Associate Professor of Radiology, Massachusetts General Hospital
Neurology
Dr. Cenk Ayata, MD
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Cenk Ayata works in the field of Neurology. Cenk Ayata is a published physician. He has 72 publications published. The most current publication "Supply-Demand Mismatch Transients in Susceptible Peri-infarct Hot Zones Explain the Origins of Spreading Injury Depolarizations." He is registered with Medicare and accepts Medicare payments.
Publications
- Supply-Demand Mismatch Transients in Susceptible Peri-infarct Hot Zones Explain the Origins of Spreading Injury Depolarizations.
- Sustained functional improvement by hepatocyte growth factor-like small molecule BB3 after focal Cerebral ischemia in rats and mice.
- Lasting pure-motor deficits after focal posterior internal capsule white-matter infarcts in rats.
- Migraine mutations impair hippocampal learning despite enhanced long-term potentiation.
- Aag-initiated base excision repair promotes ischemia reperfusion injury in liver, brain, and kidney.
- Migraine Prophylaxis, Ischemic Depolarizations, and Stroke Outcomes in Mice.
- Endothelial dysfunction abrogates the efficacy of normobaric hyperoxia in Stroke.
- Migraine and Stroke: In search of shared mechanisms.
- Fingolimod exerts neuroprotective effects in a mouse model of intraCerebral hemorrhage.
- Rho-kinase inhibition improves ischemic perfusion deficit in hyperlipidemic mice.
- Selective ROCK2 Inhibition In Focal Cerebral Ischemia.
- Pearls and pitfalls in experimental models of spreading depression.
- Calabadion: A New Agent to Reverse the Effects of Benzylisoquinoline and Steroidal Neuromuscular-blocking Agents.
- Hyperlipidemia disrupts cerebrovascular reflexes and worsens ischemic perfusion defect.
- Glucose modulation of spreading depression susceptibility.
- Quantitative imaging of Cerebral blood flow velocity and intracellular motility using dynamic light scattering-optical coherence tomography.
- CONCUSSIVE INJURY BEFORE OR AFTER CONTROLLED CORTICAL IMPACT EXACERBATES HISTOPATHOLOGY AND FUNCTIONAL OUTCOME IN A MIXED TRAUMATIC BRAIN INJURY MODEL IN MICE.
- Increased glucose availability does not restore prolonged spreading depression durations in hypotensive rats without brain injury.
- Rho-kinase inhibition acutely augments blood flow in focal Cerebral ischemia via endothelial mechanisms.
- Normobaric hyperoxia improves Cerebral blood flow and oxygenation, and inhibits peri-infarct depolarizations in experimental focal ischaemia.
- The phosphorylation state of eNOS modulates vascular reactivity and outcome of Cerebral ischemia in vivo.
- Age-dependent cerebrovascular dysfunction in a transgenic mouse model of Cerebral amyloid angiopathy.
- A computerized algorithm for etiologic classification of ischemic Stroke: the Causative Classification of Stroke System.
- Mild induced hypertension improves blood flow and oxygen metabolism in transient focal Cerebral ischemia.
- Linking Notch signaling to ischemic Stroke.
- Hypoxia and hypotension transform the blood flow response to cortical spreading depression from hyperemia into hypoperfusion in the rat.
- The impact of anesthetics and hyperoxia on cortical spreading depression.
- Large arteriolar component of oxygen delivery implies a safe margin of oxygen supply to Cerebral tissue.
- The sirtuin-2 inhibitor AK7 is neuroprotective in models of Parkinson's disease but not amyotrophic lateral sclerosis and Cerebral ischemia.
- Subarachnoid hemorrhage, spreading depolarizations and impaired neurovascular coupling.
- Multiparametric, longitudinal optical coherence tomography imaging reveals acute injury and chronic recovery in experimental ischemic Stroke.
- BDNF and its receptors in human myasthenic thymus: implications for cell fate in thymic pathology.
