Associate Professor, Pharmacology and Therapeutics, University of Florida College of Medicine
Hematology
Dr. Bradley S. Fletcher, MD
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Doctor Bradley S. Fletcher, MD practices Hematology. He went to David Geffen School of Medicine at UCLA. David Geffen School of Medicine at UCLA is a top medical college. David Geffen School of Medicine at UCLA has a rank of 13 in research and a 7 in primary care Doctor Bradley S. Fletcher, MD is a published doctor and has 39 publications published. The lastest was: 'Expression of the protein product of the prostaglandin synthase-2/TIS10 gene in mitogen-stimulated Swiss 3T3 cells.' Bradley Fletcher accepts Medicare payments and is listed with Medicare.gov.
Publications
- Delivery of small interfering RNA (siRNA) using the sleeping beauty transposon.
- PiggyBac transposon-based inducible gene expression in vivo after somatic Cell gene transfer.
- Targeting tumor endothelial marker 8 in the tumor vasculature of colorectal carcinomas in mice.
- Sleeping Beauty-mediated eNOS gene therapy attenuates monocrotaline-induced pulmonary hypertension in rats.
- Sleeping Beauty-based gene therapy with indoleamine 2,3-dioxygenase inhibits lung allograft fibrosis.
- Sustained FVIII expression and phenotypic correction of Hemophilia A in neonatal mice using an endothelial-targeted sleeping beauty transposon.
- Novel action of indoleamine 2,3-dioxygenase attenuating Acute lung allograft injury.
- Hyperactive transposase mutants of the Sleeping Beauty transposon.
- The WD-repeat protein GRWD1: potential roles in Myeloid differentiation and ribosome biogenesis.
- Modulation of epithelial morphology, monolayer permeability, and Cell migration by growth arrest specific 3/peripheral myelin protein 22.
- Endothelial targeting of the Sleeping Beauty transposon within lung.
- Transposon-based RNAi delivery system for generating knockdown Cell lines.
- Glucose deprivation enhances targeting of GLUT1 to lipid rafts in 3T3-L1 adipocytes.
- The TIS11 primary response gene is a member of a gene family that encodes proteins with a highly conserved sequence containing an unusual Cys-His repeat.
- Transcriptional induction of prostaglandin G/H synthase-2 by basic fibroblast growth factor.
- TIS10, a mitogen-inducible glucocorticoid-inhibited gene that encodes a second prostaglandin synthase/cyclooxygenase enzyme.
- Platelet-activating factor and retinoic acid synergistically activate the inducible prostaglandin synthase gene.
- Structure and expression of TIS21, a primary response gene induced by growth factors and tumor promoters.
- v-src induction of the TIS10/PGS2 prostaglandin synthase gene is mediated by an ATF/CRE transcription response element.
- Expression of the protein product of the prostaglandin synthase-2/TIS10 gene in mitogen-stimulated Swiss 3T3 Cells.
- Chromosomal organization of the inducible and constitutive prostaglandin synthase/cyclooxygenase genes in mouse.
- TIS10, a phorbol ester tumor promoter-inducible mRNA from Swiss 3T3 Cells, encodes a novel prostaglandin synthase/cyclooxygenase homologue.
- Structure of the mitogen-inducible TIS10 gene and demonstration that the TIS10-encoded protein is a functional prostaglandin G/H synthase.
- Asparagine synthetase expression alone is sufficient to induce l-asparaginase resistance in MOLT-4 human leukaemia Cells.
- The tis genes, primary response genes induced by growth factors and tumor promoters in 3T3 Cells.
- Bcl-2 mediated modulation of vascularization in proState cancer xenografts.
- The promise of nanotechnology for solving clinical problems in breast cancer.
- Molecular evolution of a novel hyperactive Sleeping Beauty transposase enables robust stable gene transfer in vertebrates.
- Potent anti-tumor effects of an active site mutant of human manganese-superoxide dismutase. Evolutionary conservation of product inhibition.
- Functional cloning of SPIN-2, a nuclear anti-apoptotic protein with roles in Cell cycle progression.
- Peripheral myelin protein 22 is a constituent of interCellular junctions in epithelia.
- Indoleamine 2,3-dioxygenase attenuates inhibitor development in gene-therapy-treated Hemophilia A mice.
- Molecular alterations resulting from frameshift mutations in peripheral myelin protein 22: implications for neuropathy severity.
- Tumor endothelial marker 8 expression in triple-negative breast cancer.
- Glycogen phosphorylase is activated in response to glucose deprivation but is not responsible for enhanced glucose transport activity in 3T3-L1 adipocytes.
- Impaired proteasome activity and accumulation of ubiquitinated substrates in a hereditary neuropathy model.
- Sleeping beauty transposon-mediated nonviral gene therapy.
- Development and validation of an approach to produce large-scale quantities of CpG-methylated plasmid DNA.
Schools
David Geffen School Of Medicine At UCLA University Of California Los Angeles
Stanford Med Center
Stanford Hospital
Procedures Preformed
- Plasmapheresis
Conditions Treated
- Acute Leukemia
- Anemia
- Antiphospholipid Syndrome (APS)
- Aplastic Anemia
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Doctors Specialties
- Hematology
Accepted Insurances
Education
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Stanford University -
David Geffen School of Medicine at UCLA -
UCLA
Hospital
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UF Health Shands Hospital
Drug Facts
| NPI NUMBER | 1427099423 | |
| NPPES Provider LastName | FLETCHER | |
| NPPES Provider FirstName | BRADLEY | |
| NPPES Provider ZIPCode | 326103003 | |
| NPPES Provider State | FL | |
| Specialty Description | Hematology | |
| Total Claim Count | 658.0 | |
| Distinct Opioid Count | 7.0 | |
| Opioid Claim Count | 288.0 | |
| Percent Opioid Claims | 43.77 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1427099423 |
| Last Name Of The Provider | FLETCHER |
| First Name Of The Provider | BRADLEY |
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