Physician
Dermatology
Dr. Bruce J. Longley Jr., MD
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Bruce Longley works in the field of Dermatology. He researched medicine at University of Wisconsin School of Medicine and Public Health. Bruce Longley is a published doctor and has 51 publications published. The lastest was: 'Activating and dominant inactivating c-KIT catalytic domain mutations in distinct clinical forms of human mastocytosis.' He accepts Medicare payments and is registered with Medicare.gov.
Publications
- Upregulation of PI3K/AKT/mTOR, FABP5 and PPARβ/δ in Human Psoriasis and Imiquimod-induced Murine Psoriasiform Dermatitis Model.
- MAGE proteins regulate KRAB zinc finger transcription factors and KAP1 E3 ligase activity.
- Role of CRD-BP in the Growth of Human Basal Cell Carcinoma Cells.
- Expression of CD31/PECAM-1 (Platelet Endothelial Cell Adhesion Molecule 1) by Blastic Plasmacytoid Dendritic Cell Neoplasms.
- Expression of angiopoietin-TIE system components in angiosarcoma.
- Angiosarcoma Outcomes and Prognostic Factors: A 25-Year Single Institution Experience.
- Familial Urticaria Pigmentosa: Report of a Family and Review of the Role of KIT Mutations.
- Identification of novel small molecules that inhibit protein-protein interactions between MAGE and KAP-1.
- Serpiginous erythematous plaques on the feet.
- Induction of autophagy and inhibition of melanoma growth in vitro and in vivo by hyperactivation of oncogenic BRAF.
- Desmoplastic trichoepithelioma, infiltrative/morpheaform BCC, and microcystic adnexal carcinoma: differentiation by immunohistochemistry and determining the need for M...
- Mohs surgical excision of a granular cell tumor with plexiform features on the arm of a 7-year-old child.
- KIT masters mast cells in kids, too.
- Kikuchi's disease: case report and systematic review of cutaneous and histopathologic presentations.
- MAGE-A, mMage-b, and MAGE-C proteins form complexes with KAP1 and suppress p53-dependent apoptosis in MAGE-positive cell lines.
- Epigenetic control of MAGE gene expression by the KIT tyrosine kinase.
- Select cancer testes antigens of the MAGE-A, -B, and -C families are expressed in mast cell lines and promote cell viability in vitro and in vivo.
- Thrombotic lymphocytic vasculitic reaction to intravenous immunoglobulin.
- A novel biomarker for staging human prostate adenocarcinoma: overexpression of matriptase with concomitant loss of its inhibitor, hepatocyte growth factor activator in...
- Novel germline mutation of KIT associated with familial gastrointestinal stromal tumors and mastocytosis.
- Chemoprevention of skin cancer by grape constituent resveratrol: relevance to human disease?
- Generalized bullae in an infant.
- Diagnosis of gastrointestinal stromal tumors: A consensus approach.
- Biology and genetic aspects of gastrointestinal stromal tumors: KIT activation and cytogenetic alterations.
- The c-KIT mutation causing human mastocytosis is resistant to STI571 and other KIT kinase inhibitors; kinases with enzymatic site mutations show different inhibitor se...
- Kwashiorkor in patients with AIDS.
- SCF/c-kit signaling is required for cyclic regeneration of the hair pigmentation unit.
- Fusarium fingernail infection responsive to fluconazole intermittent therapy.
- New approaches to therapy for mastocytosis. A case for treatment with kit kinase inhibitors.
- A proposed classification of mastocytosis incorporating molecular genetics.
- Indolinone derivatives inhibit constitutively activated KIT mutants and kill neoplastic mast cells.
- P-glycoprotein expression and multidrug resistance in cutaneous T-cell lymphoma.
- What dermatologists need to know about mast cell disease: a dermatopathologist's view.
- Cutaneous cryoanesthesia does not affect histopathology of biopsy specimens.
- Eruptive paraneoplastic keloids.
- SCF-KIT pathway in human epidermal melanocyte homeostasis.
- Inhibition of spontaneous receptor phosphorylation by residues in a putative alpha-helix in the KIT intracellular juxtamembrane region.
- Activating and dominant inactivating c-KIT catalytic domain mutations in distinct clinical forms of human mastocytosis.
- Clustering of activating mutations in c-KIT's juxtamembrane coding region in canine mast cell neoplasms.
- Murine cutaneous mastocytosis and epidermal melanocytosis induced by keratinocyte expression of transgenic stem cell factor.
- Tumor-specific peptides in cutaneous T-cell lymphoma: association with class I major histocompatibility complex and possible derivation from the clonotypic T-cell rece...
- Chymase cleavage of stem cell factor yields a bioactive, soluble product.
- Human skin/SCID mouse chimeras as an in vivo model for human cutaneous mast cell hyperplasia.
- Role of CD4+ T cells in pathogenesis associated with Leishmania amazonensis infection.
- Chronically KIT-stimulated clonally-derived human mast cells show heterogeneity in different tissue microenvironments.
- Multiple tumors of the follicular infundibulum.
- Somatic c-KIT activating mutation in urticaria pigmentosa and aggressive mastocytosis: establishment of clonality in a human mast cell neoplasm.
- Disruption of CD40-CD40 ligand interactions results in an enhanced susceptibility to Leishmania amazonensis infection.
- Papilloma formation in human foreskin xenografts after inoculation of human papillomavirus type 16 DNA.
- Targetoid hemangioma associated with pregnancy and the menstrual cycle.
Schools
University of Wisconsin Madison
Presby Hospital
Columbia Presby Med Center
Doctors Specialties
- Dermatopathology
- Dermatology
Accepted Insurances
Education
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New York Presbyterian Hospital - Columbia Campus -
University of Wisconsin School of Medicine and Public Health
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