Assistant Professor, Pediatrics, UCSF School of Medicine
Pediatric Hematology & Oncology
Dr. Benjamin S. Braun, MD
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Doctor Benjamin S. Braun, MD works in the field of Pediatric Hematology & Oncology. He studied medicine at David Geffen School of Medicine at UCLA. David Geffen School of Medicine at UCLA is a top medical university. David Geffen School of Medicine at UCLA has a rank of 13 in research and a 7 in primary care He received awards:"CMS Meaningful Use Stage 1 Certification". Benjamin S. Braun has written publications. He has 27 publications published with PubMed. The lastest publication is: 'EWS/FLI1-induced manic fringe renders NIH 3T3 cells tumorigenic.'
Publications
- Subclonal mutations in SETBP1 confer a poor prognosis in juvenile myelomonocytic Leukemia.
- Sustained MEK inhibition abrogates myeloproliferative disease in Nf1 mutant mice.
- KIT's ship comes in.
- Targeting oncogenic Ras signaling in hematologic malignancies.
- Essential role for Ptpn11 in survival of hematopoietic stem and progenitor Cells.
- A MEK inhibitor abrogates myeloproliferative disease in Kras mutant mice.
- Hematopoiesis and leukemogenesis in mice expressing oncogenic NrasG12D from the endogenous locus.
- Germline CBL mutations cause developmental abnormalities and predispose to juvenile myelomonocytic Leukemia.
- Mutant Ikzf1, KrasG12D, and Notch1 cooperate in T lineage leukemogenesis and modulate responses to targeted agents.
- The SPS affair: a complex tale of illicit proliferation.
- JunB protects against myeloid malignancies by limiting hematopoietic stem Cell proliferation and differentiation without affecting self-renewal.
- Oncogenic Kras initiates Leukemia in hematopoietic stem cells.
- Targeting Ras in myeloid Leukemias.
- K-RasG12D expression induces hyperproliferation and aberrant signaling in primary hematopoietic stem/progenitor Cells.
- Somatic activation of a conditional KrasG12D allele causes ineffective erythropoiesis in vivo.
- Inherited predispositions and hyperactive Ras in myeloid leukemogenesis.
- Congenital Leukemia cutis with subsequent development of Leukemia.
- Somatic inactivation of Nf1 in hematopoietic Cells results in a progressive myeloproliferative disorder.
- The sum is greater than the FGFR1 partner.
- Somatic activation of oncogenic Kras in hematopoietic Cells initiates a rapidly fatal myeloproliferative disorder.
- PLC-γ and PI3K Link Cytokines to ERK Activation in Hematopoietic Cells with Normal and Oncogenic Kras.
- Identification of target genes for the Ewing's Sarcoma EWS/FLI fusion protein by representational difference analysis.
- EWS/FLI1-induced manic fringe renders NIH 3T3 Cells tumorigenic.
- Multiple domains mediate transformation by the Ewing''s Sarcoma EWS/FLI-1 fusion gene.
- Pediatric malignancies: update on sarcomas and Leukemia development in children.
- Targeting the PI3K/Akt pathway in murine MDS/MPN driven by hyperactive Ras.
Schools
David Geffen School Of Medicine At UCLA University Of California Los Angeles
University Ca Sf School Of Med
Conditions Treated
- Acute Leukemia
- Adrenal Gland Cancer
- Anemia
- Brain and Nervous System Cancer (incl. Gliomas, Astrocytoma, Schwannoma, Medulloblastoma, Chordoma)
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Doctors Specialties
- Pediatric Hematology
- Oncology
Accepted Insurances
Awards
- CMS Meaningful Use Stage 1 Certification
Education
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UCSF -
David Geffen School of Medicine at UCLA
Hospital
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UCSF Medical Center
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