factors.
- Epoxyeicosatrienoic acids, TRP channels, and intracellular Ca2+ in the vasculature: an endothelium-derived endothelium-hyperpolarizing factor?
- Ebselen reduces nitration and restores voltage-gated potassium channel function in small Coronary arteries of diabetic rats.
- Athletic amenorrhea and endothelial dysfunction.
- Combating nitrate tolerance: a novel endogenous mechanism.
- The female athlete triad and Cardiovascular dysfunction.
- Mitochondrial reactive oxygen species-mediated signaling in endothelial cells.
- The mechanism of flow-induced dilation in human adipose arterioles involves hydrogen peroxide during CAD.
- Relating cardiac performance with oxygen consumption: historical observations continue to spawn scientific discovery.
- Protective effects of epoxyeicosatrienoic acids on human endothelial cells from the pulmonary and Coronary vasculature.
- The effect of exertional Hypertension evoked by weight lifting on vascular endothelial function.
- The Coronary microcirculation in cyanotic congenital heart disease.
- Hypoxia, Coronary dilation, and the pentose phosphate pathway.
- Epoxyeicosatrienoic and dihydroxyeicosatrienoic acids dilate human Coronary arterioles via BK(Ca) channels: implications for soluble epoxide hydrolase inhibition.
- Radiation induces endothelial dysfunction in murine intestinal arterioles via enhanced production of reactive oxygen species.
- Peroxynitrite reduces the endothelium-derived hyperpolarizing factor component of Coronary flow-mediated dilation in PECAM-1-knockout mice.
- American College of Chest Physicians evidence-based guidelines--the next generation: considering resource use and evolution to a single grading system.
- Novel mechanism of vasodilation in inflammatory bowel Disease.
- Mitochondria and reactive oxygen species: an evolution in function.
- Mechanism of vasodilation to adenosine in Coronary arterioles from patients with heart disease.
- Redox modulation of vascular tone: focus of potassium channel mechanisms of dilation.
- Role of hydrogen peroxide in ACh-induced dilation of human submucosal intestinal microvessels.
- Nitration and functional loss of voltage-gated K+ channels in rat Coronary microvessels exposed to high glucose.
- Coronary vasospasm and the regulation of Coronary blood flow.
- Mechanism of dilation to reactive oxygen species in human Coronary arterioles.
- Mitochondrial sources of H2O2 generation play a key role in flow-mediated dilation in human Coronary resistance arteries.
- Enhancement of antral contractions and vagal afferent signaling with synchronized electrical stimulation.
- Elevated glucose impairs cAMP-mediated dilation by reducing Kv channel activity in rat small Coronary smooth muscle cells.
- Acquired microvascular dysfunction in inflammatory bowel Disease: Loss of nitric oxide-mediated vasodilation.
- L-4F, an apolipoprotein A-1 mimetic, dramatically improves vasodilation in hypercholesterolemia and sickle cell Disease.
- Mechanism of thrombin-induced vasodilation in human Coronary arterioles.
- Is there an association between athletic amenorrhea and endothelial cell dysfunction?
- Diabetes mellitus impairs vasodilation to hypoxia in human Coronary arterioles: reduced activity of ATP-sensitive potassium channels.
- Role for hydrogen peroxide in flow-induced dilation of human Coronary arterioles.
- The association of hypertension and secondary cardiovascular Disease with sleep-disordered breathing.
- Peroxynitrite inhibits Ca2+-activated K+ channel activity in smooth muscle of human Coronary arterioles.
- Effect of gender on endothelium-dependent dilation to bradykinin in human adipose microvessels.
- Vascular dysfunction in hyperglycemia: is protein kinase C the culprit?
- The Coronary circulation in diabetes: influence of reactive oxygen species on K+ channel-mediated vasodilation.
- Arachidonic Acid-Induced Dilation in Human Coronary Arterioles: Convergence of Signaling Mechanisms on Endothelial TRPV4-Mediated Ca2+ Entry.
Schools
University Of North Carolina Chapel Hill School Of Medicine
University Iowa Hosps and Clinics
Procedures Preformed
- Cardiac Imaging
Conditions Treated
- Angina and Acute Coronary Syndrome
- Aortic Aneurysm
- Aortic Valve Disease
- Arrhythmias (incl. Atrial Fibrillation)
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Doctors Specialties
- Cardiology
Accepted Insurances
Awards
- Fellow (FACC)
- Senior Associate Dean for Research
- President
Education
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University of Iowa Hospitals and Clinics -
University of North Carolina at Chapel Hill School of Medicine
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1609827633 |
| Last Name Of The Provider | GUTTERMAN |
| First Name Of The Provider | DAVID |
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