Mass General Hospital Division of Hematology
Hematology
Dr. David B. Sykes, MD
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Dr. David B. Sykes works in the field of Hematology. He studied medicine at University of California San Diego School of Medicine. University of California San Diego School of Medicine has a rank of 17 in research and a 19 in primary care David B. Sykes is also a published doctor. He has 18 paper published. The most recent paper is: Long-term complete clinical and hematological responses of the TEMPI syndrome after autologous stem cell transplantation. He accepts Medicare payments and is registered with Medicare.gov.
Publications
- Long-term complete clinical and hematological responses of the TEMPI Syndrome after autologous stem cell transplantation.
- Control of signaling-mediated clearance of apoptotic Cells by the tumor suppressor p53.
- Niche-Based Screening in Multiple Myeloma Identifies a Kinesin-5 Inhibitor with Improved Selectivity over Hematopoietic Progenitors.
- Hormones and the Bone Marrow: Panhypopituitarism and Pancytopenia in a Man with a Pituitary Adenoma.
- Hyperhemolysis Syndrome in a patient without a hemoglobinopathy, unresponsive to treatment with eculizumab.
- Neuregulin1-β Decreases IL-1β-Induced Neutrophil Adhesion to Human Brain Microvascular Endothelial Cells.
- Dectin-1 activation controls maturation of ?-1,3-glucan-containing phagosomes.
- SIRT1 regulates differentiation of mesenchymal stem Cells by deacetylating ?-catenin.
- The hematopoietic stem Cell niche.
- The TEMPI Syndrome--a novel multisystem disease.
- Quantitative expansion of ES cell-derived Myeloid progenitors capable of differentiating into macrophages.
- Quantitative production of macrophages or neutrophils ex vivo using conditional Hoxb8.
- E2a/Pbx1 induces the rapid proliferation of stem cell factor-dependent murine pro-T cells that cause Acute T-lymphoid or myeloid leukemias in mice.
- TH2 cytokines and allergic challenge induce Ym1 expression in macrophages by a STAT6-dependent mechanism.
- An induced Ets repressor complex regulates growth arrest during terminal macrophage differentiation.
- Case report: a 37-year-old male with telangiectasias, Polycythemia vera, perinephric fluid collections, and intrapulmonary shunting.
- Putative RNA-splicing gene LUC7L2 on 7q34 represents a candidate gene in pathogenesis of Myeloid malignancies.
Schools
University of California At San Diego
Massachusetts General Hospital
Dana Farber Cancer Institute
Conditions Treated
- Acute Leukemia
- Anemia
- Antiphospholipid Syndrome (APS)
- Chronic Myeloid Leukemia (CML)
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Doctors Specialties
- Hematology
Accepted Insurances
Education
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Brigham and Women's Hospital -
Massachusetts General Hospital -
University of California San Diego School of Medicine
Hospital
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Massachusetts General Hospital
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