Assistant Professor, Surgery, University of Pittsburgh School of Medicine
General Surgery
Dr. Brian S. Zuckerbraun, MD
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Brian S. Zuckerbraun, MD practices General Surgery. He researched medicine at Northwestern University Feinberg School of Medicine. Northwestern University Feinberg School of Medicine has a rank of 19 in research and a 29 in primary care He received 2 awards: "Elected Member" and "Fellow (FACS)". Brian S. Zuckerbraun is a published physician. He has 81 papers published. The most current publication "Role of nitric oxide in the regulation of osteoblast metabolism." Brian Zuckerbraun is registered with Medicare and accepts Medicare payments.
Publications
- MITOCHONDRIAL FUNCTION IN SEPSIS.
- CT abbreviated assessment of sarcopenia following trauma: The CAAST measurement predicts 6-month mortality in older adult trauma patients.
- Femoral Hernias in Patients Undergoing Total Extraperitoneal Laparoscopic Hernia Repair: Including Routine Evaluation of the Femoral CAnal in Approaches to Inguinal He...
- Operative Risk Stratification in the Older Adult.
- Clostridium Difficile Infection: Prevention, Treatment, and Surgical Management.
- Adenosine monophosphate-activated protein kinase activation protects against sepsis-induced organ injury and inflammation.
- CaMKIV-Dependent Preservation of mTOR Expression Is Required for Autophagy during Lipopolysaccharide-Induced Inflammation and Acute Kidney Injury.
- Carbon monoxide protects against hemorrhagic shock and resuscitation-induced microcirculatory injury and tissue injury.
- Experimental sepsis-induced mitochondrial biogenesis is dependent on autophagy, TLR4, and TLR9 signaling in Liver.
- Polymicrobial sepsis is associated with decreased hepatic oxidative phosphorylation and an altered metabolic profile.
- Metabolomics in pneumonia and sepsis: an Analysis of the GenIMS cohort study.
- CaMKI? Regulates AMP Kinase-Dependent, TORC-1-Independent Autophagy during Lipopolysaccharide-Induced Acute Lung Neutrophilic Inflammation.
- The role of acute care surgery in the treatment of severe, complicated Clostridium difficile-associated Disease.
- Novel management strategies in the treatment of severe Clostridium difficile infection.
- Nitrite activates AMP kinase to stimulate mitochondrial biogenesis independent of soluble guanylate cyclase.
- Activated CD47 Promotes Pulmonary Arterial Hypertension Through Targeting Caveolin-1.
- Interferon Regulatory Factor 1 (IRF-1) induces p21(WAF1/CIP1) dependent cell cycle arrest and p21(WAF1/CIP1) independent modulation of survivin in Cancer cells.
- Biliverdin inhibits Toll-like receptor-4 (TLR4) expression through nitric oxide-dependent nuclear translocation of biliverdin reDuctase.
- Diverting Loop Ileostomy and Colonic Lavage: An Alternative to Total Abdominal Colectomy for the Treatment of Severe, Complicated Clostridium difficile Associated Dise...
- Nitrite-generated nitric oxide to protect against intimal hyperplasia formation.
- Robotic-assisted laparoscopic side-to-side lateral Pancreaticojejunostomy.
- Nitrite-generated NO circumvents dysregulated arginine/NOS signaling to protect against intimal hyperplasia in Sprague-Dawley rats.
- Heme oxygenase-1-mediated autophagy protects against hepatocyte cell death and hepatic injury from infection/sepsis in mice.
- Lipopolysaccaride induces autophagic signaling in macrophages via a TLR4, heme oxygenase-1 dependent pathway.
- Heme oxygenase 1 protects against hepatic hypoxia and injury from hemorrhage via regulation of cellular respiration.
- Nitrite potently inhibits hypoxic and inflammatory pulmonary arterial hypertension and smooth muscle proliferation via xanthine oxidoreDuctase-dependent nitric oxide g...
- HO-1 and CO decrease platelet-derived growth factor-induced Vascular smooth muscle cell migration via inhibition of Nox1.
- Nitrate and nitrite in biology, nutrition and therapeutics.
- Cell surface biliverdin reDuctase mediates biliverdin-induced anti-inflammatory effects via phosphatidylinositol 3-kinase and Akt.
- Estrogen therapy for trauma/hemorrhage: the heart follows suit.
- Ketamine-induced hepatoprotection: the role of heme oxygenase-1.
- Mechanisms of Toll-like receptor 4 (TLR4)-mediated inflammation after cold ischemia/reperfusion in the heart.
- Cystic parathyroid lesions: functional and nonfunctional parathyroid cysts.
- Carbon monoxide activates NF-kappaB via ROS generation and Akt pathways to protect against cell death of hepatocytes.
- Galantamine and carbon monoxide protect brain microvascular endothelial cells by heme oxygenase-1 inDuction.
- Protective functions of heme oxygenase-1 and carbon monoxide in the respiratory system.
- Toll-like receptor 4 mediates the early inflammatory response after cold ischemia/reperfusion.
- Hypoxia activates c-Jun N-terminal kinase via Rac1-dependent reactive oxygen species proDuction in hepatocytes.
