Associate Professor, Pulmonology, Baylor College of Medicine
Pulmonology
Dr. Burton F. Dickey, MD
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Burton Dickey is an expert in the field of Pulmonology. He researched medicine at University of Connecticut School of Medicine. University of Connecticut School of Medicine is ranked 60/49 in Research/PrimaryCare. He received awards:"Regional Top Doctor". Burton Dickey is also published. He has 100 publications published. The lastest publication: "Agonist-induced sorting of human beta2-adrenergic receptors to lysosomes during downregulation.' Doctor Burton F. Dickey, MD is registered with Medicare and accepts Medicare payments.
Publications
- Activation of Lung toll-like receptors does not exacerbate sickness responses to lipopolysaccharide in mice.
- Muc5b is required for airway defence.
- Pneumonia during Remission Induction Chemotherapy in Patients with Acute Leukemia.
- Deletion of the Gene Encoding Calcitonin and CGRP? Does Not Affect the Outcome of Severe Infection in Mice.
- TLR2/6 and TLR9 Agonists Suppress Viral Replication but not Airway Hyperreactivity in Guinea Pigs.
- ss2-adrenoceptor Agonists are Required for Development of the Asthma Phenotype in a Murine Model.
- Munc18b is an essential gene in mice whose expression is limiting for secretion by airway epithelial and mast cells.
- Biochemistry. Walking on solid ground.
- Enhancement of Lung Tumorigenesis in a Gprc5a Knockout Mouse by Chronic Extrinsic Airway Inflammation.
- Assessment of intracellular mucin content in vivo.
- Complementary anti-inflammatory effects of a ?-blocker and a corticosteroid in an Asthma model.
- Synergistic Interactions of TLR2/6 and TLR9 Induce a High Level of Resistance to Lung Infection in Mice.
- Muc5ac: a critical component mediating the rejection of enteric nematodes.
- A common MUC5B promoter polymorphism and Pulmonary fibrosis.
- Performance of a standardized bronchoalveolar lavage protocol in a comprehensive Cancer center: A Prospective 2-Year Study.
- Interleukin 6, but not T helper 2 cytokines, promotes Lung carcinogenesis.
- Airway mucus function and dysfunction.
- Helicobacter pylori bacteremia with sepsis syndrome.
- Inducible innate resistance of Lung epithelium to infection.
- Clinical implications of the intrinsic efficacy of beta-adrenoceptor drugs in Asthma: full, partial and inverse agonism.
- Stimulated innate resistance of Lung epithelium protects mice broadly against bacteria and fungi.
- Synaptotagmin-2 controls regulated exocytosis but not other secretory responses of mast cells.
- Synaptotagmin 2 couples mucin granule exocytosis to Ca2+ signaling from endoplasmic reticulum.
- Promotion of lung carcinogenesis by Chronic obstructive pulmonary disease-like airway inflammation in a K-ras-induced mouse model.
- Beta2-adrenoceptor signaling is required for the development of an Asthma phenotype in a murine model.
- Augmented Lung inflammation protects against influenza A pneumonia.
- Mucus hypersecretion in Asthma: causes and effects.
- Small airway mucous metaplasia and inflammation in Chronic obstructive pulmonary disease.
- The BH3-only protein Bik/Blk/Nbk inhibits nuclear translocation of activated ERK1/2 to mediate IFNgamma-induced cell death.
- In vivo demonstration and quantification of intracellular Bacillus anthracis in Lung epithelial cells.
- Stimulation of Lung innate immunity protects against lethal pneumococcal pneumonia in mice.
- Haemophilus influenzae lysate induces aspects of the Chronic obstructive pulmonary disease phenotype.
- Cre-mediated recombination in mouse Clara cells.
- Chronic exposure to beta-blockers attenuates inflammation and mucin content in a murine Asthma model.
- Regulated airway goblet cell mucin secretion.
- Exoskeletons and exhalation.
- Central role of Muc5ac expression in mucous metaplasia and its regulation by conserved 5' elements.
- Salmeterol stimulation dissociates beta2-adrenergic receptor phosphorylation and internalization.
- Synaptotagmin-2 is essential for survival and contributes to Ca2+ triggering of neurotransmitter release in central and neuromuscular synapses.
- A3 adenosine receptor signaling contributes to airway mucin secretion after allergen challenge.
- Recombinant interferon gamma1b immune enhancement in 20 patients with hematologic malignancies and systemic opportunistic Infections treated with donor granulocyte tra...
- Airway mucus: From production to secretion.
- Growth factor independence-1 is expressed in primary human neuroendocrine lung carcinomas and mediates the differentiation of murine Pulmonary neuroendocrine cells.
- Mucin is produced by clara cells in the proximal airways of antigen-challenged mice.
- Beta-agonist intrinsic efficacy: measurement and clinical significance.
- Direct interaction between Rab3b and the polymeric immunoglobulin receptor controls ligand-stimulated transcytosis in epithelial cells.
- Regulated Mucin Secretion from Airway Epithelial Cells.
- The polymeric mucin Muc5ac is required for allergic airway hyperreactivity.
- Inhaled innate immune ligands to prevent pneumonia.
- Nontypeable Haemophilus influenzae in Chronic obstructive pulmonary disease and lung cancer.
- beta-Adrenoceptor inverse agonists in Asthma.
- Host Lung gene expression patterns predict infectious etiology in a mouse model of pneumonia.