- Purinergic receptor type 6 contributes to airway inflammation and remodeling in experimental allergic airway inflammation.
- PNAS Plus: Hypomorphic Notch 3 alleles link Notch signaling to ischemic Cerebral small-vessel disease.
- Cortical spreading depression impairs oxygen delivery and metabolism in mice.
- Chronic daily cortical spreading depressions suppress spreading depression susceptibility.
- Two-photon microscopy of cortical NADH fluorescence intensity changes: correcting contamination from the hemodynamic response.
- Cerebrovascular lesions induce transient β-amyloid deposition.
- Genetic animal models of Cerebral vasculopathies.
- Migraine Mutations Increase Stroke Vulnerability by Facilitating Ischemic Depolarizations.
- Pharmacological targeting of spreading depression in migraine.
- A randomized, double-blind, placebo-controlled pilot study of simvastatin in aneurysmal subarachnoid hemorrhage.
- Targeting cerebrovascular Rho-kinase in Stroke.
- Simultaneous multispectral reflectance imaging and laser speckle flowmetry of Cerebral blood flow and oxygen metabolism in focal Cerebral ischemia.
- Genetic and hormonal factors modulate spreading depression and transient hemiparesis in mouse models of familial hemiplegic migraine type 1.
- Simultaneous imaging of Cerebral partial pressure of oxygen and blood flow during functional activation and cortical spreading depression.
- Androgenic suppression of spreading depression in familial hemiplegic migraine type 1 mutant mice.
- Perfusion pressure-dependent recovery of cortical spreading depression is independent of tissue oxygenation over a wide physiologic range.
- Nutrient-sensitized screening for drugs that shift energy metabolism from mitochondrial respiration to glycolysis.
- Microemboli may link spreading depression, migraine aura, and patent foramen ovale.
- Cortical spreading depression and migraine.
- Cortical spreading depression triggers migraine attack: pro.
- Gabapentin suppresses cortical spreading depression susceptibility.
- Soluble guanylate cyclase alpha1beta1 limits Stroke size and attenuates neurological injury.
- CADASIL: experimental insights from animal models.
- Oxcarbazepine does not suppress cortical spreading depression.
- Optical coherence tomography for the quantitative study of cerebrovascular physiology.
- Cerebral autosomal dominant arteriopathy with subcortical infarcts and leukoencephalopathy syndrome mutations increase susceptibility to spreading depression.
- Enhanced subcortical spreading depression in familial hemiplegic migraine type 1 mutant mice.
- Gabapentin reduces infarct volume but does not suppress peri-infarct depolarizations.
- Ischaemic brain oedema.
- Timing of neurologic deterioration in massive middle Cerebral artery infarction: a multicenter review.
- Intensive care management of specific Stroke treatment.
- Etomidate and Ketamine: Residual Motor and Adrenal Dysfunction that Persist beyond Recovery from Loss of Righting Reflex in Rats.
- Vasculitis of the spinal cord.
- Laser speckle flowmetry for the study of cerebrovascular physiology in normal and ischemic mouse cortex.
- Pronounced hypoperfusion during spreading depression in mouse cortex.
- Achieving normothermia in patients with febrile subarachnoid hemorrhage: feasibility and safety of a novel intravascular cooling catheter.
- Cortical spreading depression confounds concentration-dependent pial arteriolar dilation during N-methyl-D-aspartate superfusion.
- Vasoconstrictive neurovascular coupling during focal ischemic depolarizations.
- Suppression of cortical spreading depression in migraine prophylaxis.
Schools
Hacettepe Universitesi Tip Fakultesi
Newton Wellesley Hospital
New England Med Center
Mass General Hospital Boston
Conditions Treated
- Cerebral Artery Thrombosis
- Stroke
Doctors Specialties
- Neurology
Accepted Insurances
Education
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Tufts Medical Center
Hospital
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Massachusetts General Hospital
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1326038043 |
| Last Name Of The Provider | AYATA |
| First Name Of The Provider | CENK |
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