Professor, Pediatrics, University of Colorado School of Medicine
Pediatric Hematology & Oncology
Dr. Christopher C. Silliman, MD
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Doctor Christopher C. Silliman, MD is an expert in the field of Pediatric Hematology & Oncology. He attended Tulane University School of Medicine. Christopher Silliman is also a published MD. He has 33 publication published. The most recent publication is: Plasma First Resuscitation Reduces Lactate Acidosis, Enhances Redox Homeostasis, Amino Acid and Purine Catabolism in a Rat Model of Profound Hemorrhagic Shock.
Publications
- Plasma First Resuscitation Reduces Lactate Acidosis, Enhances Redox Homeostasis, Amino Acid and Purine Catabolism in a Rat Model of Profound Hemorrhagic Shock.
- Viscoelastic Measurements of Platelet Function not Fibrinogen Function Predicts Sensitivity to Tissue Plasminogen Activator in Trauma Patients.
- COMBAT: Initial experience with a randomized clinical trial of plasma-based resuscitation in the field for traumatic hemorrhagic shock.
- Hemolysis exacerbates hyperfibrinolysis, whereas platelolysis shuts down fibrinolysis: evolving concepts of the spectrum of fibrinolysis in response to severe injury.
- Routine storage of red blood Cell (RBC) units in additive solution-3: a comprehensive investigation of the RBC metabolome.
- Hyperfibrinolysis, physiologic fibrinolysis, and fibrinolysis shutdown: The spectrum of postinjury fibrinolysis and relevance to antifibrinolytic therapy.
- Metabolomics of AS-5 RBC supernatants following routine storage.
- Proteomics of apheresis platelet supernatants during routine storage: Gender-related differences.
- Dynamic Changes in Rat Mesenteric Lymph Proteins Following Trauma Using Label-Free Mass Spectrometry.
- Lymph is not a Plasma Ultrafiltrate: A Proteomic Analysis of Injured Patients.
- Clathrin complexes with the inhibitor kappa B kinase signalosome: imaging the interactome.
- Plasma first in the field for postinjury hemorrhagic shock.
- Trauma-Induced Coagulopathy: An Institution's 35 year Perspective on Practice and Research.
- Experimental pre-storage filtration removes antibodies and decreases lipid bioactivity accumulation in RBC supernatants mitigating TRALI in an animal model.
- Postinjury hyperfibrinogenemia compromises efficacy of heparin-based venous thromboembolism prophylaxis.
- Antibodies to the HLA-A2 antigen prime neutrophils and serve as the second event in an in vitro model of transfusion-related Acute lung injury.
- Platelet: RBC Ratios: Randomized Controlled Trials Are Needed for Optimal Resuscitation of Injured Patients*.
- Fibrinolysis greater than 3% is the critical value for initiation of antifibrinolytic therapy.
- Proteomic analysis of the supernatant of red blood Cell units: the effects of storage and leucoreduction.
- The incidence and magnitude of fibrinolytic activation in trauma patients: a rebuttal.
- It's not your grandfather's field plasma.
- Platelets are dominant contributors to hypercoagulability after injury.
- Antiplatelet therapy is associated with decreased transfusion-associated risk of lung dysfunction, multiple organ failure, and mortality in trauma patients.
- Treatment of refractory hemorrhage with factor XIII in a patient with Hemophilia A with inhibitor.
- Hyperosmolarity Attenuates TNF?-Mediated Pro-Inflammatory Activation of Human Pulmonary Microvascular Endothelial Cells.
- Hemoglobin-Based Oxygen Carrier Mitigates Transfusion-Mediated Pancreas Cancer Progression.
- Functional fibrinogen assay indicates that fibrinogen is critical in correcting abnormal clot strength following trauma.
- Plasma from stored packed red blood cells and MHC class I antibodies causes Acute lung injury in a 2-event in vivo rat model.
- Stored red blood Cell transfusion induces regulatory T Cells.
- Platelet Vascular Endothelial Growth Factor is a Potential Mediator of Transfusion-Related Acute Lung Injury.
- Hyperosmolarity invokes distinct anti-inflammatory mechanisms in pulmonary epithelial Cells: evidence from signaling and transcription layers.
- Hb Grand Junction (HBB: c.348_349delinsG; p.His117IlefsX42): a new hyperunstable hemoglobin variant.
Schools
Tulane University School Of Medicine
University Of Virginia Program
University Of Colorado Children S Hospital Colorado Program
Conditions Treated
- Acute Leukemia
- Adrenal Gland Cancer
- Anemia
- Coagulation Disorders (incl. Hemophilia)
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Doctors Specialties
- Pediatric Hematology
- Oncology
Accepted Insurances
- Choice Plus POS II
- Whole Health Colorado Front Range
- Pathway X Bronze Direct CACA IN HIX
- Anthem Blue Cross Blue Shield PPO
- Blue Card PPO
- Cigna HMO
- Cigna PPO
- Great West Healthcare-Cigna PPO
- LocalPlus
- Open Access Plus
- Cofinity PPO
- Commercial Insurance Company
- Golden Rule
- Kaiser Permanente
- Medicaid
- Medicare MCR
- MultiPlan PPO
- PHCS Network PPO
- Choice Plus POS
- Navigate HMO
- Navigate POS
- Options PPO
- United Healthcare
Education
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University of Colorado -
University of Virginia -
Tulane University School of Medicine
Hospital
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Children's Hospital Colorado
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