Associate Professor of Medicine, University Iowa College of Medicine
Gastroenterology
Dr. David A. Elliott, MD
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David A. Elliott, MD works in the field of Gastroenterology. He went to Wayne State University School of Medicine. Wayne State University School of Medicine has a rank of 63 in research and a 85 in primary care David Elliott is also published. He has 32 publications published. The lastest publication: "Intestinal Helminths Regulate Lethal Acute Graft-versus-Host Disease and Preserve the Graft-versus-Tumor Effect in Mice.'
Publications
- Intestinal Helminths Regulate Lethal Acute Graft-versus-Host Disease and Preserve the Graft-versus-Tumor Effect in Mice.
- Helminth Infections Decrease Host Susceptibility to Immune-Mediated Diseases.
- Innate Immunity in Disease.
- Randomised clinical trial: the safety and tolerability of Trichuris suis ova in patients with Crohn's disease.
- Translatability of helminth therapy in inflammatory bowel diseases.
- Nematode Asparaginyl-tRNA Synthetase Resolves Intestinal Inflammation in Mice with T-Cell Transfer Colitis.
- Where are we on worms?
- Helminth-host immunological interactions: prevention and control of immune-mediated diseases.
- Trichuris suis might be effective in treating allergic rhinitis.
- Advances in the pathogenesis and treatment of IBD.
- Helminths and the IBD hygiene hypothesis.
- Helminthic therapy: using worms to treat immune-mediated disease.
- Colonization with Heligmosomoides polygyrus suppresses mucosal IL-17 production.
- Heligmosomoides polygyrus promotes regulatory T-cell cytokine production in the murine normal distal intestine.
- Immunologic and molecular mechanisms in inflammatory bowel disease.
- Therapeutic colonization with Trichuris suis.
- Induction of CD8+ regulatory T cells in the intestine by Heligmosomoides polygyrus infection.
- Intestinal helminths protect in a murine model of asthma.
- Heligmosomoides polygyrus induces TLR4 on murine mucosal T cells that produce TGFbeta after lipopolysaccharide stimulation.
- Why Trichuris suis should prove safe for use in inflammatory bowel diseases.
- Trichuris suis therapy for active ulcerative colitis: a randomized controlled trial.
- IL-12 induction of mRNA encoding substance P in murine macrophages from the spleen and sites of inflammation.
- Helminths and the modulation of mucosal inflammation.
- CD4+ T cells from IL-10-deficient mice transfer susceptibility to NSAID-induced Rag colitis.
- Cutting edge: hemokinin has substance P-like function and expression in inflammation.
- Trichuris suis seems to be safe and possibly effective in the treatment of inflammatory bowel disease.
- Exposure to schistosome eggs protects mice from TNBS-induced colitis.
- T cell substance P receptor governs antigen-elicited IFN-gamma production.
- Interleukin-4 receptor alpha chain and STAT6 signaling inhibit gamma interferon but not Th2 cytokine expression within schistosome granulomas.
- IL-4 inhibits vasoactive intestinal peptide production by macrophages.
- The possible link between de-worming and the emergence of immunological disease.
Schools
Wayne State University
Doctors Specialties
- Radiation Oncology
- Gastroenterology
Accepted Insurances
Education
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University of Iowa Hospitals and Clinics -
Johns Hopkins University -
Wayne State University School of Medicine
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