Assistant Professor, Pathology, University of Texas Southwestern Medical Center, Southwestern Medical School
Pathology
Dr. Daniel R. Nussenzveig, MD
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Daniel Nussenzveig is an expert in the field of Pathology. He attended NYU School of Medicine. Dr. Daniel R. Nussenzveig has written publications. He has 32 publications published with PubMed. The lastest paper is: 'A hydrophobic cluster between transmembrane helices 5 and 6 constrains the thyrotropin-releasing hormone receptor in an inactive conformation.'
Publications
- NFAR-1 and -2 modulate translation and are required for efficient host defense.
- Influenza virus targets the mRNA export machinery and the nuclear pore complex.
- The nucleoporin Nup96 is required for proper expression of interferon-regulated proteins and functions.
- Viral interactions with the nuclear transport machinery: discovering and disrupting pathways.
- Vesicular stomatitis virus inhibits mitotic progression and triggers cell death.
- Cellular mechanisms of the clearance function of type C receptors of atrial natriuretic factor.
- Atrial natriuretic factor receptors in cultured renomedullary interstitial cells.
- DuP 753 is a potent nonpeptide antagonist of angiotensin II receptors in isolated perfused rat kidney and cultured renal cells.
- Dynamics of atrial natriuretic factor-guanylate cyclase receptors and receptor-ligand complexes in cultured glomerular mesangial and renomedullary interstitial cells.
- Thyrotropin-releasing hormone binding to the mouse pituitary receptor does not involve ionic interactions. A model for neutral peptide binding to G protein-coupled rec...
- Glycosylation is important for binding to human calcitonin receptors.
- A hydrophobic cluster between transmembrane helices 5 and 6 constrains the thyrotropin-releasing hormone receptor in an inactive conformation.
- Human calcitonin receptor is directly targeted to and retained in the basolateral surface of MDCK cells.
- Human calcitonin receptors exhibit agonist-independent (constitutive) signaling activity.
- Intracellular retention and rapid degradation of human calcitonin receptors overexpressed in COS cells.
- Iodocalcitonin binds to human calcitonin receptors with higher affinity than calcitonin.
- A constitutively active mutant thyrotropin-releasing hormone receptor is Chronically down-regulated in pituitary cells: evidence using chlordiazepoxide as a negative a...
- Agonist-stimulated internalization of the thyrotropin-releasing hormone receptor is dependent on two domains in the receptor carboxyl terminus.
- Decreased levels of internalized thyrotropin-releasing hormone receptors after uncoupling from guanine nucleotide-binding protein and phospholipase-C.
- Expression of thyrotropin-releasing hormone receptors by adenovirus-mediated gene transfer reveals that thyrotropin-releasing hormone desensitization is cell specific.
- Thyrotropin-releasing hormone (TRH) receptor number determines the size of the TRH-responsive phosphoinositide pool. Demonstration using controlled expression of TRH r...
- Thyrotropin-releasing hormone receptor activation does not elevate intracellular cyclic adenosine 3',5'-monophosphate in cells expressing high levels of receptors.
- Inhibition of inositol phosphate second messenger formation by intracellular loop one of a human calcitonin receptor. Expression and mutational analysis of synthetic r...
- Truncation of the thyrotropin-releasing hormone receptor carboxyl tail causes constitutive activity and leads to impaired responsiveness in Xenopus oocytes and AtT20 c...
- Alternative splicing of a 48-nucleotide exon generates two isoforms of the human calcitonin receptor.
- A disulfide bond between conserved extracellular cysteines in the thyrotropin-releasing hormone receptor is critical for binding.
- Desensitization of the response to thyrotropin-releasing hormone in Xenopus oocytes is an amplified process that precedes calcium mobilization.
- The hemispheric functional expression of the thyrotropin-releasing-hormone receptor is not determined by the receptors'' physical distribution.
- Binding and functional effects of atrial natriuretic factor in isolated rat kidney.
- Clearance receptors of atrial natriuretic factor.
- Physiological role of silent receptors of atrial natriuretic factor.
Conditions Treated
- Anemia
- Chronic Myeloid Leukemia (CML)
- Lymphoma
- Myeloproliferative Disorders
Doctors Specialties
- Hematology
- Hematopathology
- Pathology
Accepted Insurances
Education
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Jackson Memorial Hospital - Jackson Health System -
NYU School of Medicine -
Universidad de Sao Paulo Faculty of Medicine
Hospital
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Veterans Affairs North Texas Health Care System -
University of Texas Southwestern Medical Center
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