- Thioredoxin catalyzes the denitrosation of low-molecular mass and protein S-nitrosothiols.
- Carbon monoxide signals via inhibition of cytochrome c oxidase and generation of mitochondrial reactive oxygen species.
- Nitric oxide-induced inhibition of smooth muscle cell proliferation involves S-nitrosation and inactivation of RhoA.
- Carbon monoxide reverses established pulmonary hypertension.
- Carbon monoxide protects hepatocytes from TNF-alpha/Actinomycin D by inhibition of the caspase-8-mediated apoptotic pathway.
- Carbon monoxide protects against the development of experimental necrotizing enterocolitis.
- Nitric oxide modulates Vascular inflammation and intimal hyperplasia in insulin resistance and the metabolic syndrome.
- Carbon monoxide prevents multiple organ injury in a model of hemorrhagic shock and resuscitation.
- Inhibition of geranylgeranyltransferase I decreases generation of vascular reactive oxygen species and increases vascular nitric oxide proDuction.
- Carbon monoxide protection against endotoxic shock involves reciprocal effects on iNOS in the Lung and liver.
- Effect of age on cervical spine injuries in children after motor vehicle collisions: effectiveness of restraint devices.
- Carbon monoxide protects against Liver failure through nitric oxide-induced heme oxygenase 1.
- A role for angiotensin II in the activation of extracellular signal-regulated kinases in the Liver during hemorrhagic shock.
- Carbon monoxide inhalation protects rat Intestinal grafts from ischemia/reperfusion injury.
- Overexpression of mutated IkappaBalpha inhibits Vascular smooth muscle cell proliferation and intimal hyperplasia formation.
- RhoA influences the nuclear localization of extracellular signal-regulated kinases to modulate p21Waf/Cip1 expression.
- Protective effect of carbon monoxide inhalation for cold-preserved small Intestinal grafts.
- Carbon monoxide induces cytoprotection in rat orthotopic Lung transplantation via anti-inflammatory and anti-apoptotic effects.
- Heme oxygenase-1: a cellular Hercules.
- Carbon monoxide suppresses arteriosclerotic lesions associated with chronic graft rejection and with balloon injury.
- Hepatocyte Fas-associating death domain protein/mediator of receptor-induced toxicity (FADD/MORT1) levels increase in response to pro-apoptotic stimuli.
- Vascular gene therapy: a reality of the 21st century.
- Massive transfusion protocol activation does not result in preferential use of older red blood cells.
- Heme oxygenase-1 derived carbon monoxide permits maturation of myeloid cells.
- Mitochondrial signaling: forwards, backwards, and in between.
- Therapeutic Potential of the Nitrite-Generated NO Pathway in Vascular Dysfunction.
- Augmenting Autophagy to Treat Acute Kidney Injury during Endotoxemia in Mice.
- Avoiding colectomy during surgical management of fulminant Clostridium difficile colitis.
- Isolated skeletal muscle metastasis following successful treatment of laryngeal Cancer: case report.
- Hypoxia renders hepatocytes susceptible to cell death by nitric oxide.
- Intestinal and hepatic expression of BNIP3 in necrotizing enterocolitis: regulation by nitric oxide and peroxynitrite.
- Immunomodulatory effects of inhaled carbon monoxide on rat syngeneic small bowel graft motility.
- Role of nitric oxide in Liver injury.
- Regulation of tissue factor expression in smooth muscle cells with nitric oxide.
- Management of parotid leakage following rhytidectomy.
- An alternative to a graft of skin in the treatment of lagophthalmos.
- Role of nitric oxide in the regulation of osteoblast metabolism.
- Preoperative psychological evaluation and intervention in prospective amputees.
- Carbon monoxide: medicinal chemistry and biological effects.
- Nitrite in pulmonary Arterial hypertension: therapeutic avenues in the setting of dysregulated arginine/nitric oxide synthase signalling.
- ''Til cell death do us part: nitric oxide and mechanisms of hepatotoxicity.
- IRF-1 expression induces apoptosis and inhibits tumor growth in mouse mammary Cancer cells in vitro and in vivo.
Schools
Northwestern Center Feinberg School of Medicine
Procedures Preformed
- Appendectomy, Laparoscopic
- Colectomy
- Decortication and Pleurectomy
- Dressing and-or Debridement of Wound, Infection, or Burn (incl. Negative Pressure Wound Therapy)
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Conditions Treated
- Abdominal Pain
- Anal and Rectal Cancer
- Anorectal Abscess
- Appendicitis
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Doctors Specialties
- General Surgery
Accepted Insurances
Awards
- Elected Member
- Fellow (FACS)
Education
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University of Pittsburgh Medical Center -
Northwestern University Feinberg School of Medicine
Hospital
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UPMC Presbyterian Shadyside
Drug Facts
| NPI NUMBER | 1639145824 | |
| NPPES Provider LastName | ZUCKERBRAUN | |
| NPPES Provider FirstName | BRIAN | |
| NPPES Provider ZIPCode | 152132546 | |
| NPPES Provider State | PA | |
| Specialty Description | General Surgery | |
| Total Claim Count | 38.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1639145824 |
| Last Name Of The Provider | ZUCKERBRAUN |
| First Name Of The Provider | BRIAN |
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