Associate Professor, Internal Medicine, Washington University School of Medicine
Rheumatology
Dr. Christine T. Pham, MD
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Dr. Christine T. Pham practices Rheumatology. She went to University of Florida College of Medicine. University of Florida College of Medicine is ranked 43/78 in Research/PrimaryCare. She received 1 awards: "Fellow (FACR)". Christine T. Pham, MD is a published doctor. She published 49 publications, including: 'The 5' flanking region of the human granzyme H gene directs expression to T/natural killer cell progenitors and lymphokine-activated killer cells in transgenic mice.' Dr. Christine T. Pham accepts Medicare payments and is listed with Medicare.gov.
Publications
- Treatment With Dimethyl Fumarate Attenuates Calcineurin Inhibitor-Induced Nephrotoxicity.
- Odour receptors and neurons for DEET and new insect repellents.
- Neutrophil-derived cathelicidin protects from neointimal hyperplasia.
- Inflammatory Arthritis in caspase 1 gene-deficient mice: contribution of proteinase 3 to caspase 1-independent production of bioactive interleukin-1beta.
- Neutrophil secretion products pave the way for inflammatory monocytes.
- Salutary effect of pre-treatment with an Nrf2 inducer on ischemia reperfusion injury in the rat liver.
- Inhibition of pepsin by analogues of pepsinogen-(1-12)-peptide with substitutions in the 4-7 sequence region.
- Assessing tumor angiogenesis using macromolecular MR imaging contrast media.
- Plasma levels of 8-methoxypsoralen after topical Paint PUVA.
- Psychodermatology. Practical guidelines on pharmacotherapy.
- DPPI may activate KLK4 during enamel formation.
- Inhibition of the complement membrane attack complex by the galactose-specific adhesion of Entamoeba histolytica.
- Functional analysis of the human complement receptor 2 (CR2/CD21) promoter: characterization of basal transcriptional mechanisms.
- An intronic silencer regulates B lymphocyte cell- and stage-specific expression of the human complement receptor type 2 (CR2, CD21) gene.
- Requirement for the SLP-76 adaptor GADS in T cell development.
- How do cytotoxic lymphocytes kill their targets?
- Characterization of hematopoietic progenitor mobilization in protease-deficient mice.
- Transgenic analysis of a 100-kb human beta-globin cluster-containing DNA fragment propagated as a bacterial artificial chromosome.
- Production of fully active recombinant murine granzyme B in yeast.
- The role of granzyme B cluster proteases in cell-mediated cytotoxicity.
- Long-range disruption of gene expression by a selectable marker cassette.
- Molecular cloning, chromosomal localization, and expression of murine dipeptidyl peptidase I.
- The 5' flanking region of the human granzyme H gene directs expression to T/natural killer cell progenitors and lymphokine-activated killer cells in transgenic mice.
- Dipeptidyl peptidase I is essential for activation of mast cell chymases, but not tryptases, in mice.
- Normal neutrophil function in cathepsin G-deficient mice.
- Dipeptidyl peptidase I is required for the processing and activation of granzymes A and B in vivo.
- Dipeptidyl peptidase I-dependent neutrophil recruitment modulates the inflammatory response to Sendai virus infection.
- Neutrophil serine proteases fine-tune the inflammatory response.
- Critical role of dipeptidyl peptidase I in neutrophil recruitment during the development of experimental abdominal aortic aneurysms.
- Pathophysiology of abdominal aortic aneurysms: insights from the elastase-induced model in mice with different genetic Backgrounds.
- Neutrophil serine proteases: specific regulators of inflammation.
- Neutrophil-derived serine proteases in immune complex-mediated diseases.
- Dipeptidyl peptidase I regulates the development of collagen-induced Arthritis.
- Serine protease cathepsin G regulates adhesion-dependent neutrophil effector functions by modulating integrin clustering.
- Papillon-Lefèvre Syndrome: correlating the molecular, cellular, and clinical consequences of cathepsin C/dipeptidyl peptidase I deficiency in humans.
- Regulation of the symmetry and intensity of immune complex-mediated synovitis by nuclear factor of activated T cells.
- Characterization of murine cathepsin W and its role in cell-mediated cytotoxicity.
- Mast cell dipeptidyl peptidase I mediates survival from sepsis.
- Dipeptidyl peptidase I activates neutrophil-derived serine proteases and regulates the development of acute experimental Arthritis.
- Alphavbeta3-targeted nanotherapy suppresses inflammatory Arthritis in mice.
- Complement-dependent neutrophil recruitment is critical for the development of elastase-induced abdominal aortic aneurysm.
- Dipeptidyl-peptidase I does not functionally compensate for the loss of tripeptidyl-peptidase I in the neurodegenerative disease late-infantile neuronal ceroid lipofus...
- Variable antibody-dependent activation of complement by functionalized phospholipid nanoparticle surfaces.
- Synergistic effect of antiangiogenic nanotherapy combined with methotrexate in the treatment of experimental inflammatory Arthritis.
- Simultaneous dual frequency 1H and 19F open coil imaging of arthritic rabbit Knee at 3T.
- Antibody directs properdin-dependent activation of the complement alternative pathway in a mouse model of abdominal aortic aneurysm.
- Neutrophil Serine Proteases Promote IL-1? Generation and Injury in Necrotizing Crescentic Glomerulonephritis.
- Role of NADPH oxidase versus neutrophil proteases in antimicrobial host defense.
Schools
University Of Florida
Barnes Hospital
Wash University
Conditions Treated
- Background: yellow;'>Arthritis
- Chronic Neck Background: yellow;'>Pain
- Cryoglobulinemia
- Fibromyalgia
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Doctors Specialties
- Rheumatology
Accepted Insurances
Awards
- Fellow (FACR)
Education
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Washington University - Barnes-Jewish Hospital -
University of Florida College of Medicine
Hospital
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Barnes-Jewish Hospital
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1396853511 |
| Last Name Of The Provider | PHAM |
| First Name Of The Provider | CHRISTINE |
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