Professor of Surgery, University of Utah School of Medicine
Thoracic Surgery
Dr. Craig H. Selzman, MD
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Dr. Craig H. Selzman is an expert in the field of Thoracic Surgery. He studied medicine at Baylor College of Medicine. He received awards:"Regional Top Doctor", "CMS Meaningful Use Stage 1 Certification" and "Fellow (FACS)". Craig H. Selzman is a published MD. He has 101 papers published. The most current publication "Estrogen replacement inhibits intimal hyperplasia and the accumulation and effects of transforming growth factor beta1." He is registered with Medicare.
Publications
- Minimally Invasive Temporary Right Ventricular Assist Device for Acute Right Ventricular Failure.
- Risk Assessment and Comparative Effectiveness of Left Ventricular Assist Device and Medical Management in Ambulatory Heart Failure Patients: Results From the ROADMAP S...
- Further Peripheral Vascular Dysfunction in Heart Failure Patients With a Continuous-Flow Left Ventricular Assist Device: The Role of Pulsatility.
- Gold Standard in Anticoagulation Assessment of Left Ventricular Assist Device Patients?: How About Bronze.
- Cardiomyocyte p65 NF-κB Is Necessary for Compensatory Adaptation to Pressure-Overload.
- Animal model of reversible, right ventricular Failure.
- Bridge to Removal: A Paradigm Shift for Left Ventricular Assist Device Therapy.
- Myocardial atrophy and chronic mechanical unloading of the failing human Heart: implications for cardiac assist device-induced myocardial recovery.
- Characterization of diffuse fibrosis in the failing human Heart via diffusion tensor imaging and quantitative histological validation.
- Baseline red blood cell osmotic fragility does not predict the degree of post-LVAD hemolysis.
- Pharmacologic therapies for Acute cardiogenic shock.
- Ventricular Assist Devices or Inotropic Agents in Status 1A Patients? Survival Analysis of the United Network of Organ Sharing Database.
- The STICH Trial (Surgical Treatment for Ischemic Heart Failure): Mode-of-Death Results.
- Cardiomyocyte-specific p65 NF-κB deletion protects the injured Heart by preservation of calcium handling.
- In vitro hydrodynamic analysis of pin and cone bearing designs of the jarvik 2000 adult ventricular assist device.
- Reply to Letter to the Editor: Left ventricular assist devices in chronic Heart failure: more questions than answers?
- Minimally invasive Aortic valve replacement with orthotopic liver transplantation: report of a case.
- Magnitude and Time Course of Changes Induced by Continuous-Flow Left Ventricular Assist Device Unloading in Chronic Heart Failure: Insights into Cardiac Recovery.
- Pulsatility and the Risk of Non-Surgical Bleeding in Patients Supported With the Continuous-Flow Left Ventricular Assist Device HeartMate II.
- Impact of Ventricular Assist Device Complications on Posttransplant Survival: An Analysis of the United Network of Organ Sharing Database.
- Morbidity and mortality in Heart transplant candidates supported with mechanical circulatory support: is reappraisal of the current United network for organ sharing th...
- Inhibitory kappa-B kinase-β inhibition prevents adaptive left ventricular hypertrophy.
- Recovery from decompensated Heart failure is associated with a distinct, phase-dependent gene expression profile.
- A longitudinal study of the course of asymptomatic antibody-mediated rejection in Heart transplantation.
- Improved survival in Heart transplant patients living at high altitude.
- Allograft rejection in patients supported with continuous-flow left ventricular assist devices.
- Changing outcomes in patients bridged to Heart transplantation with continuous- versus pulsatile-flow ventricular assist devices: an analysis of the registry of the In...
- Left ventricular assist device unloading effects on Myocardial structure and function: current status of the field and call for action.
- Antibody testing for cardiac antibody-mediated rejection: which panel correlates best with cardiovascular death?
- Impact of mechanical unloading on microvasculature and associated central remodeling features of the failing human Heart.
- The impact of bridge-to-transplant ventricular assist device support on survival after cardiac transplantation.
- Obesity and left ventricular assist device driveline exit site infection.
- Contributions of extravascular and intravascular cells to fibrin network formation, structure, and stability.
- Experience with the Levitronix CentriMag in the pediatric population as a bridge to decision and recovery.
- MicroRNA-208a is a regulator of cardiac hypertrophy and conduction in mice.
- Prior human leukocyte antigen-allosensitization and left ventricular assist device type affect degree of post-implantation human leukocyte antigen-allosensitization.
- Muscle ring finger 1 mediates cardiac atrophy in vivo.
- Regression of pressure-induced left ventricular hypertrophy is characterized by a distinct gene expression profile.
- Inhibitory kappa B kinase-beta is a target for specific nuclear factor kappa B-mediated delayed cardioprotection.
- Myocardin inhibits cellular proliferation by inhibiting NF-kappaB(p65)-dependent cell cycle progression.
- Targeted deletion of Dicer in the Heart leads to dilated cardiomyopathy and Heart failure.
- Proteasome inhibition promotes regression of left ventricular hypertrophy.
- IKKbeta inhibition attenuates myocardial injury and dysfunction following Acute ischemia-reperfusion injury.
- Aortoesophageal fistula after thoracic endovascular Aortic repair and transthoracic embolization.
- Characterization of a model to independently study regression of ventricular hypertrophy.
- Off-pump insertion of continuous flow left ventricular assist devices.
- Use of the Jarvik 2000 continuous flow left ventricular assist device for Acute myocardial infarction and cardiogenic shock.
- Case 115: aortic pseudoAneurysm from penetrating superior vena cava stent.
- Current status of the total artificial Heart.
- The price of progress: destination left ventricular assist device therapy for terminal Heart failure.