- Munc13-2-/- baseline secretion defect reveals source of oligomeric mucins in mouse airways.
- Inhaled therapeutics for prevention and treatment of pneumonia.
- Allergic Lung inflammation alters neither susceptibility to Streptococcus pneumoniae infection nor inducibility of innate resistance in mice.
- Curcumin inhibits COPD-like airway inflammation and lung cancer progression in mice.
- The safety and effects of the beta-blocker, nadolol, in mild Asthma: an open-label pilot study.
- A better crystal ball to predict Lung-cancer survival?
- The role of intrinsic efficacy in determining response to a beta2-agonist in acute severe Asthma.
- Expression and transcriptional regulation of Munc18 isoforms in mast cells.
- Inflammatory gene polymorphisms influence risk of postoperative morbidity after Lung resection.
- Gene therapy of mucus hypersecretion in experimental Asthma.
- Rab3D, a small GTPase, is localized on mast cell secretory granules and translocates to the plasma membrane upon exocytosis.
- Salmeterol-induced desensitization, internalization and phosphorylation of the human beta2-adrenoceptor.
- Long-acting inhaled beta2-agonists in Asthma therapy.
- Atrial natriuretic peptide modulates alveolar type 2 cell adenylyl and guanylyl cyclases and inhibits surfactant secretion.
- Repetitive endocytosis and recycling of the beta 2-adrenergic receptor during agonist-induced steady state redistribution.
- Synaptotagmin II negatively regulates Ca2+-triggered exocytosis of lysosomes in mast cells.
- Gene structure and promoter function of murine Munc18-2, a nonneuronal exocytic Sec1 homolog.
- Ligand-stimulated beta 2-adrenergic receptor internalization via the constitutive endocytic pathway into rab5-containing endosomes.
- Inhibition of mucin secretion with MARCKS-related peptide improves airway obstruction in a mouse model of Asthma.
- Agonist-induced sorting of human beta2-adrenergic receptors to lysosomes during downregulation.
- New perspectives regarding β(2) -adrenoceptor ligands in the treatment of Asthma.
- A novel membrane-anchored Rab5 interacting protein required for homotypic endosome fusion.
- Biochemical and functional characterization of a recombinant GTPase, Rab5, and two of its mutants.
- Response to salbutamol in patients with mild Asthma treated with nadolol.
- Inhaled corticosteroids stabilize constrictive bronchiolitis after hematopoietic stem cell transplantation.
- Genomic organization, chromosomal localization, and expression of the murine RAB3D gene.
- Partial beta 2-agonists and their impartial assessment.
- Outcome of alveolar hemorrhage in hematopoietic stem cell transplant recipients.
- Functional and structural interactions of the Rab5 D136N mutant with xanthine nucleotides.
- Mechanisms of mastoparan-stimulated surfactant secretion from isolated Pulmonary alveolar type 2 cells.
- Evidence for a symmetrical requirement for Rab5-GTP in in vitro endosome-endosome fusion.
- Traffic control: Rab GTPases and the regulation of interorganellar transport.
- Stenotrophomonas maltophilia pneumonia in Cancer patients without traditional risk factors for infection, 1997-2004.
- Conformationally variable Rab protein surface regions mapped by limited proteolysis and homology modelling.
- Recurrent Asthma, sinusitis, and rash in a 63-year-old man.
- Retention of p53val135 wild-type function in transgenic mice.
- Identification of ras and ras-related low-molecular-mass GTP-binding proteins associated with rat Lung lamellar bodies.
- Low molecular weight GTP-binding proteins are associated with neuronal organelles involved in rapid axonal transport and exocytosis.
- Demonstration of C5 cleaving activity in bronchoalveolar fluids and cells: a mechanism of acute and Chronic alveolitis.
- Cost shifting in a mental health carve-out for the AFDC population.
- Cost-shifting in manged care.
- ExtraPulmonary thoracic restriction (hidebound chest) complicating eosinophilic fasciitis.
- Compartmentalization of low molecular mass GTP-binding proteins among neutrophil secretory granules.
- Day hospitalization and an inn instead of inpatient care for psychiatric patients.
- Specific and effective interaction of a guanine nucleotide analogue with small G proteins.
- Identification and purification of a novel G protein from neutrophils.
- Oleic-acid-induced Lung injury in the rat. Failure of indomethacin treatment or complement depletion to ablate Lung injury.
Schools
Univ Of Ct Sch Of Med
Temple University Hospital
Boston University School Med
Procedures Preformed
- Bronchoscopy
- Thoracentesis
Conditions Treated
- Asthma
- Bronchiectasis
- COPD (Chronic Obstructive Pulmonary Disease)
- Lung Cancer
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Doctors Specialties
- Critical Care Medicine
- Pulmonology
Accepted Insurances
Awards
- Regional Top Doctor
Education
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Boston University Medical Center -
Temple University Hospital -
University of Connecticut School of Medicine
Hospital
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University of Texas M.D. Anderson Cancer Center
Drug Facts
| NPI NUMBER | 1679672737 | |
| NPPES Provider LastName | DICKEY | |
| NPPES Provider FirstName | BURTON | |
| NPPES Provider ZIPCode | 770304009 | |
| NPPES Provider State | TX | |
| Specialty Description | Pulmonary Disease | |
| Total Claim Count | 113.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1679672737 |
| Last Name Of The Provider | DICKEY |
| First Name Of The Provider | BURTON |
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