Professor, Neurology, UCLA School of Medicine
Neurology
Dr. Claude G. Wasterlain, MD
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Claude G. Wasterlain works in the field of Neurology. He has 4 awards "Super Doctor", "AES-Milken Family Foundation Award for Basic Research", "Ambassador for Epilepsy" and "Pierre Gloor Award". Claude Wasterlain is a published doctor as well. He has 26 publications published. The latest was: Widespread neuronal injury in a model of cholinergic status epilepticus in postnatal day 7 rat pups.
Publications
- Widespread neuronal injury in a model of cholinergic status epilepticus in postnatal day 7 rat pups.
- Trafficking of NMDA receptors during status epilepticus: Therapeutic implications.
- Rapid surface accumulation of NMDA receptors increases glutamatergic excitation during status epilepticus.
- Rational polytherapy in the treatment of acute seizures and status epilepticus.
- Brain energy metabolism during experimental neonatal seizures.
- GalR2-positive allosteric modulator exhibits anticonvulsant effects in animal models.
- Distinct caspase pathways mediate necrosis and apoptosis in subpopulations of hippocampal neurons after status epilepticus.
- Vulnerability of postnatal hippocampal neurons to seizures varies regionally with their maturational stage.
- Molecular basis of self-sustaining seizures and pharmacoresistance during status epilepticus: The receptor trafficking hypothesis revisited.
- Contribution of a mitochondrial pathway to excitotoxic neuronal necrosis.
- Posttraumatic Epilepsy and treatment.
- Mechanistic and pharmacologic aspects of status epilepticus and its treatment with new antiepileptic drugs.
- Status epilepticus triggers caspase-3 activation and necrosis in the immature rat brain.
- Do single seizures cause neuronal death in the human hippocampus?
- Invulnerability of the immature brain to seizures: do dogmas have nine lives?
- Treatment of experimental status epilepticus in immature rats: dissociation between anticonvulsant and antiepileptogenic effects.
- Trafficking of GABA(A) receptors, loss of inhibition, and a mechanism for pharmacoresistance in status epilepticus.
- Programmed neuronal necrosis and status epilepticus.
- GABA synapses and the rapid loss of inhibition to dentate gyrus granule cells after brief perforant-path stimulation.
- Bim, Bad, and Bax: a deadly combination in epileptic seizures.
- Development of temporal lobe Epilepsy in 21-day-old rats.
- Hypoxic neuronal necrosis: protein synthesis-independent activation of a cell death program.
- Short-term plasticity of hippocampal neuropeptides and neuronal circuitry in experimental status epilepticus.
- Epileptogenesis after self-sustaining status epilepticus.
- Epileptogenesis in the developing brain.
Schools
University of Liege Faculty of Medicine
Middlesex Meml Hospital
New York Presbyterian HospitalWeill Cornell Med Center
University Brussels
Conditions Treated
- Convulsions
- Epilepsy
- Migraine
Doctors Specialties
- Neurology
Accepted Insurances
Awards
- Super Doctor
- AES-Milken Family Foundation Award for Basic Research
- Ambassador for Epilepsy
- Pierre Gloor Award
Education
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New York Presbyterian Hospital - Cornell Campus -
Universite de L'Etat a Liege Faculty of Medicine
Hospital
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Ronald Reagan UCLA Medical Center
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