Professor , David Geffin School of Medicine, UCLA
Psychiatry
Dr. Bruce L. Kagan, MD
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Bruce Kagan is an expert in the field of Psychiatry. He graduated from Albert Einstein College of Medicine of Yeshiva University. He received 3 awards: "Top Doctors:LA Area", "Top Doctors:Southern California" and "Super Doctor". Bruce L. Kagan, MD is a published physician. He published 58 publications, including: 'Use of planar lipid bilayer membranes for rapid screening of membrane active compounds.' He is registered with Medicare.
Publications
- Role of the Fast Kinetics of Pyroglutamate-Modified Amyloid-β Oligomers in Membrane Binding and Membrane Permeability.
- Activity and Architecture of Pyroglutamate-Modified Amyloid-β (AβpE3-42) Pores.
- Disordered amyloidogenic peptides may insert into the membrane and assemble into common cyclic structural motifs.
- Familial Alzheimer's Disease Osaka Mutant (ΔE22) β-Barrels Suggest an Explanation for the Different Aβ1-40/42 Preferred Conformational States Observed by Experiment.
- Alzheimer's disease: which type of amyloid-preventing Drug agents to employ?
- Mechanisms for the Insertion of Toxic, Fibril-like ?-Amyloid Oligomers into the Membrane.
- Effects of point substitutions on the structure of toxic Alzheimer's β-amyloid channels: atomic force microscopy and molecular dynamics simulations.
- Probing Structural Features of Alzheimer's Amyloid-? Pores in Bilayers Using Site-Specific Amino Acid Substitutions.
- Atomic Force Microscopy and MD Simulations Reveal Pore-Like Structures of All-D-Enantiomer of Alzheimer's ?-Amyloid Peptide: Relevance to the Ion Channel Mechanism of ...
- Antimicrobial Properties of Amyloid Peptides.
- Antimicrobial amyloids?
- Truncated beta-amyloid peptide channels provide an alternative mechanism for Alzheimer's Disease and Down syndrome.
- Amyloid peptide pores and the beta sheet conformation.
- Membrane activity of an amphiphilic alpha-helical membrane-proximal cytoplasmic domain of the MoMuLV envelope glycoprotein.
- Alzheimer Research Forum Live Discussion: Now you see them, now you don't: The amyloid channel hypothesis.
- Amyloid ion channels: a common structural link for protein-misfolding disease.
- Amyloidosis and protein folding.
- Electrophysiologic properties of channels induced by Abeta25-35 in planar lipid bilayers.
- The channel hypothesis of Alzheimer's disease: current status.
- Channel formation by serum amyloid A: a potential mechanism for amyloid pathogenesis and host defense.
- Induction of ion-permeable channels by the venom of the fanged bloodworm Glycera dibranchiata.
- Membrane permeabilization by thrombin-induced platelet microbicidal protein 1 is modulated by transmembrane voltage polarity and magnitude.
- Amphipathic beta structure of a leucine-rich repeat peptide.
- Oral S-adenosylmethionine in depression: a randomized, double-blind, placebo-Controlled trial.
- Mode of action of yeast killer toxins: channel formation in lipid bilayer membranes.
- Colicin K acts by forming voltage-dependent channels in phospholipid bilayer membranes.
- Elevated lipid levels in Vietnam veterans with chronic posttraumatic stress Disorder.
- Isolation and characterization of the outer membrane of Borrelia hermsii.
- Inositol 1,4,5-trisphosphate directly opens diphtheria toxin channels.
- Mode of action of colicin ib: formation of ion-permeable membrane channels.
- Orientation of the diphtheria toxin channel in lipid bilayers.
- Diphtheria toxin fragment forms large pores in phospholipid bilayer membranes.
- Defensins: a family of antimicrobial and cytotoxic peptides.
- Activation and mechanism of Clostridium septicum alpha toxin.
- The Oms66 (p66) protein is a Borrelia burgdorferi porin.
- The channel hypothesis of Huntington's disease.
- Virulent strain associated outer membrane proteins of Borrelia burgdorferi.
- Porin activity of the native and recombinant outer membrane protein Oms28 of Borrelia burgdorferi.
- Antimicrobial defensin peptides form voltage-dependent ion-permeable channels in planar lipid bilayer membranes.
- Use of planar lipid bilayer membranes for rapid screening of membrane active compounds.
- Use of lipid bilayer membranes to detect pore formation by toxins.
- Membrane perturbation by the neurotoxic Alzheimer amyloid fragment beta 25-35 requires aggregation and beta-sheet formation.
- Amyloid peptide channels.
- Formation of ion-permeable channels by tumor necrosis factor-alpha.
- Conformation and ion channel activity of lymphotoxin at neutral and low pH.
- Ion channel activity of the BH3 only Bcl-2 family member, BID.
- The role of channel formation in the mechanism of action of tumor necrosis factors.
- Channel formation in planar lipid bilayers by a neurotoxic fragment of the beta-amyloid peptide.
- Transthyretin channel formation in lipid membranes: implications for pathogenesis.
- Pore formation by the cytotoxic islet amyloid peptide amylin.
- Membrane channel formation by antimicrobial protegrins.
- Channel formation by a neurotoxic prion protein fragment.
- Polyglutamine-induced ion channels: a possible mechanism for the neurotoxicity of Huntington and other CAG repeat diseases.
- Impairment of hippocampal long-term potentiation by Alzheimer amyloid beta-peptides.
- Alzheimer amyloid abeta1-42 channels: effects of solvent, pH, and Congo Red.
- Pore formation by beta-2-microglobulin: a mechanism for the pathogenesis of dialysis associated amyloidosis.
- Amyloid peptide channels: blockade by zinc and inhibition by Congo red (amyloid channel block).
Schools
Albert Einstein College of Medicine of Yeshiva University
UCLA NpiWest La Vamc
Procedures Preformed
- Family Psychotherapy
- Group Psychotherapy
- Psychiatric Evaluation
Conditions Treated
- Adjustment Disorder
- Anxiety
- Attention Deficit Hyperactivity Disorder (ADHD)
- Autism
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Doctors Specialties
- Psychiatry
Accepted Insurances
- Aetna HMO
- Choice Plus POS II
- Blue Card PPO
- Commercial Insurance Company
- Coventry Health Care
- First Health (Coventry Health Care) PPO
- Blue & Gold HMO
- Covered California - Bronze PPO
- Employer Group HMO
- Health Net PPO
- Individual and Family PPO
- Medicare MCR
- Heritage Signature
- United Healthcare
Awards
- Top Doctors:LA Area
- Top Doctors:Southern California
- Super Doctor
Education
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UCLA Neuropsychiatric Hospital -
Albert Einstein College of Medicine of Yeshiva University
Hospital
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Ronald Reagan UCLA Medical Center
Drug Facts
| NPI NUMBER | 1336173921 | |
| NPPES Provider LastName | KAGAN | |
| NPPES Provider FirstName | BRUCE | |
| NPPES Provider ZIPCode | 900950001 | |
| NPPES Provider State | CA | |
| Specialty Description | Psychiatry | |
| Total Claim Count | 721.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1336173921 |
| Last Name Of The Provider | KAGAN |
| First Name Of The Provider | BRUCE |
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