- Heart transplantation for progressive cardiomyopathy as a manifestation of MELAS syndrome.
- Interleukin-10 attenuates the response to vascular injury.
- Atherogenic effects of Chlamydia pneumoniae: refuting the innocent bystander hypothesis.
- Relative contribution of the TNF-alpha receptors to murine intimal hyperplasia.
- Diagnostic implications of C-reactive protein.
- ALCAPA in an adult with preserved left ventricular function.
- Monocyte chemotactic protein-1 directly induces human vascular smooth muscle proliferation.
- Lack of TNF-alpha attenuates intimal hyperplasia after mouse Carotid artery injury.
- Interleukin-11 attenuates human vascular smooth muscle cell proliferation.
- Vasopressin in the cardiac surgery intensive care unit.
- Inhibition of intimal hyperplasia using the selective estrogen receptor modulator raloxifene.
- A modified murine model for the study of reverse cardiac remodelling.
- Impact of donor left ventricular hypertrophy on survival after Heart transplant.
- Clinical applications of cardiovascular angiogenesis.
- Functional reconstitution and regulation of IL-18 activity by the IL-18R beta chain.
- The case for beta-adrenergic blockade as prophylaxis against perioperative cardiovascular morbidity and mortality.
- Current approaches to therapy for vascular injury.
- Therapeutic implications of inflammation in atherosclerotic cardiovascular Disease.
- Suppression of tumor necrosis factor alpha production by cAMP in human monocytes: dissociation with mRNA level and independent of interleukin-10.
- Thrombin generation in vascular tissue.
- Unusual causes of mesenteric ischemia.
- Alpha-adrenergic activation of Myocardial NF kappa B during hemorrhage.
- Surgical implications of vascular endothelial physiology.
- The biology of estrogen-mediated repair of cardiovascular injury.
- Chronic estrogen replacement inhibits Aortic intimal hyperplasia independent of serum lipids.
- Interleukin-10 inhibits human vascular smooth muscle proliferation.
- Genes don't count.
- Contemporary management of isolated iliac Aneurysms.
- TNF-alpha and Myocardial depression in endotoxemic rats: temporal discordance of an obligatory relationship.
- Class II cytokine receptor ligands inhibit human vascular smooth muscle proliferation.
- Increased levels of Myocardial IkappaB-alpha protein promote tolerance to endotoxin.
- Ovarian ablation alone promotes Aortic intimal hyperplasia and accumulation of fibroblast growth factor.
- Therapeutic implications of interleukin-10 in surgical Disease.
- Thrombophilia and activated protein C resistance.
- Interleukin-10 stabilizes inhibitory kappaB-alpha in human monocytes.
- Interleukin-10 inhibits postinjury tumor necrosis factor-mediated human vascular smooth muscle proliferation.
- Estrogen replacement inhibits intimal hyperplasia and the accumulation and effects of transforming growth factor beta1.
- Surgical implications of therapeutic angiogenesis.
- Utilization of endoscopic inoculation in a mouse model of intrauterine infection-induced preterm birth: role of interleukin 1beta.
- LPS-Induced NF-kappaB activation and TNF-alpha release in human monocytes are protein tyrosine kinase dependent and protein kinase C independent.
- Liposomal delivery of purified inhibitory-kappaBalpha inhibits tumor necrosis factor-alpha-induced human vascular smooth muscle proliferation.
- The NFkappaB inhibitory peptide, IkappaBalpha, prevents human vascular smooth muscle proliferation.
- Inhibition of PARS attenuates endotoxin-induced dysfunction of Pulmonary vasorelaxation.
- Exogenous calcium preconditions myocardium from patients taking oral sulfonylurea agents.
- Interleukin-11 attenuates Pulmonary inflammation and vasomotor dysfunction in endotoxin-induced lung injury.
- Interleukin-1beta deficiency results in reduced NF-kappaB levels in pregnant mice.
- Acellular components of Chlamydia pneumoniae stimulate cytokine production in human blood mononuclear cells.
- Chlamydia pneumoniae activates nuclear factor kappaB and activator protein 1 in human vascular smooth muscle and induces cellular proliferation.
- Differential inducible nitric oxide synthase expression in systemic and Pulmonary vessels after endotoxin.
- ISHLT pathology antibody mediated rejection score correlates with increased risk of cardiovascular mortality: A retrospective validation analysis.
Schools
Baylor University
University Of Colorado
University Colo Health Scis Center
Procedures Preformed
- Aortic Repair, Open or Repair of Arterial Aneurysm, Open
- Cardiac Catheterization (incl. Coronary Angiography)
- Cardiac MRI (Magnetic Resonance Imaging) of Heart or Chest
- Cardiac Myocardial Perfusion Imaging
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Conditions Treated
- Aneurysm and Dissection of Heart
- Angina and Acute Coronary Syndrome
- Aortic Aneurysm
- Aortic Dissection
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Doctors Specialties
- Cardiac Surgery
- Thoracic Surgery
Accepted Insurances
Awards
- Regional Top Doctor
- CMS Meaningful Use Stage 1 Certification
- Fellow (FACS)
Education
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University of Colorado -
Baylor College of Medicine
Hospital
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University of Utah Health Care - Hospital and Clinics
Drug Facts
| NPI NUMBER | 1902991144 | |
| NPPES Provider LastName | SELZMAN | |
| NPPES Provider FirstName | CRAIG | |
| NPPES Provider ZIPCode | 841320100 | |
| NPPES Provider State | UT | |
| Specialty Description | General Surgery | |
| Total Claim Count | 452.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1902991144 |
| Last Name Of The Provider | SELZMAN |
| First Name Of The Provider | CRAIG |
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